Bilateral vocal cord palsy is one of the causes that trigger nocturnal stridor and obstructive sleep apnea (OSA), and three cases of OSA due to bilateral vocal cord paralysis have been described in the medical literature. In 1987, Ruff et al.1 reported a case of bilateral vocal cord palsy and sleep apnea in a patient with type I Chiari malformation. In 1996, McBrien et al.2 reported a case of b...

We wish to thank Dr. Wilson for suggesting that the bilateral vocal cord paralysis we reported in a patient with Guillain Barre syndrome may have resulted from trauma secondary to intubation with a cuffinflated endotracheal tube. Although this explanation could have contributed to the paralysis, we believe it was primarily the result of the Guillain Barr6 syndrome because: 1. Prior to intubatio...

IMPORTANCE Treatment of bilateral vocal cord paralysis is a considerable challenge for otolaryngologists. Many surgical techniques have been developed for the management of this entity to eliminate the need for tracheotomy. OBJECTIVE To evaluate the success of the unilateral carbon dioxide laser endoscopic posterior cordotomy technique for bilateral abductor vocal cord paralysis. DESIGN A r...

A patient with bilateral vocal cord paralysis developed chronic respiratory failure. Treatment with nocturnal inspiratory positive airway pressure via nasal mask improved symptoms and reduced hypercapnia.

Aim of the study was the assessment of total thyroidectomy (TT) value as a treatment method of goiter with particular emphasis on the results of laryngological examination. material and methods. From 15th April 2008 to 1st July 2009 there were 125 (72.7%) TTs, 12 (7%) TTs and appropriate lymphadenectomies, 9 (5.2%) near TTs, 14 (8.1%) lobectomies, 5 (2.9%) completion thyroidectomies and 6 (3.4%...

Familial bilateral abductor vocal cord paralysis is a rare entity with few prior descriptions in the literature. Modes of inheritance include X-linked, autosomal recessive, and autosomal dominant. A case of this condition in a father and son is presented. Signs and symptoms at presentation, diagnosis, therapeutic considerations, and modes of inheritance are discussed.

A 38-year-old man with posttraumatic bilateral vocal cord paralysis and a surgically repaired avulsion of the extrathoracic trachea presented with a slight increase of exertional dyspnea (grade 2). Spirometry showed high normal FEV1 for FVC variables, but the F-V loop was characteristic for highly variable UAO with an increased FEV1/PEF ratio of 11 ml/L/min as well as a MEF50/MIF50 of 4.55. End...