نتایج جستجو برای: 642 and 496

تعداد نتایج: 16827814  

Journal: :Circulation 2000
H Strömer M C de Groot M Horn C Faul A Leupold J P Morgan W Scholz S Neubauer

BACKGROUND Na(+)/H(+) exchange inhibition with HOE642 (cariporide) improves postischemic recovery of cardiac function, but the mechanisms of action remain speculative. Because Na(+)/H(+) exchange is activated on reperfusion, it was hypothesized that its inhibition delays realkalinization and decreases intracellular Na(+) and, via Na(+)/Ca(2+) exchange, Ca(2+) overload. Attenuated Ca(2+) overloa...

Journal: :American journal of physiology. Heart and circulatory physiology 2000
C Schäfer Y V Ladilov B Siegmund H M Piper

Isolated cardiomyocytes from adult rats were incubated in anoxic bicarbonate-buffered media at extracellular pH (pH(o)) 6.4 until a cytosolic Ca(2+) overload and intracellular pH (pH(i)) of 6.4 were reached. On reoxygenation, the pH of the medium was changed to 7.4 to activate the Na(+)/H(+)exchanger (NHE) and the Na(+)-HCO(-)(3) symporter (NBS). The reoxygenation was performed in the absence o...

Journal: :Cardiovascular research 2000
X H Xiao D G Allen

OBJECTIVE Removal of protons from the heart during ischemia and/or reperfusion by the cardiac Na(+)/H(+) exchanger (NHE1) leads to Na(+) entry; this causes Ca(2+) influx and is thought to contribute to ischemic and/or reperfusion damage. The extent to which Na(+) enters during ischemia as opposed to reperfusion is disputed and has important implications for the therapeutic use of NHE1 inhibitor...

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