نتایج جستجو برای: murine double minute2 mdm2

تعداد نتایج: 362783  

2014
Suvi Lokka Andreas H Scheel Sebastian Dango Katja Schmitz Rudolf Hesterberg Josef Rüschoff Hans-Ulrich Schildhaus

BACKGROUND Liposarcoma is the most frequent soft tissue sarcoma. Well differentiated liposarcoma may progress into dedifferentiated liposarcoma with pleomorphic histology. A minority additionally features myogenic, osteo- or chondrosarcomatous heterologous differentiation. Genomic amplification of the Mouse double minute 2 homolog (MDM2) locus is characteristic for well differentiated and dedif...

2013
Xiaofeng Chen Jinrong Qiu Dapeng Yang Jianlei Lu Caiyun Yan Xiaoming Zha Yongmei Yin

The molecular mechanisms that underpin invasive ductal breast cancer (IDC) invasion and metastasis are incompletely understood. The oncogene, mouse double minute 2 (MDM2), has been implicated in the pathogenesis of numerous cancers, where it stimulates the expression of matrix metalloproteinase 9 (MMP9), an important enzyme in the breakdown of the extracellular matrix. However, its role in brea...

2011
Osamu Nunobiki Masatsugu Ueda Eisaku Toji Michiko Yamamoto Kyoko Akashi Naomi Sato Shinji Izuma Kiyo Torii Ichiro Tanaka Yoshiaki Okamoto Sadamu Noda

It is widely accepted that specific human papillomavirus (HPV) types are the central etiologic agent of cervical carcinogenesis. However, a number of infected women do not develop invasive lesions, suggesting that other environmental and host factors may play decisive roles in the persistence of HPV infection and further malignant conversion of cervical epithelium. Although many previous report...

Journal: :The Biochemical journal 2006
Sara Sánchez-Molina José Luis Oliva Susana García-Vargas Ester Valls José M Rojas Marian A Martínez-Balbás

The CBP [CREB (cAMP-response-element-binding protein)-binding protein]/p300 acetyltransferases function as transcriptional co-activators and play critical roles in cell differentiation and proliferation. Accumulating evidence shows that alterations of the CBP/p300 protein levels are linked to human tumours. In the present study, we show that the levels of the CBP/p300 co-activators are decrease...

2014
Dongmin Gu Shuhong Wang Isere Kuiatse Hua Wang Jin He Yun Dai Richard J. Jones Chad C. Bjorklund Jing Yang Steven Grant Robert Z. Orlowski

Intracellular proteolytic pathways have been validated as rational targets in multiple myeloma with the approval of two proteasome inhibitors in this disease, and with the finding that immunomodulatory agents work through an E3 ubiquitin ligase containing Cereblon. Another E3 ligase that could be a rational target is the murine double minute (MDM) 2 protein, which plays a role in p53 turnover. ...

Journal: :The Journal of clinical investigation 2015
Dan Zhu Chenchen Li Andrew M Swanson Rosa M Villalba Jidong Guo Zhaobin Zhang Shannon Matheny Tatsuro Murakami Jason R Stephenson Sarah Daniel Masaki Fukata Randy A Hall Jeffrey J Olson Gretchen N Neigh Yoland Smith Donald G Rainnie Erwin G Van Meir

Synaptic plasticity is the ability of synapses to modulate the strength of neuronal connections; however, the molecular factors that regulate this feature are incompletely understood. Here, we demonstrated that mice lacking brain-specific angiogenesis inhibitor 1 (BAI1) have severe deficits in hippocampus-dependent spatial learning and memory that are accompanied by enhanced long-term potentiat...

Journal: :Cancer research 2007
Wan Mui Chan Randy Y C Poon

The transcription factor p53 is one of the most frequently mutated tumor suppressors. Recent progress has unraveled several novel isoforms of p53. Intriguingly, one of the p53 isoform, Deltap53, which lacks part of the DNA binding domain, was reported to be transcriptionally active toward some p53 target genes and is critical for the intra-S phase checkpoint. Here, we show that, in contrast to ...

Journal: :PloS one 2015
Mariell Pettersson Maria Quant Jaeki Min Luigi Iconaru Richard W Kriwacki M Brett Waddell R Kiplin Guy Kristina Luthman Morten Grøtli

The transcription factor p53 is the main tumour suppressor in cells and many cancer types have p53 mutations resulting in a loss of its function. In tumours that retain wild-type p53 function, p53 activity is down-regulated by MDM2 (human murine double minute 2) via a direct protein-protein interaction. We have designed and synthesised two series of 2,5-diketopiperazines as inhibitors of the MD...

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