نتایج جستجو برای: coronavirus sars cov 2

تعداد نتایج: 2561448  

Journal: :Computational biology and chemistry 2007
Yuan Zhang Nan Zheng Peng Nan Ying Cao Masami Hasegawa Yang Zhong

As a critical adaptive mechanism, amino acid replacements on the severe acute respiratory syndrome coronavirus (SARS-CoV) spike protein could alter the receptor-binding specificity of this envelope glycoprotein and in turn lead to the emergence or reemergence of this viral zoonosis. Based on the X-ray structures of SARS-CoV spike receptor-binding domain (RBD) in complex with its functional rece...

Journal: :Journal of virology 2009
Miyuki Kawase Kazuya Shirato Shutoku Matsuyama Fumihiro Taguchi

Human coronavirus 229E, classified as a group I coronavirus, utilizes human aminopeptidase N (APN) as a receptor; however, its entry mechanism has not yet been fully elucidated. We found that HeLa cells infected with 229E via APN formed syncytia when treated with trypsin or other proteases but not in a low-pH environment, a finding consistent with syncytium formation by severe acute respiratory...

Journal: :Virology 2009
Craig W Day Ralph Baric Sui Xiong Cai Matt Frieman Yohichi Kumaki John D Morrey Donald F Smee Dale L Barnard

Severe acute respiratory syndrome (SARS) is a highly lethal emerging disease caused by coronavirus SARS-CoV. New lethal animal models for SARS were needed to facilitate antiviral research. We adapted and characterized a new strain of SARS-CoV (strain v2163) that was highly lethal in 5- to 6-week-old BALB/c mice. It had nine mutations affecting 10 amino acid residues. Strain v2163 increased IL-1...

Journal: :Journal of virology 2005
Martin Spiegel Andreas Pichlmair Luis Martínez-Sobrido Jerome Cros Adolfo García-Sastre Otto Haller Friedemann Weber

Severe acute respiratory syndrome (SARS) is caused by a novel coronavirus termed SARS-CoV. We and others have previously shown that the replication of SARS-CoV can be suppressed by exogenously added interferon (IFN), a cytokine which is normally synthesized by cells as a reaction to virus infection. Here, we demonstrate that SARS-CoV escapes IFN-mediated growth inhibition by preventing the indu...

2018
Maria L. Agostini Erica L. Andres Amy C. Sims Rachel L. Graham Timothy P. Sheahan Xiaotao Lu Everett Clinton Smith James Brett Case Joy Y. Feng Robert Jordan Adrian S. Ray Tomas Cihlar Dustin Siegel Richard L. Mackman Michael O. Clarke Ralph S. Baric Mark R. Denison

Emerging coronaviruses (CoVs) cause severe disease in humans, but no approved therapeutics are available. The CoV nsp14 exoribonuclease (ExoN) has complicated development of antiviral nucleosides due to its proofreading activity. We recently reported that the nucleoside analogue GS-5734 (remdesivir) potently inhibits human and zoonotic CoVs in vitro and in a severe acute respiratory syndrome co...

Journal: :Journal of molecular biology 2004
Rong Zeng Rui-Fu Yang Mu-De Shi Man-Rong Jiang You-Hua Xie Hong-Qiang Ruan Xiao-Sheng Jiang Lv Shi Hu Zhou Lei Zhang Xiao-Dong Wu Ying Lin Yong-Yong Ji Lei Xiong Yan Jin Er-Hei Dai Xiao-Yi Wang Bin-Ying Si Jin Wang Hong-Xia Wang Cui-E Wang Yong-Hua Gan Yu-Chuan Li Ju-Tian Cao Jiang-Ping Zuo Shi-Fang Shan En Xie Song-Hua Chen Zhi-Qin Jiang Xi Zhang Yuan Wang Gang Pei Bing Sun Jia-Rui Wu

Proteomics was used to identify a protein encoded by ORF 3a in a SARS-associated coronavirus (SARS-CoV). Immuno-blotting revealed that interchain disulfide bonds might be formed between this protein and the spike protein. ELISA indicated that sera from SARS patients have significant positive reactions with synthesized peptides derived from the 3a protein. These results are concordant with that ...

2003
Ken Yan Ching Chow Chung Chau Hon Raymond Kin Hi Hui Raymond Tsz Yeung Wong Chi Wai Yip Fanya Zeng Frederick Chi Ching Leung

The sudden outbreak of severe acute respiratory syndrome (SARS) in 2002 prompted the establishment of a global scientific network subsuming most of the traditional rivalries in the competitive field of virology. Within months of the SARS outbreak, collaborative work revealed the identity of the disastrous pathogen as SARS-associated coronavirus (SARS-CoV). However, although the rapid identifica...

Journal: :PLoS Medicine 2006
Jan ter Meulen Edward N van den Brink Leo L. M Poon Wilfred E Marissen Cynthia S. W Leung Freek Cox Chung Y Cheung Arjen Q Bakker Johannes A Bogaards Els van Deventer Wolfgang Preiser Hans Wilhelm Doerr Vincent T Chow John de Kruif Joseph S. M Peiris Jaap Goudsmit

BACKGROUND Experimental animal data show that protection against severe acute respiratory syndrome coronavirus (SARS-CoV) infection with human monoclonal antibodies (mAbs) is feasible. For an effective immune prophylaxis in humans, broad coverage of different strains of SARS-CoV and control of potential neutralization escape variants will be required. Combinations of virus-neutralizing, noncomp...

Journal: :Viral immunology 2011
Sheng-Fan Wang Kuan-Hsuan Chen Marcelo Chen Wen-Yi Li Yen-Ju Chen Ching-Han Tsao Muh-yong Yen Jason C Huang Yi-Ming Arthur Chen

One-hundred and thirty confirmed cases of severe acute respiratory syndrome (SARS) were recruited to evaluate their anti-SARS-coronavirus (CoV) antibody status and human leukocyte antigen (HLA) types in September 2006, 3 y after the SARS outbreaks in Taiwan. Western blot assay showed that 6.9% of participants still had anti-spike and anti-nucleocapside antibodies. A case-control study of the as...

2004
Wenbing Chen Shousong Li Biying Shao Teng Zheng Shuxun Jiang Xiaorong Huang Kaizhen Cai Zhideng Zhang

The multiplex polymerase chain reaction (PCR) technique was applied to detect the SARS-CoV (severe acute respiratory syndrome-associated coronavirus) specific target cDNA fragments in the present study. The target cDNA fragments of SARS-CoV were synthesized artificially according to the genome sequence of SARS-CoV in GenBank submitted by The Chinese University of Hong Kong, and were used as sim...

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