نتایج جستجو برای: apolipoprotein e
تعداد نتایج: 1029260 فیلتر نتایج به سال:
The PLOS ONE Staff Figure 3 is incorrect. The first p-value should be above the second column, not the first. Please see the correct figure below. Copyright: ß 2014 The PLOS ONE Staff. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author a...
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OBJECTIVE To investigate the heritability of memory and other cognitive measures in families with multiple individuals with Alzheimer disease (AD) to determine if neuropsychological measures can be used to better understand genetic contributions to AD. METHODS The genetic contributions to the variation in declarative memory, attention, abstract reasoning, language, and visuospatial function u...
National Public Health Institute, Helsinki, Finland; Department of Human Genetics, University of Michigan, Ann Arbor, MI, USA; Department of Epidemiology, University of Michigan, Ann Arbor, MI, USA; Department of Clinical Biochemistry, Rigshospitalet, Copenhagen University Hospital, Copenhagen, Denmark; Human Genetics Center and Institute of Molecular Medicine, The University of Texas Health Sc...
2007;69;1921-1930 Neurology A. Alpérovitch P. Barberger-Gateau, C. Raffaitin, L. Letenneur, C. Berr, C. Tzourio, J. F. Dartigues and Dietary patterns and risk of dementia: The Three-City cohort study This information is current as of November 12, 2007 http://www.neurology.org/cgi/content/full/69/20/1921 located on the World Wide Web at: The online version of this article, along with updated inf...
Apolipoprotein E (apoE) and clusterin can influence structure, toxicity, and accumulation of the amyloid-beta (Abeta) peptide in brain. Both molecules may also be involved in Abeta metabolism prior to its deposition. To assess this possibility, we compared PDAPP transgenic mice that develop age-dependent Abeta accumulation in the absence of apoE or clusterin as well as in the absence of both pr...
The apolipoprotein E ε4 gene is the most important genetic risk factor for sporadic Alzheimer's disease, but the link between this gene and neurodegeneration remains unclear. Using array tomography, we analysed >50000 synapses in brains of 11 patients with Alzheimer's disease and five non-demented control subjects and found that synapse loss around senile plaques in Alzheimer's disease correlat...
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