نتایج جستجو برای: ewing

تعداد نتایج: 2735  

2017
Nathan M Cross A Luana Stanescu Erin R Rudzinski Douglas S Hawkins Marguerite T Parisi

Ewing sarcoma, including classical Ewing sarcoma of the bone and primitive neuroectodermal tumors arising in bone or extraosseous primary sites, is a highly aggressive childhood neoplasm. We present two cases of Ewing sarcoma arising from the vagina in young girls. Previously reported cases in literature focused on their pathologic rather than radiographic features. We describe the spectrum of ...

Journal: :Clinical cancer research : an official journal of the American Association for Cancer Research 2004
Iranzu González Enrique J Andreu Angel Panizo Susana Inogés Ana Fontalba José Luis Fernández-Luna Mirella Gaboli Luis Sierrasesúmaga Salvador Martín-Algarra Javier Pardo Felipe Prósper Enrique de Alava

PURPOSE AND EXPERIMENTAL DESIGN The stem cell factor/KIT receptor loop may represent a novel target for molecular-based therapies of Ewing tumor. We analyzed the in vitro impact of KIT blockade by imatinib in Ewing tumor cell lines. RESULTS KIT expression was detected in 4 of 4 Ewing tumor cell lines and in 49 of 110 patient samples (44.5%) by immunohistochemistry and/or Western blot analysis...

Journal: :Clinical cancer research : an official journal of the American Association for Cancer Research 2011
Kathleen I Pishas Fares Al-Ejeh Irene Zinonos Raman Kumar Andreas Evdokiou Michael P Brown David F Callen Paul M Neilsen

PURPOSE Although mutations in the TP53 gene occur in half of all cancers, approximately 90% of Ewing sarcomas retain a functional wild-type p53. The low frequency of TP53 alterations in Ewing sarcoma makes this tumor type an ideal candidate for p53-targeted therapies. In this study, we have examined the molecular and cellular responses of cultured Ewing sarcoma cell lines following exposure to ...

Journal: :Molecular cancer research : MCR 2012
Tyler P Robin Anna Smith Erin McKinsey Lisa Reaves Paul Jedlicka Heide L Ford

Ewing sarcoma is an aggressive pediatric cancer of the bone and soft tissue, in which patients whose tumors have a poor histologic response to initial chemotherapy have a poor overall prognosis. Therefore, it is important to identify molecules involved in resistance to chemotherapy. Herein, we show that the DNA repair protein and transcriptional cofactor, EYA3, is highly expressed in Ewing sarc...

2016
Kelly M. Bailey Merlin Airik Melanie A. Krook Elisabeth A. Pedersen Elizabeth R. Lawlor

Metastatic Ewing sarcoma has a very poor prognosis and therefore new investigations into the biologic drivers of metastatic progression are key to finding new therapeutic approaches. The tumor microenvironment is highly dynamic, leading to exposure of different regions of a growing solid tumor to changes in oxygen and nutrient availability. Tumor cells must adapt to such stress in order to surv...

Journal: :Cancer research 2013
Brian D Crompton Anne L Carlton Aaron R Thorner Amanda L Christie Jinyan Du Monica L Calicchio Miguel N Rivera Mark D Fleming Nancy E Kohl Andrew L Kung Kimberly Stegmaier

Limited progress has been made in the treatment of advanced-stage pediatric solid tumors despite the accelerated pace of cancer discovery over the last decade. Tyrosine kinase inhibition is one tractable therapeutic modality for treating human malignancy. However, little is known about the kinases critical to the development or maintenance of many pediatric solid tumors such as Ewing sarcoma. U...

Journal: :Annals of Saudi medicine 1993
A Jamjoom N Ur-Rahman M A Hafeez Z A Jamjoom

Ewing sarcoma is a primary bone tumor which was originally described by Lucke in 1866 but was defined in such detail by Sir James Ewing in 1921 that it is now associated with his name [1]. This tumor involves the spine primarily in 3.5% to 14.9% r ~ ~ases [2,3]. The review of 22 cases of Ewing Sarcoma of the spine by Pilepich et al in 1981 [4] remains the largest study of this pathology in such...

Journal: :Molecular cancer therapeutics 2013
Hae-June Lee Changhwan Yoon Benjamin Schmidt Do Joong Park Alexia Y Zhang Hayriye V Erkizan Jeffrey A Toretsky David G Kirsch Sam S Yoon

Ewing sarcomas (ES) harbor a chromosomal translocation that fuses the EWS gene to an ETS transcription factor, most commonly Friend leukemia integration 1 (FLI1). The EWS-FLI1 fusion protein acts in a positive feedback loop to maintain the expression of PARP-1, which is involved in repair of DNA damage. Here, we examine the effects of PARP-1 inhibition and radiation therapy on Ewing sarcomas. I...

Journal: :Clinical cancer research : an official journal of the American Association for Cancer Research 2015
Konstantin Agelopoulos Günther H S Richter Eva Schmidt Uta Dirksen Kristina von Heyking Benjamin Moser Hans-Ulrich Klein Udo Kontny Martin Dugas Kathrin Poos Eberhard Korsching Thorsten Buch Matthias Weckesser Isabell Schulze Regina Besoke Anika Witten Monika Stoll Gabriele Köhler Wolfgang Hartmann Eva Wardelmann Claudia Rossig Daniel Baumhoer Heribert Jürgens Stefan Burdach Wolfgang E Berdel Carsten Müller-Tidow

PURPOSE A low mutation rate seems to be a general feature of pediatric cancers, in particular in oncofusion gene-driven tumors. Genetically, Ewing sarcoma is defined by balanced chromosomal EWS/ETS translocations, which give rise to oncogenic chimeric proteins (EWS-ETS). Other contributing somatic mutations involved in disease development have only been observed at low frequency. EXPERIMENTAL...

2011
Kathleen I. Pishas Fares Al-Ejeh Irene Zinonos Raman Kumar Andreas Evdokiou Michael P. Brown David F. Callen Paul M. Neilsen

Purpose: Althoughmutations in the TP53 gene occur in half of all cancers, approximately 90% of Ewing sarcomas retain a functional wild-type p53. The low frequency of TP53 alterations in Ewing sarcomamakes this tumor type an ideal candidate for p53-targeted therapies. In this study, we have examined the molecular and cellular responses of cultured Ewing sarcoma cell lines following exposure to N...

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