نتایج جستجو برای: jak2v617f

تعداد نتایج: 776  

2012
C Sugimori E Padron G Caceres K Shain L Sokol L Zhang R Tiu C L O'Keefe M Afable M Clemente J M Lee J P Maciejewski A F List P K Epling-Burnette D J Araten

Paroxysmal nocturnal hemoglobinuria (PNH) is a hemolytic and pro-thrombotic disorder associated with the clonal expansion of hematopoietic stem cells harboring somatic mutations in the PIG-A gene. Mutations in PIG-A result in a lack of surface expression of all glycosylphosphatidylinositol (GPI)-anchored proteins, including the complement inhibitors CD55 and CD59, which is responsible for the h...

Journal: :Blood 2008
Jean-Jacques Kiladjian Bruno Cassinat Sylvie Chevret Pascal Turlure Nathalie Cambier Murielle Roussel Sylvia Bellucci Bernard Grandchamp Christine Chomienne Pierre Fenaux

Interferon-alpha (IFN-alpha) is a nonleukemogenic treatment of polycythemia vera (PV) able to induce cytogenetic remissions. Its use is limited by toxicity, leading to treatment discontinuation in approximately 20% of patients. We completed a phase 2 multicenter study of pegylated IFN-alpha-2a in 40 PV patients. Objectives included evaluation of efficacy, safety, and monitoring of residual dise...

2016
Jack W Singer Suliman Al-Fayoumi Haiching Ma Rami S Komrokji Ruben Mesa Srdan Verstovsek

Pacritinib, potent inhibitor of Janus kinase 2 (JAK2), JAK2V617F, and fms-like receptor tyrosine kinase 3, is in Phase III development in myelofibrosis. Among type 1 inhibitors, pacritinib shows a lack of myelosuppression at doses that both inhibit JAK2/signal transducer and activator of transcription 3 pathway and demonstrate clinical efficacy. To elucidate these mechanisms and identify other ...

2008
Constantine S. Tam Roberto M. Nussenzveig Uday Popat Carlos E. Bueso-Ramos Deborah A. Thomas Jorge A. Cortes Richard E. Champlin Stefan E. Ciurea Taghi Manshouri Sherry M. Pierce Hagop M. Kantarjian Srdan Verstovsek

We analyzed the outcomes of 74 patients diagnosed with BCR-ABL myeloproliferative neoplasms in blast phase receiving induction chemotherapy (55%), low-intensity therapy (16%), stem cell transplantation (SCT; 3%), or supportive care (26%). Median survival from the date of blastic transformation was 5 months. Patients receiving supportive therapy had a median survival of 6 weeks. Complete remissi...

Journal: :Blood 2010
Nils H Thoennissen Utz O Krug Dhong Hyun Tony Lee Norihiko Kawamata Gabriela B Iwanski Terra Lasho Tamara Weiss Daniel Nowak Maya Koren-Michowitz Motohiro Kato Masashi Sanada Lee-Yung Shih Arnon Nagler Sophie D Raynaud Carsten Müller-Tidow Ruben Mesa Torsten Haferlach D Gary Gilliland Ayalew Tefferi Seishi Ogawa H Phillip Koeffler

Philadelphia chromosome-negative myeloproliferative neoplasms (MPNs) including polycythemia vera, essential thrombocythemia, and primary myelofibrosis show an inherent tendency for transformation into leukemia (MPN-blast phase), which is hypothesized to be accompanied by acquisition of additional genomic lesions. We, therefore, examined chromosomal abnormalities by high-resolution single nucleo...

Journal: :Blood 2012
Stefania Bortoluzzi Andrea Bisognin Marta Biasiolo Paola Guglielmelli Flavia Biamonte Ruggiero Norfo Rossella Manfredini Alessandro M Vannucchi

To gain insights into a possible role of microRNAs in myeloproliferative neoplasms, we performed short RNA massive sequencing and extensive bioinformatic analysis in the JAK2V617F-mutated SET2 cell line. Overall, 652 known mature miRNAs were detected, of which 21 were highly expressed, thus being responsible of most of miRNA-mediated gene repression. microRNA putative targets were enriched in s...

ژورنال: پیاورد سلامت 2009
بهار, بابک, توگه, غلام رضا, درگاهی, حسین, علی مقدم, کامران, غفاری, حمیداله, قوام زاده, اردشیر, نادعلی, فاطمه, چهاردولی, بهرام, کریم زاده, پریسا,

Background and Aim: The JAK2 is an acquired mutation that is observed in majority of patients with classical Philadelphia-negative Myeloproliferative neoplasms that include polycythemia vera (PV), essential thrombocythemia (ET), primary myelofibrosis (PMF). This acquired mutation is characterized by a G to T transversion at nucleotide 1849 in exon 12 of the JAK2 gene, leading to a substitution ...

Journal: :The Korean Journal of Hematology 2010

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