Reconfigurable intelligent surface (RIS) is more likely to develop into extremely large-scale RIS (XL-RIS) efficiently boost the system capacity for future 6G communications. Beam training an effective way acquire channel state information (CSI) XL-RIS. Existing beam schemes rely on far-field codebook. However, due large aperture of XL-RIS, scatters are be in near-field region The codebook mism...

We begin by pointing out that, under this setup, it is without loss of generality to assume that the Voter ‘knows’ the Legislator’s random variable xl : Ω → R. To see this, let x∗v : Ω → R and xl : Ω → R be the actual random variables of the Voter and Legislator. Append to the game a set of mappings xl : Ω → R, viz. Xl, where Xl is a metric space. Also, append to the game a (transparent) prior ...

Introduction: Memory impairment is one of the complications of diabetes which may accompany with changes in expression of apoptotic and antiapoptotic genes. The aim of the present study was the evaluation of intra-hippocampal injection of aminoguanidine (AG), as an antioxidant and inducible nitric oxide synthase inhibitor, on passive avoidance memory and Bcl-2 family genes expression in diab...

Algebraic attacks are studied as a potential cryptanalytic procedure for various types of ciphers. The XL SGE algorithm has been recently proposed to improve the complexity of the XL attack. XL SGE uses structured Gaussian elimination (SGE) during the expansion phase of XL. In this paper, we establish that XL SGE suffers from some serious drawbacks that impair the effectiveness of SGE-based red...

INrf2 (Keap1) is an adaptor protein that facilitates INrf2-Cul3-Rbx1-mediated ubiquitination/degradation of Nrf2, a master regulator of cytoprotective gene expression. Here, we present evidence that members of the phosphoglycerate mutase family 5 (PGAM5) proteins are involved in the INrf2-mediated ubiquitination/degradation of anti-apoptotic factor Bcl-xL. Mass spectrometry and co-immunoprecipi...

The effect of Bcl-xL upon the developmental death of T cells was assessed by generating transgenic mice that expressed Bcl-xL within all thymocyte subsets. Bcl-xL protected thymocytes from a variety of apoptotic stimuli, including gamma irradiation, glucocorticoids, and anti-CD3 treatment. Bcl-xL altered thymocyte maturation, resulting in increased numbers of CD3int/hi and CD4-8+ thymocytes. Ov...