نتایج جستجو برای: tp53 protein

تعداد نتایج: 1240012  

2018
Yanli Luo Wentao Huang Huizhen Zhang Guang Liu

Triple-negative breast cancer (TNBC) is extremely aggressive and associated with poor prognosis. There are no known predictive or prognostic markers for TNBC. Inhibition of tumor protein P53 (TP53) has been demonstrated to increase the levels of cluster of differentiation 117 (CD117) in human colorectal cancer cells. However, the function of TP53 in the regulation of CD117 in TNBC has, to the b...

Journal: :Clinical cancer research : an official journal of the American Association for Cancer Research 2005
Koert P de Jong Annette S H Gouw Paul M J G Peeters Marian Bulthuis Lorian Menkema Robert J Porte Maarten J H Slooff Harry van Goor Anke van den Berg

PURPOSE To correlate TP53 mutations with angiogenic status of the tumor and prognosis after liver surgery in patients with colorectal liver metastases and to correlate immunohistochemical staining of p53 protein with TP53 gene mutations. EXPERIMENTAL DESIGN Tumors of 44 patients with surgically treated colorectal liver metastases were analyzed for (a) TP53 mutations using denaturing gradient ...

Journal: :Experimental oncology 2014
N I Bilous I V Abramenko A A Chumak I S Dyagil Z V Martіna

UNLABELLED Defects in the tumor suppressor gene TP53 are known to be important in chronic lymphocytic leukemia (CLL) and TP53 inactivation is associated with a particularly aggressive form of the disease. The single nucleotide polymorphism in the TP53 gene at codon 72 (rs1042522), results in amino acid substitution influencing apoptotic potential of TP53 protein. The aim of the study was to eva...

Journal: :Carcinogenesis 2005
Leah E Mechanic Aizen J Marrogi Judith A Welsh Elise D Bowman Mohammed A Khan Lindsey Enewold Yun-Ling Zheng Stephen Chanock Peter G Shields Curtis C Harris

The pattern of somatic mutations in TP53 is distinct for particular cancers and carcinogenic exposures, providing clues to disease etiology, e.g. G:C-->T:A mutations in TP53 are more frequently observed in smoking-associated lung cancers. In order to investigate possible causes and mechanisms of lung cancer susceptibility differences, the TP53 gene was sequenced in a case-only study of lung can...

2014
Edward A Ratovitski

Tumor protein (TP)-p63 has been discovered as TP53 homolog more than fifteen years ago and has become a master regulator of skin development, proliferation and stem cell maintenance. While TP53 is known to be the most mutated gene in human cancer, TP63 mutations are mostly associated with the various types of ectodermal dysplasia. All TP53 family members, TP53, TP63 and TP73, function as transc...

Journal: :Oncology reports 2013
Jiang Long Yan Wang Man Li Wei-Min Tong Ji-Dong Jia Jian Huang

Although it is known that chronic hepatitis C virus (HCV) infection may contribute to tumor initiation and development, the molecular processes causing hepatocellular carcinoma (HCC) remain unclear. Microindels are unique, infrequent mutations that result in inserted and deleted sequences at the same nucleotide position, and are important contributors to cancer. To date, microindels in the p53 ...

Journal: :cell journal 0

introduction: the tumor suppressor p53 protein can induce apoptosis in some cellular contexts. among the apoptosis-inducing genes, p53 has received the most attention for cancer gene therapy. in this study, the role of dendrosome and lipofectin mediated normal cdna of tp53 into molt-4, ccrf-cem(t-iymphoma) and k562 cell lines was assessed. material and methods: at first, ccrf-cem, mol t-4 and k...

Journal: :Clinical cancer research : an official journal of the American Association for Cancer Research 2002
Aurelia Peraud Friedrich W Kreth Otmar D Wiestler Paul Kleihues Hans-Jürgen Reulen

PURPOSE The purpose of this study was to analyze retrospectively timing, frequency, and prognostic impact of TP53 mutations and P53 protein accumulation in supratentorial WHO grade II astrocytomas and oligoastrocytomas of adult patients. EXPERIMENTAL DESIGN We included 159 consecutively treated patients (1991-1998), and each tumor was screened for TP53 mutations and P53 protein overexpression...

Journal: :Cancer research 2009
Henne Holstege Simon A Joosse Conny Th M van Oostrom Petra M Nederlof Annemieke de Vries Jos Jonkers

Approximately half of all hereditary breast cancers are compromised in their DNA repair mechanisms due to loss of BRCA1 or BRCA2 function. Previous research has found a strong correlation between BRCA mutation and TP53 mutation. However, TP53 mutation status is often indirectly assessed by immunohistochemical staining of accumulated p53 protein. We sequenced TP53 exons 2 to 9 in 21 BRCA1-relate...

2016
George Fountzilas Eleni Giannoulatou Zoi Alexopoulou Flora Zagouri Eleni Timotheadou Kyriaki Papadopoulou Sotiris Lakis Mattheos Bobos Christos Poulios Maria Sotiropoulou Aggeliki Lyberopoulou Helen Gogas George Pentheroudakis Dimitrios Pectasides Angelos Koutras Christos Christodoulou Christos Papandreou Epaminontas Samantas Pavlos Papakostas Paris Kosmidis Dimitrios Bafaloukos Charisios Karanikiotis Meletios-Athanassios Dimopoulos Vassiliki Kotoula

BACKGROUND We investigated the impact of PIK3CA and TP53 mutations and p53 protein status on the outcome of patients who had been treated with adjuvant anthracycline-taxane chemotherapy within clinical trials in the pre- and post-trastuzumab era. RESULTS TP53 and PIK3CA mutations were found in 380 (21.5%) and 458 (25.9%) cases, respectively, including 104 (5.9%) co-mutated tumors; p53 immunop...

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