نتایج جستجو برای: progeria

تعداد نتایج: 858  

Journal: :Biochemical Society transactions 2011
Fernando G Osorio Alejandro P Ugalde Guillermo Mariño Xose S Puente José M P Freije Carlos López-Otín

Progeroid laminopathies are accelerated aging syndromes caused by defects in nuclear envelope proteins. Accordingly, mutations in the LMNA gene and functionally related genes have been described to cause HGPS (Hutchinson-Gilford progeria syndrome), MAD (mandibuloacral dysplasia) or RD (restrictive dermopathy). Functional studies with animal and cellular models of these syndromes have facilitate...

2013
Baohua Liu Zimei Wang Le Zhang Shrestha Ghosh Huiling Zheng Zhongjun Zhou

A de novo G608G mutation in LMNA gene leads to Hutchinson-Gilford progeria syndrome. Mice lacking the prelamin A-processing metalloprotease, Zmpste24, recapitulate many of the progeroid features of Hutchinson-Gilford progeria syndrome. Here we show that A-type lamins interact with SUV39H1, and prelamin A/progerin exhibits enhanced binding capacity to SUV39H1, protecting it from proteasomal degr...

Journal: :Orphanet Journal of Rare Diseases 2010

Journal: :Archives of Disease in Childhood 1955

Journal: :Hypertension 2012
Marie Gerhard-Herman Leslie B Smoot Nicole Wake Mark W Kieran Monica E Kleinman David T Miller Armin Schwartzman Anita Giobbie-Hurder Donna Neuberg Leslie B Gordon

Hutchinson-Gilford progeria syndrome is a rare, segmental premature aging syndrome of accelerated atherosclerosis and early death from myocardial infarction or stroke. This study sought to establish comprehensive characterization of the fatal vasculopathy in Hutchinson-Gilford progeria syndrome and its relevance to normal aging. We performed cardiovascular assessments at a single clinical site ...

2013
Jorge de la Rosa José M. P. Freije Rubén Cabanillas Fernando G. Osorio Mario F. Fraga M. Soledad Fernández-García Roland Rad Víctor Fanjul Alejandro P. Ugalde Qi Liang Haydn M. Prosser Allan Bradley Juan Cadiñanos Carlos López-Otín

Defining the relationship between ageing and cancer is a crucial but challenging task. Mice deficient in Zmpste24, a metalloproteinase mutated in human progeria and involved in nuclear prelamin A maturation, recapitulate multiple features of ageing. However, their short lifespan and serious cell-intrinsic and cell-extrinsic alterations restrict the application and interpretation of carcinogenes...

2017
KAMALDEEP SINGH

Progeria also known Hutchinson–Gilford progeria syndrome (HGPS), is an extremely rare genetic disorder. The prevalence of HGPS is 1 in 4-8 million newborns. Progeria causes premature, rapid aging shortly after birth present within the first year of life. Recently, de novo point mutations in the Lmna gene at position 1824 of the coding sequence have been found in persons with HGPS. Lmna encodes ...

Journal: :Archives of disease in childhood 1970
B G Macnamara K T Farn A K Mitra J K Lloyd A S Fosbrooke

Macnamara, B. G. P., Farn, K. T., Mitra, A. K., Lloyd, J. K., and Fosbrooke, A. S. (1970). Archives of Disease in Childhood, 45, 553. Progeria: case report with long-term studies of serum lipids. A girl with the typical features of progeria died at the age of 10 years from cardiac failure following repeated myocardial infarctions. Serial studies of the serum lipids were made from the age of 2 y...

2011
Marie Gerhard-Herman Leslie B. Smoot Nicole Wake Mark W. Kieran Monica E. Kleinman David T. Miller Armin Schwartzman Anita Giobbie-Hurder Donna Neuberg Leslie B. Gordon

Hutchinson-Gilford progeria syndrome is a rare, segmental premature aging syndrome of accelerated atherosclerosis and early death from myocardial infarction or stroke. This study sought to establish comprehensive characterization of the fatal vasculopathy in Hutchinson-Gilford progeria syndrome and its relevance to normal aging. We performed cardiovascular assessments at a single clinical site ...

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