نتایج جستجو برای: ebolavirus

تعداد نتایج: 1058  

2016
Viruses Editorial Office

The Viruses Editorial Office wishes to notify its readers of corrections in [1].[...].

Journal: :Journal of virology 2014
Adrianna P P Zhang Zachary A Bornholdt Dafna M Abelson Erica Ollmann Saphire

The VP24 protein plays an essential, albeit poorly understood role in the filovirus life cycle. VP24 is only 30% identical between Marburg virus and the ebolaviruses. Furthermore, VP24 from the ebolaviruses is immunosuppressive, while that of Marburg virus is not. The crystal structure of Marburg virus VP24, presented here, reveals that although the core is similar between the viral genera, Mar...

2011
Ana Negredo Gustavo Palacios Sonia Vázquez-Morón Félix González Hernán Dopazo Francisca Molero Javier Juste Juan Quetglas Nazir Savji Maria de la Cruz Martínez Jesus Enrique Herrera Manuel Pizarro Stephen K. Hutchison Juan E. Echevarría W. Ian Lipkin Antonio Tenorio

Filoviruses, amongst the most lethal of primate pathogens, have only been reported as natural infections in sub-Saharan Africa and the Philippines. Infections of bats with the ebolaviruses and marburgviruses do not appear to be associated with disease. Here we report identification in dead insectivorous bats of a genetically distinct filovirus, provisionally named Lloviu virus, after the site o...

2015
Andrew Hickey Sarah Cheeseman Barthel Tyann Blessington Yandace K. Brown Diana Y. Wong William N. Albrecht Mark Freese Teresa Quitugua

Introduction An epidemic of ebolavirus in West Africa, which was first identified in March 2014, is now the largest Ebola Virus Disease (EVD) outbreak on record. On 8 August 2014, the WHO declared the Ebola outbreak in West Africa a Public Health Emergency of International Concern (PHEIC). As of 4 September 2014, Guinea, Liberia, Sierra Leone, and Nigeria have reported 3,707 cases (2,106 confir...

2015
Enzo Maria Vingolo Giuseppe Alessio Messano Serena Fragiotta Leopoldo Spadea Stefano Petti

Ebola virus disease (EVD--formerly known as Ebola hemorrhagic fever) is a severe hemorrhagic fever caused by lipid-enveloped, nonsegmented, negative-stranded RNA viruses belonging to the genus Ebolavirus. Case fatality rates may reach up to 76% of infected individuals, making this infection a deadly health problem in the sub-Saharan population. At the moment, there are still no indications on o...

Journal: :The New England journal of medicine 2014
Sylvain Baize Delphine Pannetier Lisa Oestereich Toni Rieger Lamine Koivogui N'Faly Magassouba Barrè Soropogui Mamadou Saliou Sow Sakoba Keïta Hilde De Clerck Amanda Tiffany Gemma Dominguez Mathieu Loua Alexis Traoré Moussa Kolié Emmanuel Roland Malano Emmanuel Heleze Anne Bocquin Stephane Mély Hervé Raoul Valérie Caro Dániel Cadar Martin Gabriel Meike Pahlmann Dennis Tappe Jonas Schmidt-Chanasit Benido Impouma Abdoul Karim Diallo Pierre Formenty Michel Van Herp Stephan Günther

In March 2014, the World Health Organization was notified of an outbreak of a communicable disease characterized by fever, severe diarrhea, vomiting, and a high fatality rate in Guinea. Virologic investigation identified Zaire ebolavirus (EBOV) as the causative agent. Full-length genome sequencing and phylogenetic analysis showed that EBOV from Guinea forms a separate clade in relationship to t...

Journal: :Cell 2014
John Misasi Nancy J. Sullivan

Ebolaviruses cause a severe hemorrhagic fever syndrome that is rapidly fatal to humans and nonhuman primates. Ebola protein interactions with host cellular proteins disrupt type I and type II interferon responses, RNAi antiviral responses, antigen presentation, T-cell-dependent B cell responses, humoral antibodies, and cell-mediated immunity. This multifaceted approach to evasion and suppressio...

Journal: :Journal of virology 2010
Mathieu Mateo St Patrick Reid Lawrence W Leung Christopher F Basler Viktor E Volchkov

The Ebolavirus VP24 protein counteracts alpha/beta interferon (IFN-alpha/beta) and IFN-gamma signaling by blocking the nuclear accumulation of tyrosine-phosphorylated STAT1 (PY-STAT1). According to the proposed model, VP24 binding to members of the NPI-1 subfamily of karyopherin alpha (KPNalpha) nuclear localization signal receptors prevents their binding to PY-STAT1, thereby preventing PY-STAT...

2016
Robert W. Cross Chad E. Mire Viktoriya Borisevich Joan B. Geisbert Karla A. Fenton Thomas W. Geisbert

Small-animal models have been developed for several Filoviridae species; however, serial adaptation was required to produce lethal infection. These adapted viruses have sequence changes in several genes, including those that modulate the host immune response. Nonhuman primate models do not require adaptation of filoviruses. Here, we describe lethal models of disease for Bundibugyo, Sudan, and Z...

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