نتایج جستجو برای: proprotein convertase

تعداد نتایج: 3949  

Journal: :Atlas of Genetics and Cytogenetics in Oncology and Haematology 2017

Journal: :Atlas of Genetics and Cytogenetics in Oncology and Haematology 2015

Journal: :European heart journal 2015
Yuichi J Shimada Christopher P Cannon

Reduction in low-density lipoprotein cholesterol (LDL-C), mainly with statins, has decreased the risk of cardiovascular events over the last few decades. However, there are several patient populations that warrant further decrease in LDL-C by additional cholesterol-lowering therapy other than statins. Proprotein convertase subtilisin/kexin type 9 (PCSK9) inhibitors are a new class of drugs that...

2001
D. Rapoport

The human thyrotropin receptor (TSHR) undergoes proteolytic cleavage closely upstream to amino acid 317. Between residues 261 and 313 are three clusters of positively charged amino acids, arginines (Arg) and lysines (Lys), which are potential subtilisin-related proprotein convertase sites. We used oligonucleotide-directed mutagenesis to perform conservative amino acid substitutions within these...

Journal: :Journal of lipid research 2010
Jason S Troutt William E Alborn Guoqing Cao Robert J Konrad

Over the past several years, proprotein convertase subtilisin kexin type 9 (PCSK9) has gained significant attention as a key regulator of serum LDL-cholesterol (LDL-C) levels. In humans, gain-of-function mutations in PCSK9 cause a form of familial hypercholesterolemia, whereas loss-of-function mutations result in significantly decreased LDL-C and cardiovascular risk. Our laboratory was the firs...

Journal: :Journal of Clinical Biochemistry and Nutrition 2023

Background: Proprotein convertase subtilisin/kexin type 9 (PCSK9) inhibitors are a new class of potent lipid-lowering drugs. Oxidized low-density lipoprotein (ox-LDL) is the key pathogenic factor leading to atherosclerosis. However, its effect on ox-LDL levels has not been clinically reported. For very high-risk Atherosclerotic cardiovascular disease (ASCVD) patients, guidelines propose stricte...

Journal: :Arteriosclerosis, thrombosis, and vascular biology 2015
Yasin Hussain Qingming Ding Philip W Connelly J Howard Brunt Matthew R Ban Adam D McIntyre Murray W Huff Robert Gros Robert A Hegele Ross D Feldman

OBJECTIVE Estrogen deficiency is linked with increased low-density lipoprotein (LDL) cholesterol. The hormone receptor mediating this effect is unknown. G-protein estrogen receptor (GPER) is a recently recognized G-protein-coupled receptor that is activated by estrogens. We recently identified a common hypofunctional missense variant of GPER, namely P16L. However, the role of GPER in LDL metabo...

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