نتایج جستجو برای: paroxysmal nocturnal hemoglobinuria

تعداد نتایج: 26047  

2011
Norio Nakamura Toshiyuki Sugawara Ken-ichi Shirato Ryuichiro Kumasaka Masayuki Nakamura Michiko Shimada Takeshi Fujita Reiichi Murakami Yuko Shimaya Hiroshi Osawa Hideaki Yamabe Ken Okumura

INTRODUCTION Paroxysmal nocturnal hemoglobinuria is an acquired disorder of hemopoiesis and is characterized by recurrent episodes of intravascular hemolysis due to an increased sensitivity to complement-mediated hemolysis. Systemic lupus erythematosus with paroxysmal nocturnal hemoglobinuria is very rare. We report a case of paroxysmal nocturnal hemoglobinuria that developed in a patient with ...

2011
Lacy Cardoso de Brito Junior Maria do Socorro de Oliveira Cardoso Euzamar Gaby Rocha Herika Anijar Mariana Cunha João Carlos Pina Saraiva

BACKGROUND Paroxysmal nocturnal hemoglobinuria is a hematological disease with complex physiopathology. It is genetically characterized by a somatic mutation in the PIG-A gene (phosphatidylinositol glycan anchor biosynthesis, class A), in which the best known antigens are DAF (decay accelerating factor or CD55) and MIRL (membrane inhibitor of reactive lysis or CD59). OBJECTIVE To determine th...

Journal: :Haematologica 2010
Phillip Scheinberg Michael Marte Olga Nunez Neal S Young

BACKGROUND Clones of glycosylphosphatidylinositol-anchor protein-deficient cells are characteristic in paroxysmal nocturnal hemoglobinuria and are present in about 40-50% of patients with severe aplastic anemia. Flow cytometry has allowed for sensitive and precise measurement of glycosylphosphatidylinositol-anchor protein-deficient red blood cells and neutrophils in severe aplastic anemia. DE...

Journal: :Blood 2009
Sara Pelucchi Raffaella Mariani Francesca Bertola Cristina Arosio Alberto Piperno

22:1-15. 8. Thomas TC, Rollins SA, Rother RP, et al. Inhibition of complement activity by humanized anti-C5 antibody and single-chain Fv. Mol Immunol. 1996;33:13891401. 9. Rother RP, Rollins SA, Mojcik CF, Brodsky RA, Bell L. Discovery and development of the complement inhibitor eculizumab for the treatment of paroxysmal nocturnal hemoglobinuria. Nat Biotechnol. 2007;25:1256-1264. 10. Hillmen P...

Journal: :Hematology, Transfusion and Cell Therapy 2020

Journal: :Nihon Naika Gakkai Zasshi 2012

Journal: :Nihon Naika Gakkai Zasshi 2012

Journal: :Haematologica 2012
David J Araten Rosario Notaro Howard T Thaler Nancy Kernan Farid Boulad Hugo Castro-Malaspina Trudy Small Andromachi Scaradavou Heather Magnan Susan Prockop Sara Chaffee Jason Gonsky Raymond Thertulien Roberto Tarquini Lucio Luzzatto

BACKGROUND Thrombosis is the major risk factor for death in patients with paroxysmal nocturnal hemoglobinuria. Previous case reports indicate that venous thrombosis in patients with paroxysmal nocturnal hemoglobinuria is amenable to thrombolysis. DESIGN AND METHODS We reviewed the outcome of thrombolytic therapy for patients with paroxysmal nocturnal hemoglobinuria who had thromboses refracto...

Journal: :Haematologica 2014
Tommaso Rondelli Antonio M Risitano Régis Peffault de Latour Michela Sica Benedetta Peruzzi Patrizia Ricci Wilma Barcellini Anna Paola Iori Carla Boschetti Veronica Valle Veronique Frémeaux-Bacchi Maria De Angioletti Gerard Socie Lucio Luzzatto Rosario Notaro

Complement blockade by eculizumab is clinically effective in hemolytic paroxysmal nocturnal hemoglobinuria. However, the response is variable and some patients remain dependent on red blood cell transfusions. In 72 patients with hemolytic paroxysmal nocturnal hemoglobinuria on eculizumab we tested the hypothesis that response may depend on genetic polymorphisms of complement-related genes. We f...

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