نتایج جستجو برای: ciliopathy

تعداد نتایج: 423  

Journal: :Reproductive Medicine and Biology 2015

Journal: :Pediatric Neurology Briefs 2013

2015
Sunil Pawar Vinay Zanwar Ashok Mohite Ravindra Surude Pravin Rathi Meenakshi Balasubramani

Congenital hepatic fibrosis is a rare cause of portal hypertension and esophageal varices in children. We report cases of siblings with biopsy proven congenital hepatic fibrosis and with atypical retinitis pigmentosa. They presented with repeated episodes of jaundice along with progressive decrease of vision in night. They had hepatosplenomegaly and portal hypertension with esophageal varices. ...

2015
Tatsuo Miyamoto Shinya Matsuura

The spindle assembly checkpoint (SAC) is a surveillance mechanism of faithful chromosome segregation during mitosis. Budding uninhibited by benzimidazole-related-1 (BubR1) plays a central role in the SAC through inhibition of anaphase promoting complex/cyclosome (APC/C) activity until all chromosomes have established proper attachment to the mitotic spindle. Loss-of-function mutations in the BU...

2016
Chen-Jei Hong Bruce A Hamilton

Zfp423 encodes a 30-zinc finger transcription factor that intersects several canonical signaling pathways. Zfp423 mutations result in ciliopathy-related phenotypes, including agenesis of the cerebellar vermis in mice and Joubert syndrome (JBTS19) and nephronophthisis (NPHP14) in humans. Unlike most ciliopathy genes, Zfp423 encodes a nuclear protein and its developmental expression is complex, l...

Journal: :Human molecular genetics 2012
Matthew Adams Roslyn J Simms Zakia Abdelhamed Helen R Dawe Katarzyna Szymanska Clare V Logan Gabrielle Wheway Eva Pitt Keith Gull Margaret A Knowles Edward Blair Sally H Cross John A Sayer Colin A Johnson

MKS3, encoding the transmembrane receptor meckelin, is mutated in Meckel-Gruber syndrome (MKS), an autosomal-recessive ciliopathy. Meckelin localizes to the primary cilium, basal body and elsewhere within the cell. Here, we found that the cytoplasmic domain of meckelin directly interacts with the actin-binding protein filamin A, potentially at the apical cell surface associated with the basal b...

Journal: :Journal of the American Society of Nephrology 2008

2015
Aoife M Waters Rowan Asfahani Paula Carroll Louise Bicknell Francesco Lescai Alison Bright Estelle Chanudet Anthony Brooks Sonja Christou-Savina Guled Osman Patrick Walsh Chiara Bacchelli Ariane Chapgier Bertrand Vernay David M Bader Charu Deshpande Mary O' Sullivan Louise Ocaka Horia Stanescu Helen S Stewart Friedhelm Hildebrandt Edgar Otto Colin A Johnson Katarzyna Szymanska Nicholas Katsanis Erica Davis Robert Kleta Mike Hubank Stephen Doxsey Andrew Jackson Elia Stupka Mark Winey Philip L Beales

BACKGROUND Mutations in microtubule-regulating genes are associated with disorders of neuronal migration and microcephaly. Regulation of centriole length has been shown to underlie the pathogenesis of certain ciliopathy phenotypes. Using a next-generation sequencing approach, we identified mutations in a novel centriolar disease gene in a kindred with an embryonic lethal ciliopathy phenotype an...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2012
Melissa C Humbert Katie Weihbrecht Charles C Searby Yalan Li Robert M Pope Val C Sheffield Seongjin Seo

Mutations affecting ciliary components cause a series of related genetic disorders in humans, including nephronophthisis (NPHP), Joubert syndrome (JBTS), Meckel-Gruber syndrome (MKS), and Bardet-Biedl syndrome (BBS), which are collectively termed "ciliopathies." Recent protein-protein interaction studies combined with genetic analyses revealed that ciliopathy-related proteins form several funct...

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