نتایج جستجو برای: excitotoxicity

تعداد نتایج: 2624  

2013
Lourdes Lorigados Sandra Orozco Lilia Morales Bárbara Estupiñán Iván García Luisa Rocha

Epilepsy is a recurrent, often progressive neurological disorder with a chronic evolution, affecting 1 to 2 % of the world population. Research with experimental models and imaging analysis of diseased patients have been used to show that recurrent episodes produce oxidative stress, most of which is related to neuronal excitability phenomena. It is known that the excessive stimulation of glutam...

Journal: :FASEB journal : official publication of the Federation of American Societies for Experimental Biology 2005
Raghuveer S Mali Mei Cheng Shravan K Chintala

Increased levels of extracellular l-glutamate have been suggested to play a role in retinal damage in a number of blinding diseases such as glaucoma and diabetic retinopathy. Although glutamate can cause retinal damage in part by hyperstimulating its receptors ("excitotoxicity"), the downstream events that lead to retinal damage are poorly understood. In this study, we injected kainic acid (KA)...

2015
Alessandro Vercelli Tiziana Borsello Alessandra Sclip Silvia Biggi Ivan Enrico Repetto Sara Cimini Fabio Falleroni Simone Tomasi Riccardo Monti Noemi Tonna Federica Morelli Valentina Grande Oriano Marin Fabio Bianco Daniele di Marino

MicroRNAs (miRNAs) are short, 22–25 nucleotide long transcripts that may suppress entire signaling pathways by interacting with the 3’-untranslated region (3’-UTR) of coding mRNA targets, interrupting translation and inducing degradation of these targets. The long 3’-UTRs of brain transcripts compared to other tissues predict important roles for brain miRNAs. Supporting this notion, we found th...

Journal: :Oxidative Medicine and Cellular Longevity 2017

Journal: :Proceedings of the National Academy of Sciences of the United States of America 1997
P E Schauwecker O Steward

Recent studies have sought to identify the genes involved in excitotoxic neurodegeneration. Here we report that certain strains of mice, including strains that are used for gene targeting studies, do not exhibit excitotoxic cell death after kainic acid seizures. Kainic acid produced excitotoxic cell death in the CA3 and CA1 subfields of the hippocampus in 129/SvEMS and FVB/N mice, in the same p...

2001
M. van der Stelt W. B. Veldhuis P. R. Bär G. A. Veldink J. F. G. Vliegenthart K. Nicolay

Excitotoxicity is a paradigm used to explain the biochemical events in both acute neuronal damage and in slowly progressive, neurodegenerative diseases. Here, we show in a longitudinal magnetic resonance imaging study that -tetrahydrocannabinol ( -THC), the main active compound in marijuana, reduces neuronal injury in neonatal rats injected intracerebrally with the Na /K -ATPase inhibitor ouaba...

2013
Sheng T. Hou Susan X. Jiang Amy Aylsworth Matthew Cooke Lei Zhou

CRMP proteins play critical regulatory roles during semaphorin-mediated neurite outgrowth, neuronal differentiation and death. Albeit having a high degree of structure and sequence resemblance to that of liver dihydropyrimidinase, purified rodent brain CRMPs do not hydrolyze dihydropyrimidinase substrates. Here we found that mouse CRMP3 has robust histone H4 deacetylase activity. During excitot...

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