نتایج جستجو برای: microphthalmia

تعداد نتایج: 1639  

Journal: :Blood 1997
H Nechushtan Z Zhang E Razin

Mice harboring a mutation in the microphthalmia (mi) gene display a variety of abnormalities, including microphthalmia, depletion of skin melanocytes, deafness, a defect in osteoclasts, and a major decrease in mast cell number and function. However, despite the possible critical role played by this protein in mast cell development and function, characterization of its mRNA and protein synthesis...

Journal: :Investigative ophthalmology & visual science 2001
O J Lehmann M F El-ashry N D Ebenezer L Ocaka P J Francis S E Wilkie R J Patel L Ficker T Jordan P T Khaw S S Bhattacharya

PURPOSE Mutations in keratocan (KERA), a small leucine-rich proteoglycan, have recently been shown to be responsible for cases of autosomal recessive cornea plana (CNA2). A consanguineous pedigree in which cornea plana cosegregated with microphthalmia was investigated by linkage analysis and direct sequencing. METHODS Linkage was sought to polymorphic microsatellite markers distributed around...

Journal: :Human mutation 2016
Alan S Ma John R Grigg Gladys Ho Ivan Prokudin Elizabeth Farnsworth Katherine Holman Anson Cheng Frank A Billson Frank Martin Clare Fraser David Mowat James Smith John Christodoulou Maree Flaherty Bruce Bennetts Robyn V Jamieson

Congenital cataracts are a significant cause of lifelong visual loss. They may be isolated or associated with microcornea, microphthalmia, anterior segment dysgenesis (ASD) and glaucoma, and there can be syndromic associations. Genetic diagnosis is challenging due to marked genetic heterogeneity. In this study, next-generation sequencing (NGS) of 32 cataract-associated genes was undertaken in 4...

Journal: :Human molecular genetics 2012
Chunqiao Liu Hirva Bakeri Tiansen Li Anand Swaroop

Nineteen Wnt ligands and 10 Frizzled (Fz) receptors mediate multiple distinct cellular events during neuronal development. However, their precise roles in cell-type specification and organogenesis are poorly delineated because of overlapping functions and expression profiles. Here, we have explored the role of two closely related Frizzled receptors, Fz5 and Fz8, in mouse retinal development. We...

2017
Tanya Bardakjian Max Krall Di Wu Richard Lao Paul Ling-Fung Tang Eunice Wan Sarina Kopinsky Adele Schneider Pui-yan Kwok Anne Slavotinek

Purpose The genetic causes of anophthalmia, microphthalmia and coloboma remain poorly understood. Missense mutations in Growth/Differentiation Factor 3 (GDF3) gene have previously been reported in patients with microphthalmia, iridial and retinal colobomas, Klippel-Feil anomaly with vertebral fusion, scoliosis, rudimentary 12th ribs and an anomalous right temporal bone. We used whole exome sequ...

Journal: :Development 2003
Eric S Green Jennifer L Stubbs Edward M Levine

Insufficient cell number is a primary cause of failed retinal development in the Chx10 mutant mouse. To determine if Chx10 regulates cell number by antagonizing p27(Kip1) activity, we generated Chx10, p27(Kip1) double null mice. The severe hypocellular defect in Chx10 single null mice is alleviated in the double null, and while Chx10-null retinas lack lamination, double null retinas have near n...

Journal: :Cytogenetic and genome research 2002
K Kutsche W Werner O Bartsch A von der Wense P Meinecke A Gal

The microphthalmia with linear skin defects syndrome (MLS) is an X-linked dominant disorder with male lethality. In the majority of the patients reported, the MLS syndrome is caused by segmental monosomy of the Xp22.3 region. To date, five male patients with MLS and 46,XX karyotype ("XX males") have been described. Here we report on the first male case with MLS and an XY complement. The patient...

Journal: :Development 2015
Séverine Marcos Monica González-Lázaro Leonardo Beccari Laura Carramolino Maria Jesus Martin-Bermejo Oana Amarie Daniel Mateos-San Martín Carlos Torroja Ozren Bogdanović Roisin Doohan Oliver Puk Martin Hrabě de Angelis Jochen Graw Jose Luis Gomez-Skarmeta Fernando Casares Miguel Torres Paola Bovolenta

Microphthalmos is a rare congenital anomaly characterized by reduced eye size and visual deficits of variable degree. Sporadic and hereditary microphthalmos have been associated with heterozygous mutations in genes fundamental for eye development. Yet, many cases are idiopathic or await the identification of molecular causes. Here we show that haploinsufficiency of Meis1, which encodes a transc...

Journal: :Investigative ophthalmology & visual science 1991
R L Zhang D A Samuelson Z G Zhang V N Reddy B S Shastry

The congenital hereditary cataracts and microphthalmia in the miniature schnauzer dog are inherited by an autosomal recessive mode. To understand the genetic basis of these diseases, the authors purified and analyzed leukocyte deoxyribonucleic acid (DNA) from affected and normal animals using a candidate gene approach. Because the genes that encode the lens-specific proteins, specifically, alph...

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