نتایج جستجو برای: hibm

تعداد نتایج: 35  

Journal: :The Journal of clinical investigation 2007
Belinda Galeano Riko Klootwijk Irini Manoli MaoSen Sun Carla Ciccone Daniel Darvish Matthew F Starost Patricia M Zerfas Victoria J Hoffmann Shelley Hoogstraten-Miller Donna M Krasnewich William A Gahl Marjan Huizing

Mutations in the key enzyme of sialic acid biosynthesis, uridine diphospho-N-acetylglucosamine 2-epimerase/N-acetylmannosamine (ManNAc) kinase (GNE/MNK), result in hereditary inclusion body myopathy (HIBM), an adult-onset, progressive neuromuscular disorder. We created knockin mice harboring the M712T Gne/Mnk mutation. Homozygous mutant (Gne(M712T/M712T)) mice did not survive beyond P3. At P2, ...

2013
Angèle Nalbandian Katrina J. Llewellyn Mallikarjun Badadani Hong Z. Yin Christopher Nguyen Vincent Caiozzo Tahseen Mozaffar

Introduction—Mutations in the valosin containing protein (VCP) gene cause hereditary Inclusion Body Myopathy (hIBM) associated with Paget disease of bone (PDB), and frontotemporal dementia (FTD). More recently they have been linked to 2% of familial ALS cases. A knock-in mouse model offers the opportunity to study VCP-associated pathogenesis. Methods—The VCPR155H/+ knock-in mouse model was asse...

Journal: :Current opinion in neurology 2008
May Christine V Malicdan Satoru Noguchi Ichizo Nishino

PURPOSE OF REVIEW Distal myopathy with rimmed vacuoles or hereditary inclusion body myopathy is an adult-onset autosomal recessive, slowly progressive and debilitating myopathy due to mutations in the gene that regulates the synthesis of sialic acid. This review aims to update our knowledge of this myopathy and to review studies about pathomechanism and therapeutic strategies. RECENT FINDINGS...

Journal: :Journal of medical and dental sciences 2008
Shoichiro Ishihara Hiroyuki Tomimitsu Hiroto Fujigasaki Fumiaki Saito Hidehiro Mizusawa

BACKGROUND UDP-N-acetylglucosamine 2-epimerase/N-acetylmannosamine kinase (GNE) is a key molecule in the pathogenesis of distal myopathy with rimmed vacuoles (DMRV) and hereditary inclusion body myopathy (HIBM) and almost all such patients have some mutations in GNE. However, subcellular localization of GNE and the mechanism of muscular damage have not been clarified. METHODS A rabbit polyclo...

Journal: :PLoS ONE 2008
Agnès Delaunay Kenneth D. Bromberg Yukiko Hayashi Massimiliano Mirabella Denise Burch Brian Kirkwood Carlo Serra May C. Malicdan Andrew P. Mizisin Roberta Morosetti Aldobrando Broccolini Ling T. Guo Stephen N. Jones Sergio A. Lira Pier Lorenzo Puri G. Diane Shelton Ze'ev Ronai

Growing evidence supports the importance of ubiquitin ligases in the pathogenesis of muscular disorders, although underlying mechanisms remain largely elusive. Here we show that the expression of RNF5 (aka RMA1), an ER-anchored RING finger E3 ligase implicated in muscle organization and in recognition and processing of malfolded proteins, is elevated and mislocalized to cytoplasmic aggregates i...

Journal: :Physiological genomics 2008
May Christine V Malicdan Satoru Noguchi Yukiko K Hayashi Ichizo Nishino

Distal myopathy with rimmed vacuoles (DMRV), also called hereditary inclusion body myopathy (hIBM), is characterized clinically by weakness and atrophy that initially involves the distal muscles and pathologically by the presence of rimmed vacuoles (RVs) or intracellular protein deposits in myofibers. It is caused by mutations in the UDP-N-acetylglucosamine 2-epimerase/N-acetylmannosamine kinas...

2012
Yan Lu Xingang Li Min Wang Xin Li Feng Zhang Yun Li Meng Zhang Yuwei Da Jun Yu Jianping Jia

We describe a novel autosomal dominant hereditary inclusion body myopathy (HIBM) that clinically mimics limb girdle muscular dystrophy in a Chinese family. We performed a detailed clinical assessment of 36 individuals spanning four generations. The age of onset ranged from the 30s to the 50s. Hip girdle, neck flexion and axial muscle weakness were involved at an early stage. This disease progre...

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