نتایج جستجو برای: ژن myh7

تعداد نتایج: 16107  

2013
K. van Engelen A. V. Postma J. B. A. van de Meerakker J. W. Roos-Hesselink A. T. J. M. Helderman-van den Enden H. W. Vliegen T. Rahman M. J. H. Baars J-W Sels U. Bauer T. Pickardt S. R. Sperling A. F. M. Moorman B. Keavney J. Goodship S. Klaassen B. J. M. Mulder

Ebstein's anomaly is a rare congenital heart malformation characterised by adherence of the septal and posterior leaflets of the tricuspid valve to the underlying myocardium. Associated abnormalities of left ventricular morphology and function including left ventricular noncompaction (LVNC) have been observed. An association between Ebstein's anomaly with LVNC and mutations in the sarcomeric pr...

2014
Ricardo H Roda Alice B Schindler Craig Blackstone Andrew L Mammen Andrea M Corse Thomas E Lloyd

Mutations in MYH7 cause autosomal dominant Laing distal myopathy. We present a family with a previously reported deletion (c.5186_5188delAGA, p.K1729del). Muscle pathology in one family member was characterized by an inflammatory myopathy with rimmed vacuoles, increased MHC Class I expression, and perivascular and endomysial muscle inflammation comprising CD3(+), CD4(+), CD8(+), and CD68(+) inf...

Journal: :Nucleic Acids Research 1990

Journal: :Revista portuguesa de cardiologia : orgao oficial da Sociedade Portuguesa de Cardiologia = Portuguese journal of cardiology : an official journal of the Portuguese Society of Cardiology 2012
Dulce Brito Gabriel Miltenberger-Miltenyi Sónia Vale Pereira Doroteia Silva António Nunes Diogo Hugo Madeira

BACKGROUND Sarcomeric hypertrophic cardiomyopathy has heterogeneous phenotypic expressions, of which sudden cardiac death is the most feared. A genetic diagnosis is essential to identify subjects at risk in each family. The spectrum of disease-causing mutations in the Portuguese population is unknown. METHODS Seventy-seven unrelated probands with hypertrophic cardiomyopathy were systematicall...

2016
C. Fiorillo G. Astrea M. Savarese D. Cassandrini G. Brisca F. Trucco M. Pedemonte R. Trovato L. Ruggiero L. Vercelli A. D’Amico G. Tasca M. Pane M. Fanin L. Bello P. Broda O. Musumeci C. Rodolico S. Messina G. L. Vita M. Sframeli S. Gibertini L. Morandi M. Mora L. Maggi A. Petrucci R. Massa M. Grandis A. Toscano E. Pegoraro E. Mercuri E. Bertini T. Mongini L. Santoro V. Nigro C. Minetti F. M. Santorelli C. Bruno

BACKGROUND Myosin heavy chain 7 (MYH7)-related myopathies are emerging as an important group of muscle diseases of childhood and adulthood, with variable clinical and histopathological expression depending on the type and location of the mutation. Mutations in the head and neck domains are a well-established cause of hypertrophic cardiomyopathy whereas mutation in the distal regions have been a...

Journal: :Biochemical and biophysical research communications 2012
Min Wang Hui Yu Yong Soo Kim Christopher A Bidwell Shihuan Kuang

Skeletal muscles in the limb and body trunk are composed of heterogeneous myofibers expressing different isoforms of myosin heavy chain (Myh), including type I (slow, Myh7), IIA (intermediate, Myh2), IIX (fast, Myh1), and IIB (very fast, Myh4). While the contraction force and speed of a muscle are known to be determined by the relative abundance of myofibers expressing each Myh isoform, it is u...

Journal: :Genetic testing and molecular biomarkers 2016
Nawel Jaafar Juan Gómez Ikram Kammoun Ihsen Zairi Wael Ben Amara Salem Kachboura Sondes Kraiem Mohamed Hammami Sara Iglesias Belén Alonso Eliecer Coto

BACKGROUND Hypertrophic cardiomyopathy (HCM) is a common cardiac genetic disorder associated with heart failure and sudden death. Mutations in the cardiac sarcomere genes are found in approximately half of HCM patients and are more common among cases with a family history of the disease. Data about the mutational spectrum of the sarcomeric genes in HCM patients from Northern Africa are limited....

Journal: :Circulation 2003
Pascale Richard Philippe Charron Lucie Carrier Céline Ledeuil Theary Cheav Claire Pichereau Abdelaziz Benaiche Richard Isnard Olivier Dubourg Marc Burban Jean-Pierre Gueffet Alain Millaire Michel Desnos Ketty Schwartz Bernard Hainque Michel Komajda

BACKGROUND Hypertrophic cardiomyopathy is an autosomal-dominant disorder in which 10 genes and numerous mutations have been reported. The aim of the present study was to perform a systematic screening of these genes in a large population, to evaluate the distribution of the disease genes, and to determine the best molecular strategy in clinical practice. METHODS AND RESULTS The entire coding ...

Journal: :International journal of molecular medicine 2015
Yue Zhao Yue Feng Yun-Mei Zhang Xiao-Xue Ding Yu-Zhu Song A-Mei Zhang Li Liu Hong Zhang Jia-Huan Ding Xue-Shan Xia

Dilated cardiomyopathy (DCM) is a major cause of sudden cardiac death and heart failure, and it is characterized by genetic and clinical heterogeneity, even for some patients with a very poor clinical prognosis; in the majority of cases, DCM necessitates a heart transplant. Genetic mutations have long been considered to be associated with this disease. At present, mutations in over 50 genes rel...

Journal: :Circulation. Cardiovascular genetics 2009
Lisa M Dellefave Peter Pytel Stephanie Mewborn Bassem Mora Deborah L Guris Savitri Fedson Darrel Waggoner Ivan Moskowitz Elizabeth M McNally

BACKGROUND Mutations in the genes encoding sarcomere proteins have been associated with both hypertrophic and dilated cardiomyopathy. Recently, mutations in myosin heavy chain (MYH7), cardiac actin (ACTC), and troponin T (TNNT2) were associated with left ventricular noncompaction, a form of cardiomyopathy characterized with hypertrabeculation that may also include reduced function of the left v...

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