نتایج جستجو برای: airway hyper

تعداد نتایج: 107108  

2012
Malcolm R. Starkey Richard Y. Kim Emma L. Beckett Heidi C. Schilter Doris Shim Ama-Tawiah Essilfie Duc H. Nguyen Kenneth W. Beagley Joerg Mattes Charles R. Mackay Jay C. Horvat Philip M. Hansbro

BACKGROUND Viral and bacterial respiratory tract infections in early-life are linked to the development of allergic airway inflammation and asthma. However, the mechanisms involved are not well understood. We have previously shown that neonatal and infant, but not adult, chlamydial lung infections in mice permanently alter inflammatory phenotype and physiology to increase the severity of allerg...

Journal: :International Journal of Basic & Clinical Pharmacology 2014

2015
Nariman A. Balenga Michael Klichinsky Zhihui Xie Eunice C. Chan Ming Zhao Joseph Jude Michel Laviolette Reynold A. Panettieri Kirk M. Druey

Asthma, a common disorder that affects >250 million people worldwide, is defined by exaggerated bronchoconstriction to inflammatory mediators including acetylcholine (ACh), bradykinin and histamine-also termed airway hyper-responsiveness. Nearly 10% of people with asthma have severe, treatment-resistant disease, which is frequently associated with immunoglobulin-E sensitization to ubiquitous fu...

2013
Salvatore Leonardi Alfina Coco Michele Miraglia Del Giudice Gianluigi L. Marseglia Mario La Rosa

Asthma is traditionally defined as a chronic, multisystem, multicellular disease characterized by bronchial hyper-responsiveness (BHR) and lung inflammation. In this illness is particularly involving the airway epithelium as the place where the inflammation begins, develops and often returns through airway remodelling. In specific way there is also an evolving awareness of the active participat...

2009
M Lommatzsch Y Lindner A Edner K Bratke M Kuepper J C Virchow

BACKGROUND Regular use of inhaled beta(2)-agonists has been associated with a paradoxical loss of asthma control and a deterioration of airway hyper-responsiveness, but the underlying mechanism is unknown. The neurotrophin brain-derived neurotrophic factor (BDNF) has recently been identified as a mediator of airway hyper-responsiveness in asthma. METHODS Eighteen patients with mild allergic a...

2013
Mohammad Afzal Khan

UNLABELLED BACKGROUND Airway inflammation stimulates proliferation of airway smooth muscle cell, which contributes to the development of hyperplasia and hypertrophy of smooth muscle cell. The increase in airway smooth muscle cell mass is believed to be due to an up-regulation of inflammatory mediators in the airway. It is now well recognized that chronic inflammation as well as airway hyper-...

2016
Chihiro Mitsui Keiichi Kajiwara Hiroaki Hayashi Jun Ito Haruhisa Mita Emiko Ono Noritaka Higashi Yuma Fukutomi Kiyoshi Sekiya Takahiro Tsuburai Kazuo Akiyama Kazuhiko Yamamoto Masami Taniguchi

Recently, the evidence for platelet association with allergic diseases has been established by many researches. Activated platelets contribute to airway hyper reactivity, bronchoconstriction, airway inflammation and airway remodeling in asthmatic patients. We aimed to clarify platelet activation and key molecules on platelets, which are mainly associated with the mechanism underlying aspirin-ex...

Journal: :Annals of clinical and laboratory science 2004
Graziano Riccioni Carmine Di Ilio Nicolantonio D'Orazio

Chronic stable asthma is characterized by inflammation of the airway wall, with abnormal accumulation of basophils, eosinophils, lymphocytes, mast cells, macrophages, dendritic cells, and myofibroblasts. Airway inflammation is not limited to severe asthma, but is also found in mild and moderate asthma. This inflammation results in a peculiar type of lymphocytic infiltration whereby Th2 lymphocy...

نمودار تعداد نتایج جستجو در هر سال

با کلیک روی نمودار نتایج را به سال انتشار فیلتر کنید