نتایج جستجو برای: cardiac dysfunction

تعداد نتایج: 409621  

خلیق فرد, سمانه, نظرعلی, پروانه , گائینی, عباسعلی,

  Introduction : Biochemical and immunological investigations have indicated cardiac dysfunction and damage with changes in cardiac markers and inflammatory cytokines after endurance exercise.Therefore, there is a rational background to study the effect of cardiac pathophysiology during endurance exercise.This study was undertaken to determine the effect of endurance exercise on cardiac and inf...

Journal: :international journal of endocrinology and metabolism 0
efren martinez-quintana cardiology service, insular-materno infantil university hospital, las palmas de gran canaria, spain; cardiology service, insular materno-infantil university hospital, 35016, las palmas de gran canaria, spain. tel:+92-8441360 beatriz saiz-udaeta cardiology service, insular-materno infantil university hospital, las palmas de gran canaria, spain natalia marrero-negrin cardiology service, insular-materno infantil university hospital, las palmas de gran canaria, spain xavier lopez-mérida endocrinology service, insular-materno infantil university hospital, las palmas de gran canaria, spain fayna rodriguez-gonzalez ophthalmology service, dr. negrin university hospital, las palmas de gran canaria, spain vicente nieto-lago cardiology service, insular-materno infantil university hospital, las palmas de gran canaria, spain

introduction anabolic-androgenic steroids (aas), a synthetic derivate of testosterone, have become a popular drug among athletes and bodybuilders to enhance muscle mass and improve the athletic performance. many pathological effects such as hepatic and endocrine dysfunction, behavioural changes and cardiovascular complications have been reported. case report within these ast ones, we find an in...

Journal: :the journal of tehran university heart center 0
saba ghaffary department of pharmacotherapy, faculty of pharmacy, tehran university of medical sciences, tehran, iran. azita hajhossein-talasaz department of pharmacotherapy, faculty of pharmacy, tehran university of medical sciences, tehran, iran and tehran heart centre, tehran university of medical sciences, tehran, iran. padideh ghaeli department of pharmacotherapy, faculty of pharmacy, tehran university of medical sciences, tehran, iran. abbasali karimi tehran heart centre, tehran university of medical sciences, tehran, iran. abbas salehiomran tehran heart centre, tehran university of medical sciences, tehran, iran. alireza hajighasemi tehran heart centre, tehran university of medical sciences, tehran, iran.

background: postoperative cognitive dysfunction (pocd) has been an important complication of cardiac surgery over the years. neurocognitive dysfunction can affect quality of life and lead to social, functional, emotional, and financial problems in the patient’s life. to reduce pocd, we sought to identify the association between cognitive dysfunction and perioperative factors in patients undergo...

Journal: :Circulation Research 2015

Journal: :Circulation: Genomic and Precision Medicine 2018

Journal: :Endocrinology and Metabolism 2017

Journal: :Interactive CardioVascular and Thoracic Surgery 2015

Journal: :Circulation 2010
Gad A Silberman Tai-Hwang M Fan Hong Liu Zhe Jiao Hong D Xiao Joshua D Lovelock Beth M Boulden Julian Widder Scott Fredd Kenneth E Bernstein Beata M Wolska Sergey Dikalov David G Harrison Samuel C Dudley

BACKGROUND Heart failure with preserved ejection fraction is 1 consequence of hypertension and is caused by impaired cardiac diastolic relaxation. Nitric oxide (NO) is a known modulator of cardiac relaxation. Hypertension can lead to a reduction in vascular NO, in part because NO synthase (NOS) becomes uncoupled when oxidative depletion of its cofactor tetrahydrobiopterin (BH(4)) occurs. Simila...

2010
Gad A. Silberman Zhe Jiao Beata M. Wolska David G. Harrison

Background—Heart failure with preserved ejection fraction is 1 consequence of hypertension and is caused by impaired cardiac diastolic relaxation. Nitric oxide (NO) is a known modulator of cardiac relaxation. Hypertension can lead to a reduction in vascular NO, in part because NO synthase (NOS) becomes uncoupled when oxidative depletion of its cofactor tetrahydrobiopterin (BH 4) occurs. Similar...

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