نتایج جستجو برای: آزمون های gof

تعداد نتایج: 512903  

2014
D. Teguig B. Scheers

In this letter, a blind spectrum sensing method based on goodness-of-fit (GoF) test using likelihood ratio (LLR) is studied. In the proposed method, a chi-square distribution is used for GoF testing. The performance of the method is evaluated through Monte Carlo simulations. It is shown that the proposed spectrum sensing method outperforms the GoF test using Anderson Darling (AD) and the conven...

Journal: :Value in health : the journal of the International Society for Pharmacoeconomics and Outcomes Research 2009
Chung Yin Kong Pamela M McMahon G Scott Gazelle

OBJECTIVES Calibrating a disease simulation model's outputs to existing clinical data is vital to generate confidence in the model's predictive ability. Calibration involves two challenges: 1) defining a total goodness-of-fit (GOF) score for multiple targets if simultaneous fitting is required, and 2) searching for the optimal parameter set that minimizes the total GOF score (i.e., yields the b...

2014
Jie Xu Jin Qian Ye Hu Jilin Wang Xiaolin Zhou Haoyan Chen Jing-Yuan Fang

Mutations of p53 cause not only loss of wild-type function but also gain of novel oncogenic functions (GOF). Accumulating evidence suggest that p53 hotspot mutations may confer different types and magnitudes of GOF. Here we add support to the heterogeneity of mutant p53 GOF by showing their unequal association with early tumor onset and spectrum of tumor types. We stratified Li-Fraumeni syndrom...

Journal: :The Journal of biological chemistry 2013
George Okeyo Wei Wang Shipeng Wei Kevin L Kirk

Cystic fibrosis transmembrane conductance regulator (CFTR) is the only ligand-gated ion channel that hydrolyzes its agonist, ATP. CFTR gating has been argued to be tightly coupled to its enzymatic activity, but channels do open occasionally in the absence of ATP and are reversibly activated (albeit weakly) by nonhydrolyzable nucleotides. Why the latter only weakly activates CFTR is not understo...

2013
Dan Xu Hong Zheng Wen-Mei Yu Cheng-Kui Qu

Gain of function (GOF) mutations in protein tyrosine phosphatase Ptpn11 have been identified in childhood leukemias, and these mutations are sufficient to drive the development of myeloproliferative disorder and malignant leukemias in mice. However, the molecular mechanisms by which Ptpn11 mutations induce these malignancies are not completely understood. Here we report that Ptpn11 GOF mutation...

Journal: :Molecular cell 2014
Ge Zhou Jiping Wang Mei Zhao Tong-Xin Xie Noriaki Tanaka Daisuke Sano Ameeta A Patel Alexandra M Ward Vlad C Sandulache Samar A Jasser Heath D Skinner Alison Lea Fitzgerald Abdullah A Osman Yongkun Wei Xuefeng Xia Zhou Songyang Gordon B Mills Mien-Chie Hung Carlos Caulin Jiyong Liang Jeffrey N Myers

Many mutant p53 proteins (mutp53s) exert oncogenic gain-of-function (GOF) properties, but the mechanisms mediating these functions remain poorly defined. We show here that GOF mutp53s inhibit AMP-activated protein kinase (AMPK) signaling in head and neck cancer cells. Conversely, downregulation of GOF mutp53s enhances AMPK activation under energy stress, decreasing the activity of the anabolic ...

2014
S. Schultz-Cherry R. J. Webby R. G. Webster A. Kelso I. G. Barr J. W. McCauley R. S. Daniels D. Wang Y. Shu E. Nobusawa S. Itamura M. Tashiro Y. Harada S. Watanabe T. Odagiri Z. Ye G. Grohmann R. Harvey O. Engelhardt D. Smith K. Hamilton F. Claes G. Dauphin

In recent years, controversy has arisen regarding the risks and benefits of certain types of gain-of-function (GOF) studies involving avian influenza viruses. In this article, we provide specific examples of how different types of data, including information garnered from GOF studies, have helped to shape the influenza vaccine production process-from selection of candidate vaccine viruses (CVVs...

2015
Paul N. Hopkins Joep Defesche Sigrid W. Fouchier Eric Bruckert Gérald Luc Bertrand Cariou Barbara Sjouke Trond P. Leren Mariko Harada-Shiba Hiroshi Mabuchi Jean-Pierre Rabès Alain Carrié Charles van Heyningen Valérie Carreau Michel Farnier Yee P. Teoh Mafalda Bourbon Masa-aki Kawashiri Atsushi Nohara Handrean Soran A. David Marais Hayato Tada Marianne Abifadel Catherine Boileau Bernard Chanu Shoji Katsuda Ichiro Kishimoto Gilles Lambert Hisashi Makino Yoshihiro Miyamoto Matthieu Pichelin Kunimasa Yagi Masakazu Yamagishi Yassine Zair Scott Mellis George D. Yancopoulos Neil Stahl Johanna Mendoza Yunling Du Sara Hamon Michel Krempf Gary D. Swergold

BACKGROUND Patients with PCSK9 gene gain of function (GOF) mutations have a rare form of autosomal dominant hypercholesterolemia. However, data examining their clinical characteristics and geographic distribution are lacking. Furthermore, no randomized treatment study in this population has been reported. METHODS AND RESULTS We compiled clinical characteristics of PCSK9 GOF mutation carriers ...

2017
Evguenia M Alexandrova Safia A Mirza Sulan Xu Ramona Schulz-Heddergott Natalia D Marchenko Ute M Moll

Missense mutations in TP53 comprise >75% of all p53 alterations in cancer, resulting in highly stabilized mutant p53 proteins that not only lose their tumor-suppressor activity, but often acquire oncogenic gain-of-functions (GOFs). GOF manifests itself in accelerated tumor onset, increased metastasis, increased drug resistance and shortened survival in patients and mice. A known prerequisite fo...

2013
Tongsen Zheng Jiabei Wang Yuhan Zhao Cen Zhang Meihua Lin Xiaowen Wang Haiyang Yu Lianxin Liu Zhaohui Feng Wenwei Hu

The tumour suppressor p53 is frequently mutated in tumours. Mutant p53 (Mutp53) proteins often gain new activities in promoting tumorigenesis, defined as gain-of-function (GOF). Mutp53 can accumulate to high levels in tumours, which promotes mutp53 GOF in tumorigenesis. The mechanism of mutp53 accumulation is poorly understood. Here we find that MDM2 isoforms promote mutp53 accumulation in tumo...

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