نتایج جستجو برای: complement c3

تعداد نتایج: 82816  

Journal: :Cell 2006
Young-Sun Kang Yoonkyung Do Hae-Kyung Lee Sung Ho Park Cheolho Cheong Rebecca M. Lynch Jutta M. Loeffler Ralph M. Steinman Chae Gyu Park

The intricate system of serum complement proteins provides resistance to infection. A pivotal step in the complement pathway is the assembly of a C3 convertase, which digests the C3 complement component to form microbial binding C3 fragments recognized by leukocytes. The spleen and C3 provide resistance against blood-borne S. pneumoniae infection. To better understand the mechanisms involved, w...

Journal: :Neoplasia 2012
Selene Nunez-Cruz Phyllis A Gimotty Matthew W Guerra Denise C Connolly You-Qiang Wu Robert A DeAngelis John D Lambris George Coukos Nathalie Scholler

Complement activation plays a critical role in controlling inflammatory responses. To assess the role of complement during ovarian cancer progression, we crossed two strains of mice with genetic complement deficiencies with transgenic mice that develop epithelial ovarian cancer (TgMISIIR-TAg). TgMISIIR-TAg mice fully or partially deficient for complement factor 3 (C3) (Tg(+)C3(KO) and Tg(+)C3(H...

Journal: :Journal of immunology 2010
Osama A Hamad Per H Nilsson Diana Wouters John D Lambris Kristina N Ekdahl Bo Nilsson

It has been reported that complement is activated on the surface of activated platelets, despite the presence of multiple regulators of complement activation. To reinvestigate the mechanisms by which activated platelets bind to complement components, the presence of complement proteins on the surfaces of nonactivated and thrombin receptor-activating peptide-activated platelets was analyzed by f...

2013
Matthew C Pickering Vivette D D'Agati Carla M Nester Richard J Smith Mark Haas Gerald B Appel Charles E Alpers Ingeborg M Bajema Camille Bedrosian Michael Braun Mittie Doyle Fadi Fakhouri Fernando C Fervenza Agnes B Fogo Véronique Frémeaux-Bacchi Daniel P Gale Elena Goicoechea de Jorge Gene Griffin Claire L Harris V Michael Holers Sally Johnson Peter J Lavin Nicholas Medjeral-Thomas B Paul Morgan Cynthia C Nast Laure-Hélène Noel D Keith Peters Santiago Rodríguez de Córdoba Aude Servais Sanjeev Sethi Wen-Chao Song Paul Tamburini Joshua M Thurman Michael Zavros H Terence Cook

C3 glomerulopathy is a recently introduced pathological entity whose original definition was glomerular pathology characterized by C3 accumulation with absent or scanty immunoglobulin deposition. In August 2012, an invited group of experts (comprising the authors of this document) in renal pathology, nephrology, complement biology, and complement therapeutics met to discuss C3 glomerulopathy in...

راسخ جم, علی اکبر, مسعود, احمد ,

Studies were undertaken to evaluate content of IgG, IgM, C3, C4, CH50 and proteines of sera from 53 drug addicts by immunological methods. Results shows an increase of IgG and no significant variatrion in the level of other Ig(S). In the same time we have seen an augmentation of gamma globuline in the protein electrophoresis pattern. The level of C3 and C4 of complement component rest unchanged.

Journal: :FASEB journal : official publication of the Federation of American Societies for Experimental Biology 2006
Conrad A Farrar Wuding Zhou Tao Lin Steven H Sacks

The third complement component (C3) is an acute phase protein that plays a central role in reperfusion injury in several organ models. To investigate the contribution of local synthesis of C3 and distinguish it from that of circulating complement mainly produced by hepatic synthesis, we employed a mouse renal isograft model. Our model demonstrated a close relationship between the extent of intr...

Journal: :Infection and immunity 2001
M X Zhang T T Brandhorst T R Kozel B S Klein

Our previous studies showed that Blastomyces dermatitidis yeast activates the human complement system, leading to deposition of opsonic complement fragments onto the yeast surface. This report examines the influence of altered surface expression of glucan or BAD1 protein (formerly WI-1) on the yeast's ability to activate and bind C3. Compared to the wild type, a glucan-deficient mutant yeast de...

Journal: :Ophthalmology 2012
Dzenita Smailhodzic Caroline C W Klaver B Jeroen Klevering Camiel J F Boon Joannes M M Groenewoud Bernd Kirchhof Mohamed R Daha Anneke I den Hollander Carel B Hoyng

PURPOSE Systemic complement activation is associated with age-related macular degeneration (AMD) and has mainly been attributed to a risk allele in the complement factor H (CFH) gene. Whether other important AMD genes also influence complement activation is unclear. In the present case-control study, complement activity and concentrations of complement components and their activation products a...

2017
Michela Sica Tommaso Rondelli Patrizia Ricci Maria De Angioletti Antonio M Risitano Rosario Notaro

BACKGROUND C5 blockade by eculizumab prevents complement-mediated intravascular hemolysis in paroxysmal nocturnal hemoglobinuria (PNH). However, C3-bound PNH red blood cells (RBCs), arising in almost all treated patients, may undergo extravascular hemolysis reducing clinical benefits. Despite the uniform deficiency of CD55 and of CD59, there are always two distinct populations of PNH RBCs, with...

2007
Michal Hammel Georgia Sfyroera Daniel Ricklin Paola Magotti John D Lambris Brian V Geisbrecht

To provide insight into bacterial suppression of complement-mediated immunity, we present here structures of a bacterial complement inhibitory protein, both free and bound to its complement target. The 1.25-Å structure of the complement component C3–inhibitory domain of Staphylococcus aureus extracellular fibrinogen-binding protein (Efb-C) demonstrated a helical motif involved in complement reg...

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