نتایج جستجو برای: tnbs

تعداد نتایج: 810  

Journal: :Biological & pharmaceutical bulletin 2011
Ken Inoue Yuji Naito Tomohisa Takagi Natsuko Hayashi Yasuko Hirai Katsura Mizushima Ryusuke Horie Kohei Fukumoto Shinya Yamada Akihito Harusato Ikuhiro Hirata Tatsushi Omatsu Naohisa Yoshida Kazuhiko Uchiyama Takeshi Ishikawa Osamu Handa Hideyuki Konishi Naoki Wakabayashi Nobuaki Yagi Hiroshi Ichikawa Satoshi Kokura Toshikazu Yoshikawa

Heat shock protein (HSP) 47 may play an important role in the pathogenesis of intestinal fibrosis. Daikenchuto (DKT), a traditional Japanese herbal (Kampo) medicine, has been reported to ameliorate intestinal inflammation. The aims of this study were to determine time-course profiles of several parameters of fibrosis in a rat model, to confirm the HSP47-expressing cells in the colon, and finall...

Journal: :Pharmacological reports : PR 2013
Prasad Byrav D S Bikash Medhi Ajay Prakash Amitava Chakrabarti Kim Vaiphei Krishan L Khanduja

BACKGROUND Inflammatory bowel disease (IBD) is an idiopathic, chronic inflammatory condition, which affects the gastrointestinal tract and has no curative treatment. The present study aimed to investigate the effect of different doses of pioglitazone alone and in combination with sulfasalazine in TNBS (trinitrobenzenesulfonic acid)-induced inflammatory bowel disease (IBD) in rats. METHODS A t...

Journal: :Medical principles and practice : international journal of the Kuwait University, Health Science Centre 2011
Dina M Jragh Islam Khan Mabayoje A Oriowo

OBJECTIVES Carbachol-induced contraction of the rat colon is impaired in rats with trinitrobenzene sulfonic acid (TNBS)-induced colitis. The main objective of this study was to examine the effect of colitis on the expression and function of muscarinic (M) receptor subtypes in the rat colon. MATERIALS AND METHODS Rats (n = 80) were treated with TNBS and used 5 days later for measurement of con...

2014
John K. Triantafillidis Georgia Douvi George Agrogiannis Efstratios Patsouris Aristofanis Gikas Apostolos E. Papalois

BACKGROUND Experimental data suggest that oral iron (I.) supplementation can worsen colitis in animals. AIM To investigate the influence of various concentrations of orally administered I. in normal gut mucosa and mucosa of animals with TNBS colitis, as well as the influence of Mesalamine (M.) and Prednisolone (P.) on the severity of TNBS colitis following orally administered I. METHODS AND...

Journal: :The Journal of nutrition 2010
Aktham Hassan Ayman Ibrahim Khaly Mbodji Moïse Coëffier Frédéric Ziegler Frédéric Bounoure Jean-Michel Chardigny Mohamed Skiba Guillaume Savoye Pierre Déchelotte Rachel Marion-Letellier

We have previously shown that α-linolenic acid (ALA), a (n-3) PUFA exerts in vitro antiinflammatory effects in the intestine. In this study, we aimed to evaluate its effect on inflammatory and oxidative stress in a colitis model. Colitis was induced in 2 groups at d 0 by intrarectal injection of 2-4-6-trinitrobenzen sulfonic acid (TNBS), whereas the control group received the vehicle. Rats we f...

2013
Øystein Brenna Marianne W. Furnes Ignat Drozdov Atle van Beelen Granlund Arnar Flatberg Arne K. Sandvik Rosalie T. M. Zwiggelaar Ronald Mårvik Ivar S. Nordrum Mark Kidd Björn I. Gustafsson

BACKGROUND Rectal instillation of trinitrobenzene sulphonic acid (TNBS) in ethanol is an established model for inflammatory bowel disease (IBD). We aimed to 1) set up a TNBS-colitis protocol resulting in an endoscopic and histologic picture resembling IBD, 2) study the correlation between endoscopic, histologic and gene expression alterations at different time points after colitis induction, an...

1996
Markus F. Neurath Ivan Fuss Brian L. Kelsall David H. Presky Wendy Waegell Warren Strober

In previous studies we showed that a chronic colitis associated with a Thl T cell response can be induced by the rectal administration of the haptenizing reagent 2,4,6-trinitrobenzene sulfonic acid (TNBS). We report here that oral administration of haptenized colonic proteins (HCP) before rectal administration of TNBS effectively suppresses the ability of the latter to induce colitis. This supp...

Journal: :American journal of physiology. Gastrointestinal and liver physiology 2011
Xuan-Zheng Shi John H Winston Sushil K Sarna

Crohn's disease and ulcerative colitis are clinically, immunologically, and morphologically distinct forms of inflammatory bowel disease (IBD). However, smooth muscle function is impaired similarly in both diseases, resulting in diarrhea. We tested the hypothesis that differential cellular, genetic, and immunological mechanisms mediate smooth muscle dysfunction in two animal models believed to ...

Journal: :The Journal of Experimental Medicine 1996
M F Neurath I Fuss B L Kelsall D H Presky W Waegell W Strober

In previous studies we showed that a chronic colitis associated with a Th1 T cell response can be induced by the rectal administration of the haptenizing reagent 2,4,6-trinitrobenzene sulfonic acid (TNBS). We report here that oral administration of haptenized colonic proteins (HCP) before rectal administration of TNBS effectively suppresses the ability of the latter to induce colitis. This supp...

Journal: :American journal of physiology. Regulatory, integrative and comparative physiology 2007
Sarah J Spencer Niall P Hyland Keith A Sharkey Quentin J Pittman

Early life events and childhood infections have been associated with the development and onset of inflammatory bowel disease in adulthood. However, the consequences of neonatal infection in the development and severity of colitis are not established. We investigated the effects of a neonatal (postnatal day 14) or juvenile (postnatal day 28) immune challenge with LPS on 2,4,6-trinitrobenzenesulf...

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