نتایج جستجو برای: tetraploidy

تعداد نتایج: 1945  

Journal: :Advances in Animal and Veterinary Sciences 2016

2012
Yumie Takata Alan R. Kristal Regina M. Santella Irena B. King David J. Duggan Johanna W. Lampe Margaret P. Rayman Patricia L. Blount Brian J. Reid Thomas L. Vaughan Ulrike Peters

Clinical trials have suggested a protective effect of selenium supplementation on the risk of esophageal cancer, which may be mediated through the antioxidant activity of selenoenzymes. We investigated whether serum selenium concentrations, selenoenzyme activity, oxidative stress and genetic variation in selenoenzymes were associated with the risk of neoplastic progression to esophageal adenoca...

2016
Yi-Jin Lu Krishna B S Swamy Jun-Yi Leu

Polyploidization has crucial impacts on the evolution of different eukaryotic lineages including fungi, plants and animals. Recent genome data suggest that, for many polyploidization events, all duplicated chromosomes are maintained and genome reorganizations occur much later during evolution. However, newly-formed polyploid genomes are intrinsically unstable and often quickly degenerate into a...

2014
Beata Ujvari Anne-Maree Pearse Kate Swift Pamela Hodson Bobby Hua Stephen Pyecroft Robyn Taylor Rodrigo Hamede Menna Jones Katherine Belov Thomas Madsen

The Tasmanian Devil Facial Tumour Disease (DFTD) provides a unique opportunity to elucidate the long-term effects of natural and anthropogenic selection on cancer evolution. Since first observed in 1996, this transmissible cancer has caused local population declines by >90%. So far, four chromosomal DFTD variants (strains) have been described and karyotypic analyses of 253 tumours showed higher...

2014
Andreas Panopoulos Cristina Pacios-Bras Justin Choi Mythili Yenjerla Mark A. Sussman Rati Fotedar Robert L. Margolis Daniel J. Lew

Tetraploidy can arise from various mitotic or cleavage defects in mammalian cells, and inheritance of multiple centrosomes induces aneuploidy when tetraploid cells continue to cycle. Arrest of the tetraploid cell cycle is therefore potentially a critical cellular control. We report here that primary rat embryo fibroblasts (REF52) and human foreskin fibroblasts become senescent in tetraploid G1 ...

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