نتایج جستجو برای: 24 d

تعداد نتایج: 894214  

Journal: :Bioscience, Biotechnology, and Biochemistry 1993

Journal: :Forum Médical Suisse ‒ Swiss Medical Forum 2011

Journal: :Anticancer research 2009
Els Vanoirbeek Guy Eelen Lieve Verlinden Kathleen Marchal Kristof Engelen Bart De Moor Ine Beullens Suzanne Marcelis Pierre De Clercq Roger Bouillon Annemieke Verstuyf

BACKGROUND 1alpha,25-Dihydroxyvitamin D3 [1,25-(OH)2D3] is the biological active form of vitamin D. Its antiproliferative capacities make it a potential drug to treat diseases such as cancer. The clinical use of 1,25-(OH)2D3 as an antiproliferative agent is hampered by its calcemic effects. Hence, structural analogs such as the seco-9,11-bisnor-17-methyl analog, WY1112, have been developed with...

Journal: :Molecular pharmacology 2006
Hiromi Hamamoto Tatsuya Kusudo Naoko Urushino Hiroyuki Masuno Keiko Yamamoto Sachiko Yamada Masaki Kamakura Miho Ohta Kuniyo Inouye Toshiyuki Sakaki

Our previous studies revealed the species-based difference of CYP24A1-dependent vitamin D metabolism. Although human CYP24A1 catalyzes both C-23 and C-24 oxidation pathways, rat CYP24A1 shows almost no C-23 oxidation pathway. We tried to identify amino acid residues that cause the species-based difference by site-directed mutagenesis. In the putative substrate-binding regions, amino acid residu...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2007
David E Prosser Martin Kaufmann Brendan O'Leary Valarie Byford Glenville Jones

Studies of 25-hydroxyvitamin D(3)-24-hydroxylase (CYP24A1) have demonstrated that it is a bifunctional enzyme capable of the 24-hydroxylation of 1alpha,25-(OH)(2)D(3), leading to the excretory form, calcitroic acid, and 23-hydroxylation, culminating in 1alpha,25-(OH)(2)D(3)-26,23-lactone. The degree to which CYP24A1 performs either 23- or 24-hydroxylation is species-dependent. In this paper, we...

Journal: :Diabetes 2007
Rebecca Bailey Jason D Cooper Lauren Zeitels Deborah J Smyth Jennie H M Yang Neil M Walker Elina Hyppönen David B Dunger Elizabeth Ramos-Lopez Klaus Badenhoop Sergey Nejentsev John A Todd

OBJECTIVE Epidemiological studies have linked vitamin D deficiency with the susceptibility to type 1 diabetes. Higher levels of the active metabolite 1 alpha,25-dihydroxyvitamin D (1 alpha,25(OH)(2)D) could protect from immune destruction of the pancreatic beta-cells. 1 alpha,25(OH)(2)D is derived from its precursor 25-hydroxyvitamin D by the enzyme 1 alpha-hydroxylase encoded by the CYP27B1 ge...

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