نتایج جستجو برای: masserann kit

تعداد نتایج: 29130  

Journal: :Cancer research 2006
Marcus M Schittenhelm Sharon Shiraga Arin Schroeder Amie S Corbin Diana Griffith Francis Y Lee Carsten Bokemeyer Michael W N Deininger Brian J Druker Michael C Heinrich

Activating mutations of the activation loop of KIT are associated with certain human neoplasms, including the majority of patients with systemic mast cell disorders, as well as cases of seminoma, acute myelogenous leukemia (AML), and gastrointestinal stromal tumors (GISTs). The small-molecule tyrosine kinase inhibitor imatinib mesylate is a potent inhibitor of wild-type (WT) KIT and certain mut...

2016
Oun Jae Park Ji Su Han Sang Hyung Lee Chan-Sik Park Chong Hyun Won Mi Woo Lee Jee Ho Choi Sung Eun Chang

BACKGROUND The survival and growth of melanocytes are controlled by the binding of stem cell factor to its cell surface receptor c-kit+ (CD117). We have observed that c-kit+ melanocytes existed in some lesions of vitiligo, while Melan A+ cells were absent. OBJECTIVE To verify possible relation between c-kit+ expression and treatment response in non-segmental vitiligo lesions. METHODS Skin b...

Journal: :New South Wales Public Health Bulletin 2000

Journal: :Nippon Ronen Igakkai Zasshi. Japanese Journal of Geriatrics 2011

Journal: :Journal of Criminal Law and Criminology (1931-1951) 1940

Journal: :Journal of Cell Biology 2008

Journal: :Blood 1995
K Ogawa Y Takeda M Tashima H Sawai T Toi T Okazaki H Sawada Y Maruyama M Okuma

We previously reported that the K562 cell line K562YO expressed a high level of the c-kit gene. In this study, we analyzed the mechanism of this expression and investigated the effects of the serine/threonine kinases such as protein kinase C (PKC) and cyclic adenosine 3',5'-monophosphate (cAMP)-dependent kinase (PKA) on it. The half-life of the c-kit mRNA in K562YO cells was greater than 10 hou...

Journal: :Clinical cancer research : an official journal of the American Association for Cancer Research 2005
Stefania Uccini Olga Mannarino Heather P McDowell Ursula Pauser Roberta Vitali Pier Giorgio Natali Pierluigi Altavista Tiziana Andreano Simona Coco Renata Boldrini Sandro Bosco Anna Clerico Denis Cozzi Alberto Donfrancesco Alessandro Inserra George Kokai Paul D Losty Maria R Nicotra Giuseppe Raschellà Gian Paolo Tonini Carlo Dominici

PURPOSE Clinicobiological characteristics of neuroblastic tumor (NT) expressing c-kit tyrosine kinase receptor and/or its ligand, stem cell factor (SCF), are debated. This study aimed at investigating the clinicobiological features of primary NTs expressing c-kit and/or SCF in order to define the clinical relevance of selective therapeutic targeting. EXPERIMENTAL DESIGN c-Kit and SCF expressi...

Journal: :Blood 1994
P S Cohen J P Chan M Lipkunskaya J L Biedler R C Seeger

During development, mice with mutations of stem cell factor (SCF) or its receptor c-kit exhibit defects in melanogenesis, as well as hematopoiesis and gonadogenesis. Because melanocytes derive from neural crest cells, the role of SCF and c-kit was investigated in the neural crest-derived childhood tumor neuroblastoma. Using reverse transcription-polymerase chain reaction analysis, simultaneous ...

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