نتایج جستجو برای: kcnj11

تعداد نتایج: 484  

Journal: :Pharmaciana 2021

Diabetes Mellitus (DM) is a metabolic disease that developed due to the pancreas does not sufficient produce insulin or body cannot effectively use it produces. Genetic factors have an essential role in development of Type 2 (DMT2), which impaired production by pancreatic β cells, resistance, and action. The single nucleotide polymorphisms (SNP) KCNJ11 rs5219 affected cell activity can inhibit ...

2017
Sorin Ioacara Sarah Flanagan Elke Fröhlich‐Reiterer Robin Goland Simona Fica

In this report, we present the first known case of intermediate developmental delay, epilepsy and permanent neonatal diabetes (DEND) syndrome caused by a Q52R mutation in the KCNJ11 gene who was successfully switched (at age 1.3 years) to sulphonylurea monotherapy, namely glibenclamide. The most recent evaluation, after 2 years, showed a glycated hemoglobin level of 6.0% (42 mmol/mol). This mut...

Journal: :African health sciences 2015
Catherine Nyangabyaki-Twesigye Michael Rugambwa Muhame Silver Bahendeka

Diabetes mellitus is a metabolic disease characterised by chronically high glucose levels. Genetic factors have been implicated in the aetiology following mutations in a single gene. An extremely rare form of diabetes mellitus is monogenic diabetes, a subset of which is permanent neonatal diabetes, and is usually suspected if a child is diagnosed with diabetes at less than 6 months of age. We p...

Journal: :Journal of the College of Physicians and Surgeons--Pakistan : JCPSP 2016
Sabeen Abid Khan Arit Parkash Mohsina Ibrahim

DEND syndrome is a very rare syndrome of permanent neonatal diabetes mellitus, with an incidence of < 1/1000,000. It is defined as a triad of developmental delay, epilepsy, and neonatal diabetes. We report the case of a 9-month infant girl who presented with the most severe form of neonatal diabetes mellitus spectrum along with developmental delay and epilepsy. Genetic mutation testing confirme...

2010
Alex Coventry Lara M. Bull-Otterson Xiaoming Liu Andrew G. Clark Taylor J. Maxwell Jacy Crosby James E. Hixson Thomas J. Rea Donna M. Muzny Lora R. Lewis David A. Wheeler Aniko Sabo Christine Lusk Kenneth G. Weiss Humeira Akbar Andrew Cree Alicia C. Hawes Irene Newsham Robin T. Varghese Donna Villasana Shannon Gross Vandita Joshi Jireh Santibanez Margaret Morgan Kyle Chang Walker Hale IV Alan R. Templeton Eric Boerwinkle Richard Gibbs Charles F. Sing

Accurately determining the distribution of rare variants is an important goal of human genetics, but resequencing of a sample large enough for this purpose has been unfeasible until now. Here, we applied Sanger sequencing of genomic PCR amplicons to resequence the diabetes-associated genes KCNJ11 and HHEX in 13,715 people (10,422 European Americans and 3,293 African Americans) and validated amp...

Journal: :Journal of pediatric endocrinology & metabolism : JPEM 2015
Shira Harel Ana S A Cohen Khalid Hussain Sarah E Flanagan Kamilla Schlade-Bartusiak Millan Patel Jaques Courtade Jenny B W Li Clara Van Karnebeek Harley Kurata Sian Ellard Jean-Pierre Chanoine William T Gibson

BACKGROUND Inheritance of two pathogenic ABCC8 alleles typically causes severe congenital hyperinsulinism. We describe a girl and her father, both homozygous for the same ABCC8 mutation, who presented with unusual phenotypes. METHODS Single nucleotide polymorphism microarray and Sanger sequencing were performed. Western blot, rubidium efflux, and patch clamp recordings interrogated the expres...

2015
Ahmad Adi Bassam Bin Abbas Mohamed Al Hamed Nada Al Tassan Dana Bakheet J. Peter W. Young

The autosomal recessive form of persistent hyperinsulinemic hypoglycemia of infancy (PHHI) is associated with mutations in either ABCC8 or KCNJ11 genes. In the present study, we describe the clinical features and results of genetic analysis of 13 Saudi Arabian patients with PHHI. Clinically, most patients presented with infantile seizures and/or developmental delay, with a subset of patients wh...

Journal: :Indian pediatrics 2016
Amit Kumar Satapathy Vandana Jain Sian Ellard Sarah E Flanagan

BACKGROUND Hyperinsulinemia is the commonest cause of persistent hypoglycemia in infancy. Inactivating mutations in the genes ABCC8 and KCNJ11 are the commonest cause. Mutation in the HADH gene, which encodes the short-chain-L-3-hydroxyacyl-CoA dehydrogenase, is a rare cause. CASE CHARACTERISTICS Two Indian sisters who presented with hyperinsulinemic hypoglycemia of infancy. OBSERVATION/INT...

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