نتایج جستجو برای: aoa1

تعداد نتایج: 38  

Journal: :iranian journal of child neurology 0
parvaneh karimzadeh 1. pediatric neurology research center, shahid beheshti university of medical sciences, tehran, iran 2. pediatric neurology department, mofid children’s hospital, faculty of medicine, shahid beheshti university of medical sciences, tehran, iran simin khayyatzadeh 1. pediatric neurology research center, shahid beheshti university of medical sciences, tehran, iran masoud houshmand 3. department of medical genetic, national institute for genetic engineering and biotechnology(nigeb), tehran, iran mohammad ghforani 1. pediatric neurology research center, shahid beheshti university of medical sciences, tehran, iran 2. pediatric neurology department, mofid children’s hospital, faculty of medicine, shahid beheshti university of medical sciences, tehran, iran

ataxia oculomotor apraxia1(aoa1) is the most frequent cause of autosomal-recessive cerebellar ataxia in japan,but it is reported from all of the world. the presentation is nearly identical to that of at without the non-neurological features,and accounts for up to 10% of autosomal-recessive cerebellar ataxias.gait imbalance and dysarthria are typical presenting features,oculomotor apraxia typica...

2013
John J. Reynolds Grant S. Stewart

Sir, We are grateful for the opportunity to respond to the correspondence from Oegema et al. (2013) and we thank the authors for their interest in our recent review article discussing the three known neurological disorders associated with defective single strand break repair, namely ataxia oculomotor apraxia 1 (AOA1), spinocerebellar ataxia with neuronal neuropathy 1 (SCAN1) and microcephaly, e...

2017
Humera Manzoor Ihtisham Bukhari Muhammad Wajid Yuanwei Zhang Huan Zhang Norbert Brüggemann Christine Klein Qinghua Shi Sadaf Naz

Dear Editor, Ataxia with oculomotor apraxia type 1 (AOA1, MIM 208920) is an autosomal recessive disorder characterized by early-onset cerebellar ataxia, polyneuropathy, oculomotor apraxia, hypoalbuminemia, and hypercholesterolemia.1 It is caused by pathogenic variants of APTX, which encodes the aprataxin protein that is involved in DNA strand-break repair.1 We recruited a consanguineous family ...

Journal: :Iranian biomedical journal 2012
Nayereh Nouri Narges Nouri Omid Aryani Behnam Kamalidehghan Maryam Sedghi Massoud Houshmand

BACKGROUND Ataxia with oculomotor apraxia type 1 (AOA1) shows early onset with autosomal recessive inheritance and is caused by a mutation in the aprataxin (APTX) gene encoding for the APTX protein. METHODS In this study, a 7-year-old girl born of a first-cousin consanguineous marriage was described with early-onset progressive ataxia and AOA, with increased cholesterol concentration and decr...

2013
Lorenzo Nanetti Simona Cavalieri Viviana Pensato Alessandra Erbetta Davide Pareyson Marta Panzeri Giovanna Zorzi Carlo Antozzi Isabella Moroni Cinzia Gellera Alfredo Brusco Caterina Mariotti

OBJECTIVES/BACKGROUND Ataxia with oculomotor apraxia defines a group of genetically distinct recessive ataxias including ataxia-telangectasia (A-T, ATM gene), ataxia with oculomotor apraxia type 1 (AOA1, APTX gene) and type 2 (AOA2, SETX gene). Although, a few unique clinical features differentiate each of these forms, the patients also share common clinical signs, such as the presence of cereb...

Behnam Kamalidehghan, Maryam Sedghi, Massoud Houshmand, Narges Nouri, Nayereh Nouri, Omid Aryani,

Background: Ataxia with oculomotor apraxia type 1 (AOA1) shows early onset with autosomal recessive inheritance and is caused by a mutation in the aprataxin (APTX) gene encoding for the APTX protein. Methods: In this study, a 7-year-old girl born of a first-cousin consanguineous marriage was described with early-onset progressive ataxia and AOA, with increased cholesterol concentration and decr...

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