نتایج جستجو برای: spink1

تعداد نتایج: 252  

Journal: :JOP : Journal of the pancreas 2003
Andrea L Ferreira Bernardino Dulce Reis Guarita Carlos Barros Mott Martha Regina Arcon Pedroso Marcel Cerqueira Cesar Machado Antonio Atilio Laudanna Claudia Megume Tani Fatima Lovatti Almeida Mayana Zatz

CONTEXT Mutations in cystic fibrosis transmembrane conductance regulator (CFTR), in cationic trypsinogen (PRSS1) and in serine protease inhibitor Kazal type 1 (SPINK1) genes have been associated with chronic pancreatitis (alcohol related, idiopathic and hereditary). However, the inheritance pattern is still not clear. PATIENTS Eighty-two unrelated Brazilian patients with chronic pancreatitis ...

2005
Asif Khalid Sydney Finkelstein Bryan Thompson Lori Kelly Christoph Hanck Tony E. Godfrey David C. Whitcomb

Background: The cationic trypsinogen (PRSS1) R122H mutation causes autosomal dominant hereditary pancreatitis (HP) with multiple attacks of acute pancreatitis, but the penetrance, frequency and severity of attacks are highly variable. HP twins study suggests that modifier genes influence severity, but not penetrance. Aim: To investigate potential trypsin-associated factors in subjects with the ...

2003
F U Weiss P Simon H Witt J Mayerle V Hlouschek K P Zimmer J Schnekenburger W Domschke J P Neoptolemos M M Lerch

Hereditary pancreatitis (HP) is an inborn disorder which leads to recurrent episodes of pancreatitis in children and young adults and is associated with exocrine pancreatic insufficiency and secondary diabetes. Several germline mutations in the cationic trypsinogen (PRSS1) gene have been found to be associated with the disease phenotype, the most common of which are the R122H, N29I, and A16V mu...

Journal: :Journal of medical genetics 2003
F U Weiss P Simon H Witt J Mayerle V Hlouschek K P Zimmer J Schnekenburger W Domschke J P Neoptolemos M M Lerch

Hereditary pancreatitis (HP) is an inborn disorder which leads to recurrent episodes of pancreatitis in children and young adults and is associated with exocrine pancreatic insufficiency and secondary diabetes. Several germline mutations in the cationic trypsinogen (PRSS1) gene have been found to be associated with the disease phenotype, the most common of which are the R122H, N29I, and A16V mu...

Journal: :The Prostate 2015
Bushra Ateeq Lakshmi P Kunju Shannon L Carskadon Swaroop K Pandey Geetika Singh Immanuel Pradeep Vini Tandon Atin Singhai Apul Goel Sonal Amit Asha Agarwal Amit K Dinda Amlesh Seth Alexander Tsodikov Arul M Chinnaiyan Nallasivam Palanisamy

BACKGROUND Molecular stratification of prostate cancer (PCa) based on genetic aberrations including ETS or RAF gene-rearrangements, PTEN deletion, and SPINK1 over-expression show clear prognostic and diagnostic utility. Gene rearrangements involving ETS transcription factors are frequent pathogenetic somatic events observed in PCa. Incidence of ETS rearrangements in Caucasian PCa patients has b...

Journal: :Urology 2014
Riina-Minna Väänänen Hans Lilja Leni Kauko Pauliina Helo Henna Kekki Angel M Cronin Andrew J Vickers Martti Nurmi Kalle Alanen Anders Bjartell Kim Pettersson

OBJECTIVE To investigate whether messenger ribonucleic acid (mRNA) expression of TMPRSS2-ERG fusion gene, a suggested prostate cancer (PCa) biomarker, was specific to cancerous lesions alone and to study the expression of SPINK1 and PCA3 mRNAs in the same cohort to also explore the proposed mutual exclusivity of TMPRSS2-ERG and SPINK1 expression. METHODS Levels of 2 TMPRSS2-ERG transcripts, P...

2014
Francesca Khani Juan Miguel Mosquera Kyung Park Mirjam Blattner Catherine O'Reilly Theresa Y. MacDonald Zhengming Chen Abhishek Srivastava Ashutosh K. Tewari Christopher E. Barbieri Mark A. Rubin Brian D. Robinson

Purpose: The aim of this study was to compare the frequency of ERG rearrangement, PTEN deletion, SPINK1 overexpression, and SPOP mutation in prostate cancer in African American and Caucasian men. Experimental design: Dominant tumor nodules from radical prostatectomy specimens of 105 African American men (AAM) were compared with 113 dominant nodules from Caucasian men (CaM). Clinical and patholo...

2017
Janaki Patel Arina Madan Amanda Gammon Michael Sossenheimer Niloy Jewel Samadder

Hereditary chronic pancreatitis associated with a mutation in the serine protease inhibitor, Kazal Type-1 (SPINK-1 gene) is extremely rare. The SPINK1 mutation results in trypsinogen activation which predisposes to chronic pancreatitis predominately when combined with CFTR gene mutations. It presents as either chronic or recurrent acute pancreatitis. Symptom control and management of complicati...

2014
Richard Flavin Andreas Pettersson Whitney K. Hendrickson Michelangelo Fiorentino Stephen Finn Lauren Kunz Gregory L. Judson Rosina Lis Dyane Bailey Christopher Fiore Elizabeth Nuttall Neil E. Martin Edward Stack Kathryn L. Penney Jennifer R. Rider Jennifer Sinnott Christopher Sweeney Howard D. Sesso Katja Fall Edward Giovannucci Philip Kantoff Meir Stampfer Massimo Loda Lorelei A. Mucci

Purpose: SPINK1overexpression has been described in prostate cancer and is linkedwith poor prognosis in many cancers. The objective of this study was to characterize the association between SPINK1 overexpression and prostate cancer–specific survival. Experimental Design: The study included 879 participants in the U.S. Physicians’ Health Study and Health Professionals Follow-Up Study, diagnosed ...

Journal: :Gut 2017
Hao Wu Arnaud Boulling David N Cooper Zhao-Shen Li Zhuan Liao Jian-Min Chen Claude Férec

We read with interest the recent publication of Beer and Sahin-Tóth reporting that exonic variants affecting pre-mRNA splicing contribute to the genetic burden in chronic pancreatitis. One particular variant, affecting the last nucleotide of exon 3 of the SPINK1 gene, c.194G>A, was found to cause an ∼80% reduction in SPINK1 mRNA expression as compared with the wild type in a minigene assay perf...

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