نتایج جستجو برای: transforming growth factor beta 1

تعداد نتایج: 3961439  

Journal: :Proceedings of the National Academy of Sciences of the United States of America 1990
Y Sakai C Rawson K Lindburg D Barnes

Serum-free mouse embryo (SFME) cells, derived in medium in which serum is replaced with growth factors and other supplements, display distinctive properties: (i) SFME cells do not lose proliferative potential or show gross chromosomal aberration upon extended culture, (ii) these cells depend on epidermal growth factor for survival; and (iii) SFME cell proliferation is reversibly inhibited by se...

Journal: :The European respiratory journal 2008
K S Lee S J Park S R Kim K H Min K Y Lee Y H Choe S H Hong Y R Lee J S Kim S J Hong Y C Lee

Vascular endothelial growth factor (VEGF) is a mediator of airway inflammation and remodelling in asthma. Transforming growth factor (TGF)-beta(1) plays pivotal roles in diverse biological processes, including tissue remodelling and repair in a number of chronic lung diseases. However, there are few studies elucidating the interactions between VEGF and TGF-beta(1) in allergic airway disease. A ...

Journal: :American journal of physiology. Lung cellular and molecular physiology 2005
Shaoping Xie Maria B Sukkar Razao Issa Ute Oltmanns Andrew G Nicholson Kian Fan Chung

Transforming growth factor (TGF)-beta may play an important role in airway remodeling, and the fibrogenic effect of TGF-beta may be mediated through connective tissue growth factor (CTGF) release. We investigated the role of MAPKs and phosphatidylinositol 3-kinase (PI3K) and the effects of inflammatory cytokines on TGF-beta-induced CTGF expression in human airway smooth muscle cells (ASMC). We ...

Journal: :The International Journal of Biochemistry & Cell Biology 2021

Alzheimer’s disease is an irreversible neurodegenerative disease, which accounts for most dementia cases. Neuroinflammation increasingly recognised its roles in pathogenesis which, part, links amyloid-beta to neuronal death. Neuroinflammatory signalling can be exhibited by neurons themselves, potentially leading widespread cell death, although neuroinflammation commonly associated with glial ce...

Journal: :The Journal of neuroscience : the official journal of the Society for Neuroscience 1997
G M Smith J H Hale

After injury to the CNS, extracellular matrix molecules such as tenascin are upregulated around the injury site and may be involved in inhibition of axon growth. In the present study, astrocytes were investigated to determine which cell types, growth factors, or cytokines are responsible for the injury-induced regulation of tenascin. The addition of activated macrophage- or microglial-condition...

Journal: :Cancer research 2005
Virginia G Kaklamani Lisa Baddi Junjian Liu Diana Rosman Sharbani Phukan Ciarán Bradley Chris Hegarty Bree McDaniel Alfred Rademaker Carole Oddoux Harry Ostrer Loren S Michel Helen Huang Yu Chen Habibul Ahsan Kenneth Offit Boris Pasche

There is growing evidence that common variants of the transforming growth factor-beta (TGF-beta) signaling pathway may modify breast cancer risk. In vitro studies have shown that some variants increase TGF-beta signaling, whereas others have an opposite effect. We tested the hypothesis that a combined genetic assessment of two well-characterized variants may predict breast cancer risk. Consecut...

Journal: :Histology and histopathology 2007
E P Alvares L R Jordão P Gama

Transforming growth factor beta (TGFbeta) isoforms are known for their antiproliferative effect on epithelial cells in vitro, but the role of each isoform in vivo is poorly understood, mainly when non-pathological conditions are considered. We correlated the presence and distribution of isoforms and receptors to physiological changes in gastric cell proliferation in developing and adult rats. W...

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