نتایج جستجو برای: ddah

تعداد نتایج: 226  

2015
Christine M. Gross Ruslan Rafikov Sanjiv Kumar Saurabh Aggarwal P. Benson Ham III Mary Louise Meadows Mary Cherian-Shaw Archana Kangath Supriya Sridhar Rudolf Lucas Stephen M. Black

Lipopolysaccharide (LPS) derived from the outer membrane of gram-negative bacteria induces acute lung injury (ALI) in mice. This injury is associated with lung edema, inflammation, diffuse alveolar damage, and severe respiratory insufficiency. We have previously reported that LPS-mediated nitric oxide synthase (NOS) uncoupling, through increases in asymmetric dimethylarginine (ADMA), plays an i...

Journal: :Arteriosclerosis, thrombosis, and vascular biology 2004
Fortunato Scalera Jürgen Borlak Bibiana Beckmann Jens Martens-Lobenhoffer Thomas Thum Michael Täger Stefanie M Bode-Böger

OBJECTIVE Asymmetrical dimethylarginine (ADMA) is an endogenous inhibitor of nitric oxide synthase (NOS), and its accumulation has been associated with cardiovascular disease. We aimed to investigate the role of ADMA in endothelial cell senescence. METHODS AND RESULTS Endothelial cells were cultured until the tenth passage. ADMA was replaced every 48 hours starting at the fourth passage. ADMA...

Journal: :Circulation 2015
Laura Dowsett Sophie Piper Anna Slaviero Neil Dufton Zhen Wang Olga Boruc Matthew Delahaye Lucy Colman Eliza Kalk James Tomlinson Graeme Birdsey Anna M Randi James Leiper

BACKGROUND Asymmetrical dimethylarginine (ADMA) is an endogenous inhibitor of nitric oxide synthesis and a risk factor for cardiovascular disease. Dimethylarginine dimethylaminohydrolase (DDAH) enzymes are responsible for ADMA breakdown. It has been reported that endothelial DDAH1 accounts for the majority of ADMA metabolism. However, we and others have shown strong DDAH1 expression in a range ...

2014
Carla S. Jung Christian Wispel Klaus Zweckberger Christopher Beynon Daniel Hertle Oliver W. Sakowitz Andreas W. Unterberg

Previous results on nitric oxide (NO) metabolism after traumatic brain injury (TBI) show variations in NO availability and controversial effects of exogenous nitric oxide synthase (NOS)-inhibitors. Furthermore, elevated levels of the endogenous NOS inhibitor asymmetric dimethylarginine (ADMA) were reported in cerebro-spinal fluid (CSF) after traumatic subarachnoid hemorrhage (SAH). Therefore, w...

2009
Agua Sobrino Manuel Mata Andrés Laguna-Fernandez Susana Novella Pilar J. Oviedo Miguel Angel García-Pérez Juan J. Tarín Antonio Cano Carlos Hermenegildo

Vascular effects of estradiol are being investigated because there are controversies among clinical and experimental studies. DNA microarrays were used to investigate global gene expression patterns in cultured human umbilical vein endothelial cells (HUVEC) exposed to 1 nmol/L estradiol for 24 hours. When compared to control, 187 genes were identified as differentially expressed with 1.9-fold c...

2012
Karsten Sydow Christine Schmitz Eike-Christin von Leitner Robin von Leitner Anna Klinke Dorothee Atzler Christian Krebs Hartwig Wieboldt Heimo Ehmke Edzard Schwedhelm Thomas Meinertz Stefan Blankenberg Rainer H. Böger Tim Magnus Stephan Baldus Ulrich Wenzel

BACKGROUND The endogenous nitric oxide synthase inhibitor asymmetric dimethylarginine (ADMA) is an independent predictor of cardiovascular and overall mortality. Moreover, elevated ADMA plasma concentrations are associated with the extent of hypertension. However, data from small-sized clinical trials and experimental approaches using murine transgenic models have revealed conflicting results r...

Journal: :World journal of gastroenterology 2008
Michiel P C Siroen Reiner Wiest Milan C Richir Tom Teerlink Jan A Rauwerda Friedrich T Drescher Niels Zorger Paul A M van Leeuwen

AIM To analyze the change of dimethylarginine plasma levels in cirrhotic patients receiving transjugular intrahepatic portosystemic shunt (TIPS). METHODS To determine arginine, asymmetric dimethylarginine (ADMA), symmetric dimethylarginine (SDMA), and nitric oxide (NO) plasma levels, blood samples were collected from the superior cava, hepatic, and portal vein just before, directly after, and...

Journal: :Hypertension 2010
Zaiming Luo Tom Teerlink Kathy Griendling Shakil Aslam William J Welch Christopher S Wilcox

Asymmetrical dimethylarginine inhibits nitric oxide synthase, cationic amino acid transport, and endothelial function. Patients with cardiovascular risk factors often have endothelial dysfunction associated with increased plasma asymmetrical dimethylarginine and markers of reactive oxygen species. We tested the hypothesis that reactive oxygen species, generated by nicotinamide adenine dinucleot...

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