نتایج جستجو برای: lineage leukemia

تعداد نتایج: 317209  

2018
Charlotta Böiers Simon E. Richardson Emma Laycock Alya Zriwil Virginia A. Turati John Brown Jason P. Wray Dapeng Wang Chela James Javier Herrero Ewa Sitnicka Stefan Karlsson Andrew J.H. Smith Sten Erik W. Jacobsen Tariq Enver

ETV6-RUNX1 is associated with childhood acute B-lymphoblastic leukemia (cALL) functioning as a first-hit mutation that initiates a clinically silent pre-leukemia in utero. Because lineage commitment hierarchies differ between embryo and adult, and the impact of oncogenes is cell-context dependent, we hypothesized that the childhood affiliation of ETV6-RUNX1 cALL reflects its origins in a progen...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2013
Kazuki Okuyama Tomokatsu Ikawa Bernhard Gentner Katsuto Hozumi Ratanakanit Harnprasopwat Jun Lu Riu Yamashita Daon Ha Takae Toyoshima Bidisha Chanda Toyotaka Kawamata Kazuaki Yokoyama Shusheng Wang Kiyoshi Ando Harvey F Lodish Arinobu Tojo Hiroshi Kawamoto Ai Kotani

Lineage specification is thought to be largely regulated at the level of transcription, where lineage-specific transcription factors drive specific cell fates. MicroRNAs (miR), vital to many cell functions, act posttranscriptionally to decrease the expression of target mRNAs. MLL-AF4 acute lymphocytic leukemia exhibits both myeloid and B-cell surface markers, suggesting that the transformed cel...

Journal: :Blood 1997
F M Uckun P Gaynon H Sather D Arthur M Trigg D Tubergen J Nachman P Steinherz M G Sensel G R Reaman

Leukemic cells from a subset of children with acute lymphoblastic leukemia (ALL) express lymphoid antigens of both T lineage and B lineage, but the clinical significance of this immunophenotype is unknown. We now report the first comprehensive comparison of treatment outcomes among a large cohort of children with CD2+ CD19+ biphenotypic ALL (N = 77), B-lineage ALL (BL) (N = 1,631), or T-lineage...

2016
Hee-Je Kim

which permits unrestricted non-commercial use, distribution, and reproduction in any medium, provided the original work is properly cited. Mixed-phenotype acute leukemia (MPAL) is a rare but difficult to treat hematologic malignancy with immuno-phenotypic co-expression of at least two cell lineages, or with only rare cases involving all three lineages, e.g. myeloid with B-or T-lymphoid or all t...

Journal: :Blood 2007
Lorna Pearn Janet Fisher Alan K Burnett Richard L Darley

Although hyperactivation of Ras is a common feature of myeloid malignancy, its role in subverting hematopoiesis is unclear. We have examined the influence of Ras on normal human uncommitted myeloid subsets and show that expression of this oncogene strongly favors monocyte lineage selection in bipotential granulocyte/macrophage progenitors while inhibiting colony formation in other uncommitted s...

2013
James D. Phelan Ingrid Saba Hui Zeng Christian Kosan Malynda S. Messer H. Andre Olsson Jennifer Fraszczak David A. Hildeman Bruce J. Aronow Tarik Möröy H. Leighton Grimes

Growth factor independent 1 (Gfi1) is a transcriptional repressor originally identified as a gene activated in T-cell leukemias induced by Moloney-murine-leukemia virus infection. Notch1 is a transmembrane receptor that is frequently mutated in human T-cell acute lymphoblastic leukemia (T-ALL). Gfi1 is an important factor in the initiation and maintenance of lymphoid leukemias and its deficienc...

2015
Elena Maino Anna Maria Scattolin Piera Viero Rosaria Sancetta Anna Pascarella Michele Vespignani Renato Bassan

The introduction of newer cytotoxic monoclonal antibodies and chimeric antigen receptor modified T cells is opening a new age in the management of B-lineage adult acute lymphoblastic leukemia. This therapeutic change must be very positively acknowledged because of the limits of intensive chemotherapy programs and allogeneic stem cell transplantation. In fact, with these traditional therapeutic ...

Journal: :Blood 1997
F M Uckun Z Yang H Sather P Steinherz J Nachman B Bostrom L Crotty M Sarquis O Ek T Zeren D Tubergen G Reaman P Gaynon

We found a marked variation in BCL-2 oncoprotein expression levels of primary leukemic cells from 338 children with newly diagnosed acute lymphoblastic leukemia (ALL). None of the high-risk features predictive of poor treatment outcome in childhood ALL, such as older age, high white blood cell (WBC) count, organomegaly, T-lineage immunophenotype, ability of leukemic cells to cause overt leukemi...

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