نتایج جستجو برای: gsk3b

تعداد نتایج: 241  

Journal: :Experimental & Molecular Medicine 2021

2012
Yong-Won Kwon Il-Jin Kim Di Wu Jing Lu William A. Stock Yueyong Liu Yurong Huang Hio Chung Kang Reyno DelRosario Kuang-Yu Jen Jesus Perez-Losada Guangwei Wei Allan Balmain Jian-Hua Mao

The Aurora-A kinase gene is frequently amplified and/or overexpressed in a variety of human cancers, leading to major efforts to develop therapeutic agents targeting this pathway. Here, we show that Aurora-A is targeted for ubiquitination and subsequent degradation by the F-box protein FBXW7 in a process that is regulated by GSK3b. Using a series of truncated Aurora-A proteins and site-directed...

Journal: :The Journal of biological chemistry 2012
Shozo Itoh Taku Saito Makoto Hirata Masahiro Ushita Toshiyuki Ikeda James R Woodgett Hana Algül Roland M Schmid Ung-Il Chung Hiroshi Kawaguchi

Here we examine the roles of two isoforms of glycogen synthase kinase-3 (GSK-3), GSK-3α and GSK-3β, in skeletal development. Both isoforms were unphosphorylated and active in chondrocyte differentiation stages during SOX9 and type II collagen (COL2A1) expression. Although knock-out of both alleles of Gsk3a (Gsk3a(-/-)) or a single allele of Gsk3b (Gsk3b(+/-)) in mice did not significantly affec...

2017
Xijing He Liying Fan Zhongheng Wu Jiaxuan He Bin Cheng

Previous gene expression profiling studies of neuropathic pain (NP) following spinal cord injury (SCI) have predominantly been performed in animal models. The present study aimed to investigate gene alterations in patients with spinal cord injury and to further examine the mechanisms underlying NP following SCI. The GSE69901 gene expression profile was downloaded from the public Gene Expression...

2017
Shuji Wakatsuki Toshiyuki Araki

Axonal degeneration is a key pathological feature of several neurological disorders. Emerging evidence has suggested a pathological connection between axonal degeneration and autophagy, a lysosomal degradation pathway. We recently reported that GSK3B-mediated phosphorylation of MCL1 regulates axonal autophagy to promote axonal degeneration. GSK3B-MCL1 pathway affects ATP production locally in d...

Journal: :The Journal of clinical investigation 2008
Risto Kerkela Lisa Kockeritz Katrina Macaulay Jibin Zhou Bradley W Doble Cara Beahm Sarah Greytak Kathleen Woulfe Chinmay M Trivedi James R Woodgett Jonathan A Epstein Thomas Force Gordon S Huggins

Based on extensive preclinical data, glycogen synthase kinase-3 (GSK-3) has been proposed to be a viable drug target for a wide variety of disease states, ranging from diabetes to bipolar disorder. Since these new drugs, which will be more powerful GSK-3 inhibitors than lithium, may potentially be given to women of childbearing potential, and since it has controversially been suggested that lit...

Journal: :The Journal of clinical investigation 2008
Yosuke Kawasaki Fumitaka Kugimiya Hirotaka Chikuda Satoru Kamekura Toshiyuki Ikeda Naohiro Kawamura Taku Saito Yusuke Shinoda Akiro Higashikawa Fumiko Yano Toru Ogasawara Naoshi Ogata Kazuto Hoshi Franz Hofmann James R Woodgett Kozo Nakamura Ung-il Chung Hiroshi Kawaguchi

cGMP-dependent protein kinase II (cGKII; encoded by PRKG2) is a serine/threonine kinase that is critical for skeletal growth in mammals; in mice, cGKII deficiency results in dwarfism. Using radiographic analysis, we determined that this growth defect was a consequence of an elongated growth plate and impaired chondrocyte hypertrophy. To investigate the mechanism of cGKII-mediated chondrocyte hy...

2015
Carlos R Reis Ping-Hung Chen Saipraveen Srinivasan François Aguet Marcel Mettlen Sandra L Schmid

Clathrin-mediated endocytosis (CME) regulates signaling from the plasma membrane. Analysis of clathrin-coated pit (CCP) dynamics led us to propose the existence of a rate-limiting, regulatory step(s) that monitor the fidelity of early stages in CCP maturation. Here we show that nascent endocytic vesicles formed in mutant cells displaying rapid, dysregulated CME are defective in early endosomal ...

2013
Giselle Izzo Orestes V. Forlenza Bernardo dos Santos Paulo H. F. Bertolucci Elida B. Ojopi Wagner F. Gattaz Daniel Shikanai Kerr

In this study, we investigated the associations between single-nucleotide polymorphisms in GAB2 (rs2373115), GSK3B (rs6438552) and SORL1 (rs641120) and Alzheimer's disease (AD), both alone and in combination with the APOE*4 allele.

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