conclusions the educational framework provided by im for at training improved attitude and behavioral beliefs that can facilitate the adoption of at behavior and decreased clbp. results significant differences were recorded before and after intervention (p < 0.001) for the model constructs of intention, perceived risk, direct attitude, behavioral beliefs, and knowledge in both groups. direct at...

silver coins of alexander of macedonia, until the arrival of parthians in the 1st century bc, have been put in wdxrf in order to determin ag, cu, pb and au as major and trace elements for fineness, debasement and refinement of silver in ancient persia. seleucid kingdom from 4th to 1st century bc was succeeded by several dynasties and kings. but the chemical composition of their silver coins an...

By considering a (not necessarily locally-flat) PL knot as the singular locus of a PL stratified pseudomanifold, we can use intersection homology theory to define intersection Alexander polynomials, a generalization of the classical Alexander polynomial invariants for smooth or PL locally-flat knots. We show that the intersection Alexander polynomials satisfy certain duality and normalization c...

Freedman, Lovász and Schrijver characterized graph parameters that can be represented as the (weighted) number of homomorphisms into a fixed graph. Several extensions of this result have been proved. We use the framework of categories to prove a general theorem of this kind. Similarly as previous resuts, the characterization uses certain infinite matrices, called connection matrices, which are ...

For a prime p and binomial axk+bxl with 1 ≤ l < k < 1 32 (p−1) 2 3 , we use Stepanov’s method to obtain the bound ∣∣∣∣∣ p−1 ∑ x=1 ep(ax k + bx) ∣∣∣∣∣ max { 1, l∆− 1 3 } 1 4 k 1 4 p 3 4 , where ∆ = k−l (k,l,p−1) .

Alexander disease is a fatal neurological illness characterized by white-matter degeneration and formation of Rosenthal fibers, which contain glial fibrillary acidic protein as astrocytic inclusion. Alexander disease is mainly caused by a gene mutation encoding glial fibrillary acidic protein, although the underlying pathomechanism remains unclear. We established induced pluripotent stem cells ...