نتایج جستجو برای: galectin

تعداد نتایج: 4550  

Journal: :The Lancet. Oncology 2008
Armando Bartolazzi Fabio Orlandi Enrico Saggiorato Marco Volante Federico Arecco Ruth Rossetto Nicola Palestini Ezio Ghigo Mauro Papotti Gianni Bussolati Marco Paolo Martegani Federico Pantellini Angelo Carpi Maria Rosaria Giovagnoli Salvatore Monti Vincenzo Toscano Salvatore Sciacchitano Gian Maria Pennelli Caterina Mian Maria Rosa Pelizzo Massimo Rugge Giancarlo Troncone Lucio Palombini Gennaro Chiappetta Gerardo Botti Aldo Vecchione Rino Bellocco

BACKGROUND In the USA, about 30 200 well-differentiated thyroid carcinomas were diagnosed in 2007, but the prevalence of thyroid nodules is much higher (about 5% of the adult population). Unfortunately, the preoperative characterisation of follicular thyroid nodules is still a challenge, and many benign lesions, which remain indeterminate after fine-needle aspiration (FNA) cytology are referred...

Journal: :Journal of the American Society of Nephrology : JASN 2015
Christiane Drechsler Graciela Delgado Christoph Wanner Katja Blouin Stefan Pilz Andreas Tomaschitz Marcus E Kleber Alexander Dressel Christoph Willmes Vera Krane Bernhard K Krämer Winfried März Eberhard Ritz Wiek H van Gilst Pim van der Harst Rudolf A de Boer

Galectin-3 has been linked to incident renal disease, experimental renal fibrosis, and nephropathy. However, the association among galectin-3, renal function, and adverse outcomes has not been described. We studied this association in two large cohorts of patients over a broad range of renal function. We measured galectin-3 concentrations in baseline samples from the German Diabetes mellitus Di...

Journal: :Circulation 2004
Umesh C Sharma Saraswati Pokharel Thomas J van Brakel Jop H van Berlo Jack P M Cleutjens Blanche Schroen Sabine André Harry J G M Crijns Hans-J Gabius Jos Maessen Yigal M Pinto

BACKGROUND Inflammatory mechanisms have been proposed to be important in heart failure (HF), and cytokines have been implicated to add to the progression of HF. However, it is unclear whether such mechanisms are already activated when hypertrophied hearts still appear well-compensated and whether such early mechanisms contribute to the development of HF. METHODS AND RESULTS In a comprehensive...

2005
Andrew L. Schwaderer Soundarapandian Vijayakumar Qais Al-Awqati George J. Schwartz

Schwaderer, Andrew L., Soundarapandian Vijayakumar, Qais Al-Awqati, and George J. Schwartz. Galectin-3 expression is induced in renal -intercalated cells during metabolic acidosis. Am J Physiol Renal Physiol 290: F148–F158, 2006. First published August 30, 2005; doi:10.1152/ajprenal.00244.2005.—The adaptation of the cortical collecting duct (CCD) to metabolic acidosis requires the polymerizatio...

Journal: :International journal of oncology 2015
Koji Fujita Hisakazu Iwama Teppei Sakamoto Ryoichi Okura Kiyoyuki Kobayashi Jitsuko Takano Akiko Katsura Miwa Tatsuta Emiko Maeda Shima Mimura Takako Nomura Joji Tani Hisaaki Miyoshi Asahiro Morishita Hirohito Yoneyama Yuka Yamana Takashi Himoto Keiichi Okano Yasuyuki Suzuki Toshiro Niki Mitsuomi Hirashima Tsutomu Masaki

Galectin-9, a soluble β-galactoside-binding animal lectin, evokes apoptosis in various human cancer cell lines. The galectin-9 antitumor effect against hepatocellular carcinoma (HCC) is, however, unknown. We investigated whether galectin-9 suppresses HCC growth in vitro and in vivo. We assessed the antitumor effect of galectin-9 on HCC cells by conducting WST-8 assay in vitro and xenograft mode...

Journal: :Clinical chemistry 2012
E Wilson Grandin Petr Jarolim Sabina A Murphy Lea Ritterova Christopher P Cannon Eugene Braunwald David A Morrow

BACKGROUND Galectin-3 is a β-galactoside-binding lectin that has been implicated in cardiac fibrosis and remodeling, is increased in models of failure-prone hearts, and has prognostic value in patients with heart failure (HF). The relationship between galectin-3 and the development of HF after acute coronary syndrome (ACS) is unknown. METHODS In a nested case-control study among patients with...

Journal: :Journal of immunology 2006
Jennifer A Fulcher Sara T Hashimi Ernest L Levroney Mabel Pang Kevin B Gurney Linda G Baum Benhur Lee

Dendritic cells (DCs) are potent mediators of the immune response, and can be activated by exogenous pathogen components. Galectin-1 is a member of the conserved beta-galactoside-binding lectin family that binds galactoside residues on cell surface glycoconjugates. Galectin-1 is known to play a role in immune regulation via action on multiple immune cells. However, its effects on human DCs are ...

Journal: :PLoS ONE 2008
Armando Bartolazzi Calogero D'Alessandria Maria Gemma Parisella Alberto Signore Fabrizio Del Prete Luca Lavra Sten Braesch-Andersen Roberto Massari Carlo Trotta Alessandro Soluri Salvatore Sciacchitano Francesco Scopinaro

BACKGROUND The prevalence of thyroid nodules increases with age, average 4-7% for the U.S.A. adult population, but it is much higher (19-67%) when sub-clinical nodules are considered. About 90% of these lesions are benign and a reliable approach to their preoperative characterization is necessary. Unfortunately conventional thyroid scintigraphy does not allow the distinction among benign and ma...

2011
Emma Salomonsson Victor L. Thijssen Arjan W. Griffioen Ulf J. Nilsson Hakon Leffler

Angiogenesis is a key event in cancer progression and therefore a promising target in cancer treatment. Galectin-1, a β-galactoside binding lectin, is up-regulated in the endothelium of tumors of different origin and has been shown to be the target for anginex, a powerful anti-angiogenic peptide with anti-tumor activity. Here we show that when bound to anginex, galectin-1 binds various glycopro...

2017
Ping Kei Yip Alejandro Carrillo-Jimenez Paul King Anna Vilalta Koji Nomura Chi Cheng Chau Alexander Michael Scott Egerton Zhuo-Hao Liu Ashray Jayaram Shetty Jordi L. Tremoleda Meirion Davies Tomas Deierborg John V. Priestley Guy Charles Brown Adina Teodora Michael-Titus Jose Luis Venero Miguel Angel Burguillos

Traumatic brain injury (TBI) is currently a major cause of morbidity and poor quality of life in Western society, with an estimate of 2.5 million people affected per year in Europe, indicating the need for advances in TBI treatment. Within the first 24 h after TBI, several inflammatory response factors become upregulated, including the lectin galectin-3. In this study, using a controlled cortic...

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