نتایج جستجو برای: mitochondrial trna

تعداد نتایج: 147584  

2014
John W. Yarham Tek N. Lamichhane Angela Pyle Sandy Mattijssen Enrico Baruffini Francesco Bruni Claudia Donnini Alex Vassilev Langping He Emma L. Blakely Helen Griffin Mauro Santibanez-Koref Laurence A. Bindoff Ileana Ferrero Patrick F. Chinnery Robert McFarland Richard J. Maraia Robert W. Taylor

Identifying the genetic basis for mitochondrial diseases is technically challenging given the size of the mitochondrial proteome and the heterogeneity of disease presentations. Using next-generation exome sequencing, we identified in a patient with severe combined mitochondrial respiratory chain defects and corresponding perturbation in mitochondrial protein synthesis, a homozygous p.Arg323Gln ...

Journal: :Nucleic acids research 2003
Louis Levinger Richard Giegé Catherine Florentz

The human mitochondrial genome encodes 22 tRNAs interspersed among the two rRNAs and 11 mRNAs, often without spacers, suggesting that tRNAs must be efficiently excised. Numerous maternally transmitted diseases and syndromes arise from mutations in mitochondrial tRNAs, likely due to defect(s) in tRNA metabolism. We have systematically explored the effect of pathogenic mutations on tRNA(Ile) prec...

2010
Seyed Mohammad Seyedhassani Massoud Houshmand Seyed Mehdi Kalantar Abbas Aflatoonian Glayol Modabber Fatemeh Hadipour Hossein Fallahzadeh

Background: Mitochondrial transfer RNAs (tRNA) genes are essential components of protein biosynthesis. These genes are hotspots for mutations. These mutations are associated with a wide spectrum of human disease. Many genetic factors are known in assessment of repeated pregnancy loss (RPL). Objective: The aim of this study was analysis of tRNA Thr and tRNA Pro in women with RPL. Materials and M...

Journal: :Journal of medical genetics 2011
Xukun Yan Xinjian Wang Zhengmin Wang Shan Sun Guoling Chen Yingzi He Jun Qin Mo Ronghua Li Pingping Jiang Qin Lin Mingzhi Sun Wen Li Yan Bai Jianning Zhang Yi Zhu Jianxin Lu Qingfeng Yan Huawei Li Min-Xin Guan

The authors report here the clinical, genetic, molecular and biochemical characterisation of a large five-generation Han Chinese pedigree with maternally transmitted non-syndromic hearing loss. 17 of 35 matrilineal relatives exhibited variable severity and age at onset of sensorineural hearing loss. The average age at onset of hearing loss in matrilineal relatives of this family is 29 years, wh...

Journal: :Circulation research 2011
Shiwen Wang Ronghua Li Andrea Fettermann Zongbin Li Yaping Qian Yuqi Liu Xinjian Wang Anna Zhou Jun Qin Mo Li Yang Pingping Jiang Andreas Taschner Walter Rossmanith Min-Xin Guan

RATIONALE Despite maternal transmission of hypertension in some pedigrees, pathophysiology of maternally inherited hypertension remains poorly understood. OBJECTIVE To establish a causative link between mitochondrial dysfunction and essential hypertension. METHOD AND RESULTS A total of 106 subjects from a large Chinese family underwent clinical, genetic, molecular, and biochemical evaluatio...

2011
Shiwen Wang Ronghua Li Andrea Fettermann Zongbin Li Yaping Qian Yuqi Liu Xinjian Wang Anna Zhou Jun Qin Mo Li Yang Pingping Jiang Andreas Taschner Walter Rossmanith Min-Xin Guan

Rational: Despite maternal transmission of hypertension in some pedigrees, pathophysiology of maternally inherited hypertension remains poorly understood. Objective: To establish a causative link between mitochondrial dysfunction and essential hypertension. Method and Results: A total of 106 subjects from a large Chinese family underwent clinical, genetic, molecular, and biochemical evaluations...

2014
Diego Calderon

M itochondria are integral to proper cell function, and mutations in its small genome (mtDNA) are associated with many diseases, along with the progression of normal aging [9]. While mtDNA has been extensively studied, not much is known about transcriptional variations of mitochondrial genes [3]. Recently, tRNA modifications have been the focus of intense study owing to their putative role in d...

2011
Silvia Francisci Arianna Montanari Cristina De Luca Laura Frontali

Recent results from several laboratories have confirmed that human and yeast leucyl- and valyl-tRNA synthetases can rescue the respiratory defects due to mutations in mitochondrial tRNA genes. In this report we show that this effect cannot be ascribed to the catalytic activity per se and that isolated domains of aminoacyl-tRNA synthetases and even short peptides thereof have suppressing effects.

Journal: :Archives of neurology 2010
Tim M Young Emma L Blakely Helen Swalwell Janet E Carter Luke D Kartsounis Dominic G O'Donovan Douglass M Turnbull Robert W Taylor Rajith N de Silva

BACKGROUND Mitochondrial diseases are characterized by wide phenotypic and genetic variability, but presentations in adults with akinetic rigidity and hyperkinetic movement disorders are rare. OBJECTIVES To describe clinically a subject with progressive neurodegeneration characterized by psychosis, dementia, and akinesia-rigidity, and to associate this phenotype with a novel mitochondrial tra...

Journal: :Journal of medical genetics 1998
H Onishi T Hanihara N Sugiyama C Kawanishi E Iseki Y Maruyama Y Yamada K Kosaka S Yagishita H Sekihara S Satoh

We report on pancreatic exocrine dysfunction in families that have the mitochondrial tRNA(Leu)(UUR) gene mutation. These families exhibited maternally inherited diabetes mellitus (DM) and an A to G substitution at nt 3243 of the mitochondrial tRNA(Leu)(UUR) gene (A3243G mutation). Pancreatic necropsy samples from one proband showed accumulation of degenerated mitochondria in pancreatic acinar c...

نمودار تعداد نتایج جستجو در هر سال

با کلیک روی نمودار نتایج را به سال انتشار فیلتر کنید