نتایج جستجو برای: dlbcl

تعداد نتایج: 2906  

Journal: :Hematological oncology 2015
Leticia Quintanilla-Martinez

Diffuse large B-cell lymphoma (DLBCL) is the most common subtype of non-Hodgkin lymphoma (NHL), representing around 30% to 40% of all newly diagnosed lymphomas [1]. DLBCL is clinically, morphologically and biologically a heterogeneous disease reflected in the highly variable clinical course. The 2008 World Health Organization (WHO) classification of lymphoid malignancies recognizes within the g...

2014
Harumi Kato Kennosuke Karube Kazuhito Yamamoto Jun Takizawa Shinobu Tsuzuki Yasushi Yatabe Teru Kanda Miyuki Katayama Yukiyasu Ozawa Kenji Ishitsuka Masataka Okamoto Tomohiro Kinoshita Koichi Ohshima Shigeo Nakamura Yasuo Morishima Masao Seto

Epstein-Barr virus (EBV)-positive diffuse large B-cell lymphoma (DLBCL) of the elderly (EBV[+]DLBCL-E) is classified as a subtype of DLBCL. Until now, its molecular pathogenesis has remained unknown. To identify pathways characteristic of EBV(+)DLBCL-E, gene expression profiling of five EBV(+)DLBCL-E and seven EBV-negative DLBCL (EBV[-]DLBCL) cases was undertaken using human oligonucleotide mic...

2013
Charusheila Ramkumar Hang Cui Yahui Kong Stephen N. Jones Rachel M. Gerstein Hong Zhang

About half of patients with diffuse large B-cell lymphoma (DLBCL) do not respond to or relapse soon after the standard chemotherapy, indicating a critical need to better understand the specific pathways perturbed in DLBCL for developing effective therapeutic approaches. Mice deficient in the E3 ubiquitin ligase Smurf2 spontaneously develop B-cell lymphomas that resemble human DLBCL with molecul...

Journal: :Sao Paulo medical journal = Revista paulista de medicina 2010
Abrahão Elias Hallack Neto Sheila Aparecida Coelho Siqueira Frederico Luiz Dulley Alfredo Chauobah Marcelo Belesso Rosaura Saboia Milton Artur Ruiz Dalton Alencar Fischer Chamone Juliana Pereira

CONTEXT AND OBJECTIVE Gene expression and immunohistochemical profiling of diffuse large B-cell lymphoma (DLBCL) have revealed important prognostic subgroups: germinal center B-cell-like (GCB-like) DLBCL and activated B cell-like (ABC-like) DLBCL. Although few reports on high-risk DLBCL are available, the prognosis for the GCB-like subgroup has been shown to be better than that of the ABC-like ...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2008
Georg Lenz George W Wright N C Tolga Emre Holger Kohlhammer Sandeep S Dave R Eric Davis Shannon Carty Lloyd T Lam A L Shaffer Wenming Xiao John Powell Andreas Rosenwald German Ott Hans Konrad Muller-Hermelink Randy D Gascoyne Joseph M Connors Elias Campo Elaine S Jaffe Jan Delabie Erlend B Smeland Lisa M Rimsza Richard I Fisher Dennis D Weisenburger Wing C Chan Louis M Staudt

Gene-expression profiling has been used to define 3 molecular subtypes of diffuse large B-cell lymphoma (DLBCL), termed germinal center B-cell-like (GCB) DLBCL, activated B-cell-like (ABC) DLBCL, and primary mediastinal B-cell lymphoma (PMBL). To investigate whether these DLBCL subtypes arise by distinct pathogenetic mechanisms, we analyzed 203 DLBCL biopsy samples by high-resolution, genome-wi...

Journal: :Hematological oncology 2013
Louis M Staudt

The activated B-cell-like (ABC) and germinal centre B-cell-like (GCB) subtypes of diffuse large B-cell lymphoma (DLBCL) were defined by their apparent derivation from different stages of B-cell differentiation and their differential response to chemotherapy [1]. With current chemotherapy supplemented with Rituximab, the ABC DLBCL subtype remains less curable [2,3], necessitating new approaches ...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2013
Matthias Pfeifer Michael Grau Dido Lenze Sören-Sebastian Wenzel Annette Wolf Brigitte Wollert-Wulf Kerstin Dietze Hendrik Nogai Benjamin Storek Hannelore Madle Bernd Dörken Martin Janz Stephan Dirnhofer Peter Lenz Michael Hummel Alexandar Tzankov Georg Lenz

Diffuse large B-cell lymphoma (DLBCL) represents a heterogeneous diagnostic category with distinct molecular subtypes that can be defined by gene expression profiling. However, even within these defined subtypes, heterogeneity prevails. To further elucidate the pathogenesis of these entities, we determined the expression of the tumor suppressor phosphatase and tensin homolog (PTEN) in 248 prima...

2014
KUNHAO WANG ZHIWEN XU NING WANG TING XU MINGHUI ZHU

Molecular biologists have collected considerable data regarding the involvement of genes and microRNAs (miRNAs) in cancer. However the underlying mechanisms of cancer with regard to genes and miRNAs remain unclear. The aim of the present study was to evaluate diffuse large B-cell lymphoma (DLBCL) and construct regulatory networks of genes and miRNAs to gradually reveal the underlying mechanisms...

Journal: :Blood 2005
Silvia Bea Andreas Zettl George Wright Itziar Salaverria Philipp Jehn Victor Moreno Christof Burek German Ott Xavier Puig Liming Yang Armando Lopez-Guillermo Wing C Chan Timothy C Greiner Dennis D Weisenburger James O Armitage Randy D Gascoyne Joseph M Connors Thomas M Grogan Rita Braziel Richard I Fisher Erlend B Smeland Stein Kvaloy Harald Holte Jan Delabie Richard Simon John Powell Wyndham H Wilson Elaine S Jaffe Emili Montserrat Hans-Konrad Muller-Hermelink Louis M Staudt Elias Campo Andreas Rosenwald

Gene-expression profiling has identified 3 major subgroups of diffuse large B-cell lymphoma (DLBCL): germinal center B-cell-like (GCB), activated B-cell-like (ABC), and primary mediastinal DLBCL (PMBCL). Using comparative genomic hybridization (CGH), we investigated the genetic alterations of 224 cases of untreated DLBCL (87 GCB-DLBCL, 77 ABC-DLBCL, 19 PMBCL, and 41 unclassified DLBCL) previous...

Journal: :The Journal of Experimental Medicine 2001
R. Eric Davis Keith D. Brown Ulrich Siebenlist Louis M. Staudt

Gene expression profiling has revealed that diffuse large B cell lymphoma (DLBCL) consists of at least two distinct diseases. Patients with one DLBCL subtype, termed activated B cell-like (ABC) DLBCL, have a distinctly inferior prognosis. An untapped potential of gene expression profiling is its ability to identify pathogenic signaling pathways in cancer that are amenable to therapeutic attack....

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