نتایج جستجو برای: mutated genes

تعداد نتایج: 445603  

2015
Jan Budczies Michael Bockmayr Carsten Denkert Frederick Klauschen Jochen K Lennerz Balázs Györffy Manfred Dietel Sibylle Loibl Wilko Weichert Albrecht Stenzinger

Breast cancer is a complex molecular disease comprising several biological subtypes. However, daily routine diagnosis is still based on a small set of well-characterized clinico-pathological variables. Here, we try to link the two worlds of surgical pathology and multilayered molecular profiling by analyzing the relationships between clinico-pathological phenotypes and mutational loads of breas...

Journal: :Proceedings of the National Academy of Sciences 2015

Muhammad Haris Lucky, Saeeda Baig

Objective: The role of vitamin D has soared to a pinnacle in recent years with functions affecting 229 human genes linked to cardiovascular, autoimmune, humoral, pulmonary and neurological diseases. Relationship between obesity and vitamin D has not as yet been fully established. The objective of the study was to determine the possible relationship between Vitamin D receptor (VDR) gene polymorp...

Muhammad Haris Lucky, Saeeda Baig

Objective: The role of vitamin D has soared to a pinnacle in recent years with functions affecting 229 human genes linked to cardiovascular, autoimmune, humoral, pulmonary and neurological diseases. Relationship between obesity and vitamin D has not as yet been fully established. The objective of the study was to determine the possible relationship between Vitamin D receptor (VDR) gene polymorp...

Journal: :Cancer research 1991
T A Lehman W P Bennett R A Metcalf J A Welsh J Ecker R V Modali S Ullrich J W Romano E Appella J R Testa

The p53 tumor suppressor gene is frequently mutated and the K-ras oncogene is occasionally mutated in primary specimens of human lung carcinomas. These mutated genes also cooperate in the immortalization and neoplastic transformation of rodent cells. To determine whether these mutations are necessary for maintenance of the immortalized and/or neoplastically transformed states of human bronchial...

Journal: :Cell systems 2017
Borislav H. Hristov Mona Singh

A central goal in cancer genomics is to identify the somatic alterations that underpin tumor initiation and progression. While commonly mutated cancer genes are readily identifiable, those that are rarely mutated across samples are difficult to distinguish from the large numbers of other infrequently mutated genes. We introduce a method, nCOP, that considers per-individual mutational profiles w...

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