نتایج جستجو برای: ifnl4

تعداد نتایج: 81  

Journal: :Nature Reviews Gastroenterology & Hepatology 2013

2014
Felix M. Key Benjamin Peter Megan Y. Dennis Emilia Huerta-Sánchez Wei Tang Ludmila Prokunina-Olsson Rasmus Nielsen Aida M. Andrés

Interferon lambda 4 gene (IFNL4) encodes IFN-l4, a new member of the IFN-l family with antiviral activity. In humans IFNL4 open reading frame is truncated by a polymorphic frame-shift insertion that eliminates IFN-l4 and turns IFNL4 into a polymorphic pseudogene. Functional IFN-l4 has antiviral activity but the elimination of IFN-l4 through pseudogenization is strongly associated with improved ...

Journal: :BMC Infectious Diseases 2021

Abstract Background In non-uremic subjects, IFNL4 rs368234815 predicts HCV clearance. We investigated whether is associated with spontaneous clearance in haemodialysis patients and it a stronger predictor of resolution than the IFNL polymorphisms already dialysis subjects. also evaluated an association patients` survival alterations transcription factor binding sites (TFBS) caused by polymorphi...

2016
E. G. Meissner A. Kohli K. Virtaneva D. Sturdevant C. Martens S. F. Porcella J. G. McHutchison H. Masur S. Kottilil

Chronic hepatitis C virus (HCV) infection can now be treated with oral directly acting antiviral agents, either with or without ribavirin (RBV). Virologic relapse after treatment can occur, and in some studies was more common in cirrhotic subjects. We previously observed changes in hepatic immunity during interferon (IFN)-free therapy that correlated with favourable outcome in subjects with ear...

2014
Lilliana Morales-Vásquez Edgar I. Miranda-Ávalo José R. Rodríguez-Medina Rafael Pastrana Cynthia M. Pérez-Cardona Yasuhiro Yamamura Esther A. Torres

Address correspondence to: Esther A. Torres, MD, Gastroenterology Research Unit, Department of Medicine, University of Puerto Rico Medical Sciences Campus, San Juan, PR. Email: [email protected] Objective: To describe the risk factors for infection, complications, treatment received and response in Puerto Ricans with HCV attending gastroenterology clinics at UPR-MSC, and the prevalence ...

2015
Mohammed Eslam Ahmed M. Hashem Reynold Leung Manuel Romero-Gomez Thomas Berg Gregory J. Dore Henry L.K. Chan William L. Irving David Sheridan Maria L. Abate Leon A. Adams Alessandra Mangia Martin Weltman Elisabetta Bugianesi Ulrich Spengler Olfat Shaker Janett Fischer Lindsay Mollison Wendy Cheng Elizabeth Powell Jacob Nattermann Stephen Riordan Duncan McLeod Nicola J. Armstrong Mark W. Douglas Christopher Liddle David R. Booth Jacob George Golo Ahlenstiel Javier Ampuero Margaret Bassendine Vincent W. S. Wong Chiara Rosso Rose White Lavinia Mezzabotta Vijayaprakash Suppiah Monika Michalk Barbara Malik Gail Matthews Tanya Applegate Jason Grebely Vincenzo Fragomeli Julie R. Jonsson Rosanna Santaro

Tissue fibrosis is a core pathologic process that contributes to mortality in ~45% of the population and is likely to be influenced by the host genetic architecture. Here we demonstrate, using liver disease as a model, that a single-nucleotide polymorphism (rs12979860) in the intronic region of interferon-λ4 (IFNL4) is a strong predictor of fibrosis in an aetiology-independent manner. In a coho...

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