نتایج جستجو برای: سنجنده irs

تعداد نتایج: 7324  

Journal: :The Biochemical journal 2005
Ingeborg Hers Jeremy M Tavaré

Serine and threonine phosphorylation of IRS-1 (insulin receptor substrate-1) has been reported to decrease its ability to be tyrosine-phosphorylated by the insulin receptor. Insulin itself may negatively regulate tyrosine phosphorylation of IRS-1 through a PI3K (phosphoinositide 3-kinase)-dependent feedback pathway. In the present study, we examined the regulation and role of IRS-1 serine phosp...

2003
Massimo Federici Antonio Petrone Ottavia Porzio Carla Bizzarri Davide Lauro Rossella D’Alfonso Ippolita Patera Marco Cappa Lorenza Nisticò Marco Baroni Giorgio Sesti Umberto di Mario Renato Lauro Raffaella Buzzetti

The Arg insulin receptor substrate-1 (IRS-1) variant has been hypothesized to play a role in pancreatic -cell stimulus-coupled insulin secretion and survival. We analyzed the relations between type 1 diabetes and the Arg IRS-1 variant. The frequency of the IRS-1 Arg variant was investigated in two independent sets of unrelated patients: a case-control study and a collection of type 1 diabetes s...

Journal: :The Iowa Review 2004

Journal: :Arteriosclerosis, Thrombosis, and Vascular Biology 2000

Journal: :IEEE Signal Processing Letters 2022

Intelligent reflecting surface (IRS) is a rapidly emerging paradigm to enable non-line-of-sight (NLoS) wireless transmission. In this paper, we focus on IRS-aided radar estimation performance of moving hidden or NLoS target. Unlike prior works that employ single IRS, investigate problem using multiple IRS platforms and assess the by deriving associated Cramér-Rao lower bound (CRLB). We then des...

2017
Leonard H. Calabrese Arthur Kavanaugh Anthony E. Yeo Peter E. Lipsky

BACKGROUND To assess frequency and distribution of infusion reactions (IRs) in responders and nonresponders in randomized clinical trials (RCTs) of intravenous pegloticase and the utility of the National Institute of Allergy and Infectious Disease/Food and Allergy and Anaphylaxis Network (NIAID/FAAN) criteria for identifying anaphylaxis in subjects experiencing IRs. METHODS IRs from two RCTs ...

2010
Diana Gurevitch Sigalit Boura-Halfon Roi Isaac Galit Shahaf Moti Alberstein Denise Ronen Eli C. Lewis Yehiel Zick

OBJECTIVE Cellular stress and proinflammatory cytokines induce phosphorylation of insulin receptor substrate (IRS) proteins at Ser sites that inhibit insulin and IGF-1 signaling. Here, we examined the role of Ser phosphorylation of IRS-2 in mediating the inhibitory effects of proinflammatory cytokines and cellular stress on beta-cell function. RESEARCH DESIGN AND METHODS Five potential inhibi...

Journal: :Archives of disease in childhood 2005
R M Viner T Y Segal E Lichtarowicz-Krynska P Hindmarsh

AIMS To assess prevalence of the insulin resistance syndrome (IRS: obesity, abnormal glucose homoeostasis, dyslipidaemia, and hypertension) in obese UK children and adolescents of different ethnicities and to assess whether fasting data is sufficient to identify IRS in childhood obesity. METHODS A total of 103 obese (BMI >95th centile) children and adolescents 2-18 years of age referred for a...

Journal: :Molecular medicine 2002
Cecilia Annerén Michael Welsh

BACKGROUND Insulin receptor substrate proteins (IRS) mediate various effects of insulin, including regulation of glucose homeostasis, cell growth and survival. To understand the underlying mechanisms explaining the effects of the Src-related tyrosine kinase GTK on beta-cell proliferation and survival, insulin-signalling pathways involving IRS-1 and IRS-2 were studied in islet cells and RINm5F c...

Journal: :The Journal of clinical investigation 2012
Konrad Talbot Hoau-Yan Wang Hala Kazi Li-Ying Han Kalindi P Bakshi Andres Stucky Robert L Fuino Krista R Kawaguchi Andrew J Samoyedny Robert S Wilson Zoe Arvanitakis Julie A Schneider Bryan A Wolf David A Bennett John Q Trojanowski Steven E Arnold

While a potential causal factor in Alzheimer's disease (AD), brain insulin resistance has not been demonstrated directly in that disorder. We provide such a demonstration here by showing that the hippocampal formation (HF) and, to a lesser degree, the cerebellar cortex in AD cases without diabetes exhibit markedly reduced responses to insulin signaling in the IR→IRS-1→PI3K signaling pathway wit...

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