Inflammatory gingival enlargement is an excessive growth of tissue due to inflammation in response plaque accumulation. Enlargement gingiva hampers the efficiency daily oral hygiene practices which turn accumulates more plaque, thus causing inflammation. Phase I therapy, consisting scaling and root planning only reduces inflammatory component enlarged tissues but fibrotic remaining thereafter t...

Clinical Feasibility of Molecular Imaging of Plaque Inflammation in Atherosclerosis Molecular imaging of various components of atherosclerotic plaques has been proposed, and proof of principle has been demonstrated in experimental models of disease (1). These preclinical studies have predominantly targeted plaque inflammation with the premise that the extent of inflammation would determine the ...

Clinical Feasibility of Molecular Imaging of Plaque Inflammation in Atherosclerosis M olecular imaging of various components of atherosclerotic plaques has been proposed, and proof of principle has been demonstrated in experimental models of disease (1). These preclinical studies have predominantly targeted plaque inflammation with the premise that the extent of inflammation would determine the...

Clinical Feasibility of Molecular Imaging of Plaque Inflammation in Atherosclerosis Molecular imaging of various components of atherosclerotic plaques has been proposed, and proof of principle has been demonstrated in experimental models of disease (1). These preclinical studies have predominantly targeted plaque inflammation with the premise that the extent of inflammation would determine the ...

OBJECTIVES This study was designed to determine whether arterial remodeling and plaque vulnerability are influenced by systemic factors. BACKGROUND Atherosclerotic luminal narrowing is caused by gradual plaque growth and arterial remodeling. In the acute phase, luminal narrowing may be accelerated by acute thrombus formation, usually precipitated by rupture of a vulnerable plaque. METHODS F...

Diabetes mellitus, the major cardiovascular risk factor, accentuates the inflammation and neovascularization processes leading to enhanced progression of atherosclerotic complications. Inflammation in diabetes mellitus is the key initiator of atherosclerotic process, which results in acute coronary events. Atherosclerosis evolves from the endothelial cell dysfunction and succeeding entry of hem...

Diabetes mellitus is increasing worldwide, resulting from the interaction of obesity, inflammation, and hyperglycemia. Activated immunity and cytokine production lead to insulin resistance and other components of the metabolic syndrome, establishing the link between diabetes and atherosclerosis. Hyperglycemia-induced endothelial dysfunction is mediated by increased oxidative stress, a promoter ...