نتایج جستجو برای: jak2v617f mutation

تعداد نتایج: 291772  

2013
Costanza Bogani Niccolò Bartalucci Serena Martinelli Lorenzo Tozzi Paola Guglielmelli Alberto Bosi Alessandro M. Vannucchi

BACKGROUND Dysregulated signaling of the JAK/STAT pathway is a common feature of chronic myeloproliferative neoplasms (MPN), usually associated with JAK2V617F mutation. Recent clinical trials with JAK2 inhibitors showed significant improvements in splenomegaly and constitutional symptoms in patients with myelofibrosis but meaningful molecular responses were not documented. Accordingly, there re...

Journal: :Blood 2011
Lisa G van den Hengel Begüm Kocatürk Pieter H Reitsma Wolfram Ruf Henri H Versteeg

previous PV diagnosis, achieved at least CCyR. In summary, in this larger cohort of CML patients compared with that studied by Makishima et al,1 we found 2.55% of cases presenting concomitant BCR/ABL rearrangement and JAK2V617F mutation, indicating that the simultaneous occurrence of these mutations is rare event but it is not a phoenix; however, while the pathophysiologic significance of this ...

Journal: :Haematologica 2010
Hong Chang Allan Jiang Joseph Brandwein

Blood. 2007;110(4):1092-7. 10. Palandri F, Ottaviani E, Salmi F, Catani L, Polverelli N, Fiacchini M, et al. JAK2V617F mutation in essential thrombocythemia: correlation with clinical characteristics, responce to therapy and long-term out come in a cohort of 275 patients. Leuk Lymphoma 2009;50(2):247-53. 11. Finazzi G, Rambaldi A, Guerini V, Carobbo A, Barbui T. Risk of thrombosis in patients w...

Journal: :Blood 2009
Paola Guglielmelli Giovanni Barosi Giorgina Specchia Alessandro Rambaldi Francesco Lo Coco Elisabetta Antonioli Lisa Pieri Alessandro Pancrazzi Vanessa Ponziani Federica Delaini Giovanni Longo Emanuele Ammatuna Vincenzo Liso Alberto Bosi Tiziano Barbui Alessandro M Vannucchi

A total of 186 patients with primary myelofibrosis (PMF) were genotyped for JAK2V617F at diagnosis aimed at analyzing the correlation of mutational status and mutated allele burden with outcome variables, including time to anemia, leukocytosis, leukopenia, thrombocytopenia, massive splenomegaly, leukemia, and with overall survival. A total of 127 JAK2V617F-mutated patients (68% of whole series)...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2014
Raajit Rampal Jihae Ahn Omar Abdel-Wahab Michelle Nahas Kai Wang Doron Lipson Geoff A Otto Roman Yelensky Todd Hricik Anna Sophia McKenney Gabriela Chiosis Young Rock Chung Suveg Pandey Marcel R M van den Brink Scott A Armstrong Ahmet Dogan Andrew Intlekofer Taghi Manshouri Christopher Y Park Srdan Verstovsek Franck Rapaport Philip J Stephens Vincent A Miller Ross L Levine

Patients with myeloproliferative neoplasms (MPNs) are at significant, cumulative risk of leukemic transformation to acute myeloid leukemia (AML), which is associated with adverse clinical outcome and resistance to standard AML therapies. We performed genomic profiling of post-MPN AML samples; these studies demonstrate somatic tumor protein 53 (TP53) mutations are common in JAK2V617F-mutant, pos...

Journal: :research in molecular medicine 0
kaveh tari department of hematology, faculty of medical sciences, tarbiat modares university, tehran, irandepartment of hematology, faculty of medical sciences, tarbiat modares university, tehran, iran amir atashi department of hematology, faculty of medical sciences, tarbiat modares university, tehran, iran reza yarahmadi department of laboratory sciences, school of paramedical sciences, ahvaz jundishapur university of medical sciences, ahvaz, iran saeid abroun department of hematology, faculty of medical sciences, tarbiat modares university, tehran, iran abbas hajifathali taleghani hospital, shahid beheshti university of medical sciences, tehran, iran saeid kaviani department of hematology, faculty of medical sciences, tarbiat modares university, tehran, iran

mpns including a heterogeneous group of clonal or oligoclonal hamtopathies characterized by proliferation and accumulation of mature myeloid cells. jak2 tyrosine kinase mutation is the most common molecular lesion identified in 90% of cases. jak2 is involved in epo signaling pathway, and mutations in it lead to epo-independent spontaneous phosphorylation. most tyrosine kinase inhibitors (tki) a...

2009
Phillip C.C. Liu Eian Caulder Jun Li Paul Waeltz Alex Margulis Richard Wynn Mary Becker-Pasha Yanlong Li Erin Crowgey Gregory Hollis Patrick Haley Richard B. Sparks Andrew P. Combs James D. Rodgers Timothy C. Burn Kris Vaddi Jordan S. Fridman

Purpose: Deregulation of the Janus kinase-signal transducers and activators of transcription (JAK-STAT) pathway is a hallmark for the Philadelphia chromosome–negative myeloproliferative diseases polycythemia vera, essential thrombocythemia, and primary myelofibrosis. We tested the efficacy of a selective JAK1/2 inhibitor in cellular and in vivo models of JAK2-driven malignancy. Experimental Des...

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