نتایج جستجو برای: foxp2

تعداد نتایج: 433  

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2015
Abidemi A Adegbola Gerald F Cox Elizabeth M Bradshaw David A Hafler Alexander Gimelbrant Andrew Chess

The recent descriptions of widespread random monoallelic expression (RMAE) of genes distributed throughout the autosomal genome indicate that there are more genes subject to RMAE on autosomes than the number of genes on the X chromosome where X-inactivation dictates RMAE of X-linked genes. Several of the autosomal genes that undergo RMAE have independently been implicated in human Mendelian dis...

Journal: :The Journal of neuroscience : the official journal of the Society for Neuroscience 2013
Zhimin Shi Guanzheng Luo Lijuan Fu Zhide Fang XiuJie Wang XiaoChing Li

Mutations in the FOXP2 gene cause speech and language impairments, accompanied by structural and functional abnormalities in brain regions underlying speech-related sensory-motor processing, including the striatum and cerebellum. The sequence and expression patterns of FOXP2 are highly conserved among higher vertebrates. In the zebra finch brain, FoxP2 is expressed in Area X, a striatal nucleus...

Journal: :Human molecular genetics 2010
Patrice Roll Sonja C Vernes Nadine Bruneau Jennifer Cillario Magali Ponsole-Lenfant Annick Massacrier Gabrielle Rudolf Manal Khalife Edouard Hirsch Simon E Fisher Pierre Szepetowski

It is a challenge to identify the molecular networks contributing to the neural basis of human speech. Mutations in transcription factor FOXP2 cause difficulties mastering fluent speech (developmental verbal dyspraxia, DVD), whereas mutations of sushi-repeat protein SRPX2 lead to epilepsy of the rolandic (sylvian) speech areas, with DVD or with bilateral perisylvian polymicrogyria. Pathophysiol...

Journal: :American journal of human genetics 2007
Elizabeth Spiteri Genevieve Konopka Giovanni Coppola Jamee Bomar Michael Oldham Jing Ou Sonja C Vernes Simon E Fisher Bing Ren Daniel H Geschwind

Mutations in FOXP2, a member of the forkhead family of transcription factor genes, are the only known cause of developmental speech and language disorders in humans. To date, there are no known targets of human FOXP2 in the nervous system. The identification of FOXP2 targets in the developing human brain, therefore, provides a unique tool with which to explore the development of human language ...

Journal: :Wiley Interdisciplinary Reviews: Cognitive Science 2013

Journal: :PLoS Biology 2007
Sebastian Haesler Christelle Rochefort Benjamin Georgi Pawel Licznerski Pavel Osten Constance Scharff

The gene encoding the forkhead box transcription factor, FOXP2, is essential for developing the full articulatory power of human language. Mutations of FOXP2 cause developmental verbal dyspraxia (DVD), a speech and language disorder that compromises the fluent production of words and the correct use and comprehension of grammar. FOXP2 patients have structural and functional abnormalities in the...

Journal: :The Journal of heredity 2005
D M Webb J Zhang

FoxP2 is the first identified gene that is specifically involved in speech and language development in humans. Population genetic studies of FoxP2 revealed a selective sweep in recent human history associated with two amino acid substitutions in exon 7. Avian song learning and human language acquisition share many behavioral and neurological similarities. To determine whether FoxP2 plays a simi...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2014
Christiane Schreiweis Ulrich Bornschein Eric Burguière Cemil Kerimoglu Sven Schreiter Michael Dannemann Shubhi Goyal Ellis Rea Catherine A French Rathi Puliyadi Matthias Groszer Simon E Fisher Roger Mundry Christine Winter Wulf Hevers Svante Pääbo Wolfgang Enard Ann M Graybiel

The acquisition of language and speech is uniquely human, but how genetic changes might have adapted the nervous system to this capacity is not well understood. Two human-specific amino acid substitutions in the transcription factor forkhead box P2 (FOXP2) are outstanding mechanistic candidates, as they could have been positively selected during human evolution and as FOXP2 is the sole gene to ...

2016
Kah Keng Wong Duncan M. Gascoyne Elizabeth J. Soilleux Linden Lyne Hayley Spearman Giovanna Roncador Lars M. Pedersen Michael B. Møller Tina M. Green Alison H. Banham

FOXP2 shares partially overlapping normal tissue expression and functionality with FOXP1; an established diffuse large B-cell lymphoma (DLBCL) oncogene and marker of poor prognosis. FOXP2 is expressed in the plasma cell malignancy multiple myeloma but has not been studied in DLBCL, where a poor prognosis activated B-cell (ABC)-like subtype display partially blocked plasma cell differentiation. ...

2017
Moritz Negwer Dirk Schubert

A commentary on FOXP2 drives neuronal differentiation by interacting with retinoic acid signaling pathways FOXP2 was the first identified monogenic cause of a speech disorder (for review see Graham et al., 2015). However, it remains to be answered how it affects the development of speech related neuronal circuits and what the relevant molecular pathways may be. Devanna et al. (2014) reported a ...

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