نتایج جستجو برای: apaf1

تعداد نتایج: 548  

2001
Monika Lachner

Recombinant WT and mutant caspase-9 proteins (20 nM) were incubated in the presence or absence of recombinant Apaf-1 (20 nM) in buffer A. The reaction mixtures were stimulated with cytochrome c (5 ng ml) and dATP (1 mM) and incubated for 0±60 min at 30 8C with puri®ed procaspase-3 C163A, and the processing of procaspase-3 was analysed by western blot analysis with anti-caspase-3 antibody (Fig. ...

Journal: :The Journal of neuroscience : the official journal of the Society for Neuroscience 2005
Eric C C Cheung Lysanne Melanson-Drapeau Sean P Cregan Jacqueline L Vanderluit Kerry L Ferguson William C McIntosh David S Park Steffany A L Bennett Ruth S Slack

Mitochondria release proteins that propagate both caspase-dependent and caspase-independent cell death pathways. AIF (apoptosis-inducing factor) is an important caspase-independent death regulator in multiple neuronal injury pathways. Presently, there is considerable controversy as to whether AIF is neuroprotective or proapoptotic in neuronal injury, such as oxidative stress or excitotoxicity. ...

2015
Yong Tang Ya-chen Zhang Yu Chen Yin Xiang Cheng-xing Shen Yi-gang Li

The biological effects of microRNAs (miRNAs) and TNF-α in atherosclerosis have been widely studied. The circulating miR-17-92 cluster has been recently shown to be significantly downregulated in patients with injured vascular endothelium. However, it remains unclear whether the miR-17-92 cluster plays a significant role in vascular endothelial repair. The aim of this study was to investigate th...

Journal: :The Journal of biological chemistry 2004
Gen Nishitai Nao Shimizu Takahiro Negishi Hiroyuki Kishimoto Kentaro Nakagawa Daiju Kitagawa Tomomi Watanabe Haruka Momose Shinya Ohata Shuhei Tanemura Satoshi Asaka Junko Kubota Ryota Saito Hiroki Yoshida Tak W Mak Teiji Wada Josef M Penninger Noriyuki Azuma Hiroshi Nishina Toshiaki Katada

SAPK/JNK, which belongs to the family of mitogen-activated protein kinase (MAPK), is activated by many types of cellular stresses or extracellular signals and is involved in embryonic development, immune responses, and cell survival or apoptosis. However, the physiological roles of SAPK/JNK in the signaling of stress-induced apoptosis are still controversial. To evaluate the precise function, S...

Journal: :The Journal of biological chemistry 2006
Federica Di Sano Elisabetta Ferraro Roberta Tufi Tilmann Achsel Mauro Piacentini Francesco Cecconi

The endoplasmic reticulum (ER) is the cellular site of polypeptide folding and modification. When these processes are hampered, an unfolded protein response (UPR) is activated. If the damage is too broad, the mammalian UPR launches the apoptotic program. As a consequence, mobilization of ER calcium stores sensitizes mitochondria to direct proapoptotic stimuli. We make use of a mouse Apaf1-defic...

Journal: :Cancer research 2004
Akihide Fujimoto Steven J O'Day Bret Taback David Elashoff Dave S B Hoon

Allelic imbalance (AI) encompassing the apoptotic protease-activating factor 1 (APAF-1) locus (12q22-23) is found frequently in metastatic melanoma. Circulating DNA with AI on 12q22-23 in serum was evaluated as a surrogate marker to predict biochemotherapy (BC) treatment response in melanoma patients. Sera were collected from 49 American Joint Committee on Cancer stage IV melanoma patients trea...

Journal: :Current opinion in plant biology 1998
J Ellis D Jones

Recently recognised structural and amino acid sequence similarities between plant disease resistance (R) proteins and animal proteins such as Apaf-1 and CED-4 are providing conceptual models for resistance protein function. Data from extensive DNA sequencing of resistance gene families are indicating that the leucine-rich repeat motif is an important determinant of gene-for-gene specificity and...

Journal: :European journal of medicinal chemistry 2013
Miriam Corredor Jordi Bujons Mar Orzáez Mónica Sancho Enrique Pérez-Payá Ignacio Alfonso Angel Messeguer

Apoptosis is a biological process relevant to several human diseases that is strongly regulated through protein-protein complex formation. We have previously reported a peptidomimetic compound as potent apoptotic modulator. Structural studies of this compound showed the presence of cis/trans isomers of the exocyclic tertiary amide bond in slow exchange. This information encouraged us to perform...

Journal: :Molecular and cellular neurosciences 2005
Muhammad Z Khan Saori Shimizu Jeegar P Patel Autumn Nelson My-Thao Le Anna Mullen-Przeworski Renato Brandimarti Alessandro Fatatis Olimpia Meucci

Abnormal activation of CXCR 4 during inflammatory/infectious states may lead to neuronal dysfunction or damage. The major goal of this study was to determine the coupling of CXCR 4 to p53-dependent survival pathways in primary neurons. Neurons were stimulated with the HIV envelope protein gp120(IIIB) or the endogenous CXCR 4 agonist, SDF-1 alpha. We found that gp120 stimulates p53 activity and ...

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