Dr. Chittaranjan Andrade shared a case in a letter to the editor in the July 2001 issue of Indian Journal of Psychiatry (Andrade, 2001) and he, as proposed by Dr. Fink (Fink, 2001) must be congratulated for bringing this treatable, life threatening condition to the notice of psychiatric fraternity and we must also thank Dr. Fink for giving guidelines to manage excited manic patients in the same...

We report a case of prolonged loss of consciousness due to hypoglycaemia following ethanol abuse in a non-diabetic. The patient also reported symptoms compatible with hypoglycaemia following heavy manual work. Further investigations revealed a pancreatic insulinoma, which was successfully removed surgically. The patient remains asymptomatic 18 months later, despite occasional episodes of ethano...

a hypothetical neuroleptic-induced neurotoxic state of a nature different from the NMS? It is very unlikely: why would neuroleptic-induced neurotoxicity suddenly give way to catatonic excitement and then return the patient to a state of stupor? A patient experiencing a drug-induced state of suppression would be expected to remain in the state of suppression until the effect of the drug wore off...

Altered mental status is a common chief compliant among older patients in the emergency department (ED). Acute changes in mental status are more concerning and are usually secondary to delirium, stupor, and coma. Although stupor and coma are easily identifiable, the clinical presentation of delirium can be subtle and is often missed without actively screening for it. For patients with acute cha...

Presentation of patients with recurrent stupor associated with apparently elevated levels of an endogenous benzodiazepine-like agent, endozepine-4, has been reported from several centers, and a new syndrome, endozepine stupor has been proposed. We recently reported a case with typical features of this syndrome, which proved to be an example of surreptitious administration of exogenous benzodiaz...

We describe a case of catatonic stupor following simultaneous biparietal infarction. The patient recovered, a result not previously described in catatonia caused by this pattern of cerebral infarct.