نتایج جستجو برای: yq microdeletion

تعداد نتایج: 1741  

2013
Ramaswamy Suganthi VV Vijesh Sanjay Jayachandran Jahangir Ali Fathima Benazir

BACKGROUND Y chromosomal microdeletion is an important genetic disorder, which may arise due to intrachromosomal recombination between homologous sequences in the male specific region of the human Y chromosome. It is frequently associated with the quantitative reduction of sperm. The screening for Y chromosomal microdeletions has a great clinical value. OBJECTIVE To develop a sequence tagged ...

Journal: :Clinical genetics 2016
X Ning S Farschtschi A Jones H Kehrer-Sawatzki V-F Mautner J M Friedman

Microdeletions of the entire NF1 gene and surrounding genomic region occur in about 5% of patients with neurofibromatosis 1 (NF1). NF1 microdeletion patients usually have more cutaneous and plexiform neurofibromas and a higher risk of developing malignant peripheral nerve sheath tumors than other people with NF1. Somatic overgrowth has also been observed in NF1 microdeletion patients, an observ...

Journal: :Journal of medical genetics 2008
D A Koolen A J Sharp J A Hurst H V Firth S J L Knight A Goldenberg P Saugier-Veber R Pfundt L E L M Vissers A Destrée B Grisart L Rooms N Van der Aa M Field A Hackett K Bell M J M Nowaczyk G M S Mancini P J Poddighe C E Schwartz E Rossi M De Gregori L L Antonacci-Fulton M D McLellan J M Garrett M A Wiechert T L Miner S Crosby R Ciccone L Willatt A Rauch M Zenker S Aradhya M A Manning T M Strom J Wagenstaller A C Krepischi-Santos A M Vianna-Morgante C Rosenberg S M Price H Stewart C Shaw-Smith H G Brunner A O M Wilkie J A Veltman O Zuffardi E E Eichler B B A de Vries

BACKGROUND The chromosome 17q21.31 microdeletion syndrome is a novel genomic disorder that has originally been identified using high resolution genome analyses in patients with unexplained mental retardation. AIM We report the molecular and/or clinical characterisation of 22 individuals with the 17q21.31 microdeletion syndrome. RESULTS We estimate the prevalence of the syndrome to be 1 in 1...

Journal: :Turk pediatri arsivi 2014
Murat Özkale İlknur Erol

22q11.2 microdeletion which involves DiGeorge syndrome, velo-cardiofacial syndrome and conotruncal anomaly face syndrome occurs as a result of a deletion in the short segment of the long arm of the 22th chromosome. Patients with this syndrome have a wide clinical spectrum including learning difficulty, dysmorphic face, cardiac anomalies, hypocalcemia, hypoparathyroidism, cleft palate, thymus an...

2011
Roberta Lelis Dutra Patrícia de Campos Pieri Ana Carolina Dias Teixeira Rachel Sayuri Honjo Debora Romeo Bertola Chong Ae Kim

INTRODUCTION Williams-Beuren syndrome (WBS; OMIM 194050) is caused by a hemizygous contiguous gene microdeletion at 7q11.23. Supravalvular aortic stenosis, mental retardation, overfriendliness, and ocular and renal abnormalities comprise typical symptoms in WBS. Although fluorescence in situ hybridization is widely used for diagnostic confirmation, microsatellite DNA markers are considered high...

Journal: :CoRR 2017
B. O. Akinkunmi

A logical theory of regular double or multiple recurrence of eventualities, which are regular patterns of occurrences that are repeated, in time, has been developed within the context of temporal reasoning that enabled reasoning about the problem of coincidence. i.e. if two complex eventualities, or eventuality sequences consisting respectively of component eventualities x0, x1,....,xr and y0, ...

2015
Rachel Sayuri Honjo Roberta Lelis Dutra Erika Arai Furusawa Evelin Aline Zanardo Larissa Sampaio de Athayde Costa Leslie Domenici Kulikowski Debora Romeo Bertola Chong Ae Kim

Williams-Beuren syndrome (WBS) is a genetic disease caused by a microdeletion in the 7q11.23 region. It is characterized by congenital heart disease, mainly supravalvular aortic stenosis, mental retardation, mild short stature, facial dysmorphisms, and variable abnormalities in different systems. Objectives. To report the clinical findings of 55 Brazilian patients confirmed by multiplex ligatio...

2013
Xiaohui Gong Xi Wu Xiaojing Ma Dandan Wu Ting Zhang Li He Shengying Qin Xiaotian Li

OBJECTIVE The current study aimed to develop a reliable targeted array comparative genomic hybridization (aCGH) to detect microdeletions and microduplications in congenital conotruncal defects (CTDs), especially on 22q11.2 region, and for some other chromosomal aberrations, such as 5p15-5p, 7q11.23 and 4p16.3. METHODS Twenty-seven patients with CTDs, including 12 pulmonary atresia (PA), 10 do...

Journal: :The Laryngoscope 2014
Brendan P O'Connell Allison M Dobbie Samuel L Oyer Melissa Montiel Kathryn Hufnagle Krishna G Patel Christopher M Discolo Shaun A Nguyen David R White

OBJECTIVES/HYPOTHESIS Determine the impact of adenoid size and prior adenoidectomy on outcomes of sphincter pharyngoplasty. STUDY DESIGN Retrospective review. METHODS Retrospective review of patients 18 years of age or younger, who underwent sphincter pharyngoplasty for velopharyngeal insufficiency (VPI) from 2007 to 2012. Nasal endoscopy and nasometry testing were administered pre- and pos...

2013
Heike Vogel Dirk Montag Timo Kanzleiter Wenke Jonas Daniela Matzke Stephan Scherneck Alexandra Chadt Jonas Töle Reinhart Kluge Hans-Georg Joost Annette Schürmann

A region on mouse distal chromosome 1 (Chr. 1) that is highly enriched in quantitative trait loci (QTLs) controlling neural and behavioral phenotypes overlaps with the peak region of a major obesity QTL (Nob3.38), which we identified in an intercross of New Zealand Obese (NZO) mice with C57BL/6J (B6). By positional cloning we recently identified a microdeletion within this locus causing the dis...

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