نتایج جستجو برای: med12 mutations

تعداد نتایج: 172942  

Journal: :American journal of cancer research 2015
Shirin Shahbazi Neda Fatahi Soheila Amini-Moghaddam

Uterine leiomyomas are steroid-hormone dependent tumors of myometrial smooth muscle cells that affect numerous women throughout the world. Based on previous studies, we evaluated the mutations of MED12 gene which encodes a co-activator protein involved in transcription regulation of the vast majority of RNA polymerase II-dependent genes. Exon 2 of MED12 gene was genotyped by PCR-sequencing meth...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2006
Xiaoqun Wang Nan Yang Etsuko Uno Robert G Roeder Su Guo

The unique profiles of gene expression dictate distinct cellular identity. How these profiles are established during development is not clear. Here we report that the mutant motionless (mot), identified in a genetic screen for mutations that affect neuronal development in zebrafish, displays deficits of monoaminergic neurons and cranial sensory ganglia, whereas expression of the pan-neuronal ma...

2011
Netta Mäkinen Hanna-Riikka Heinonen Shane Moore Ian P.M. Tomlinson Zephne M. van der Spuy Lauri A. Aaltonen

Uterine leiomyomas, or fibroids, are extremely common tumors. Regardless of their benign nature, fibroids can cause considerable morbidity. Women with African ancestry have a threefold increased risk of developing uterine leiomyomas with a greater symptom severity when compared to white women. Recently, we demonstrated that exon 2 of the MED12 gene is somatically altered in up to 70 per cent of...

2016
Yoshifumi Shimada Yosuke Tajima Hitoshi Kameyama Ryoma Yagi Takuma Okamura Yuki Hirose Jun Sakata Takashi Kobayashi Yasunobu Matsuda Yoichi Ajioka Shin-ichi Kosugi Toshifumi Wakai

MED12 is a transcriptional mediator complex subunit, which negatively regulates the transforming growth factor β (TGF-β) pathway. The TGF-β pathway plays a major role in the induction of epithelial-mesenchymal transition (EMT). MED12 loss induces activation of the TGF-β pathway, resulting in EMT and drug resistance to epidermal growth factor receptor (EGFR)-targeted therapy. We aimed to investi...

Journal: :JCI insight 2017
Kedryn K Baskin Catherine A Makarewich Susan M DeLeon Wenduo Ye Beibei Chen Nadine Beetz Heinrich Schrewe Rhonda Bassel-Duby Eric N Olson

The Mediator complex regulates gene transcription by linking basal transcriptional machinery with DNA-bound transcription factors. The activity of the Mediator complex is mainly controlled by a kinase submodule that is composed of 4 proteins, including MED12. Although ubiquitously expressed, Mediator subunits can differentially regulate gene expression in a tissue-specific manner. Here, we repo...

Journal: :Frontiers in pharmacology 2016
Qiwei Yang Michael P. Diamond Ayman Al-Hendy

Uterine Fibroids [UF(s), AKA: leiomyoma] are the most important benign neoplastic threat to women's health. They are the most common cause of hysterectomy imposing untold personal consequences and 100s of billions of healthcare dollars, worldwide. Currently, there is no long term effective FDA-approved medical treatment available, and surgery is the mainstay. The etiology of UFs is not fully un...

2016
Netta Mäkinen Mervi Aavikko Tuomas Heikkinen Minna Taipale Jussi Taipale Riitta Koivisto-Korander Ralf Bützow Pia Vahteristo Cynthia C. Morton

Uterine leiomyosarcomas (ULMSs) are aggressive smooth muscle tumors associated with poor clinical outcome. Despite previous cytogenetic and molecular studies, their molecular background has remained elusive. To examine somatic variation in ULMS, we performed exome sequencing on 19 tumors. Altogether, 43 genes were mutated in at least two ULMSs. Most frequently mutated genes included tumor prote...

2015
Habib Bouazzi Gaetan Lesca Carlos Trujillo Mohammad Khalid Alwasiyah Arnold Munnich

X-linked intellectual deficiency (XLID) is a large group of genetic disorders. MED12 gene causes syndromic and nonsyndromic forms of XLID. Only seven pathological mutations have been identified in this gene. Here, we report a novel mutation segregating with XLID phenotype. This mutation could be in favor of genotype-phenotype correlations.

Journal: :Molecular cancer research : MCR 2015
Andi K Cani Daniel H Hovelson Andrew S McDaniel Seth Sadis Michaela J Haller Venkata Yadati Anmol M Amin Jarred Bratley Santhoshi Bandla Paul D Williams Kate Rhodes Chia-Jen Liu Michael J Quist Daniel R Rhodes Catherine S Grasso Celina G Kleer Scott A Tomlins

UNLABELLED Phyllodes tumors are rare fibroepithelial tumors with variable clinical behavior accounting for a small subset of all breast neoplasms, yet little is known about the genetic alterations that drive tumor initiation and/or progression. Here, targeted next-generation sequencing (NGS) was used to identify somatic alterations in formalin-fixed paraffin-embedded (FFPE) patient specimens fr...

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