نتایج جستجو برای: btk expression

تعداد نتایج: 873168  

Journal: :The Journal of Experimental Medicine 1998
Nanette Solvason Wei Wei Wu Nisha Kabra Fridjtof Lund-Johansen Maria Grazia Roncarolo Timothy W. Behrens Didier A.M. Grillot Gabriel Nunez Emma Lees Maureen Howard

Mutations in the tyrosine kinase, Btk, result in a mild immunodeficiency in mice (xid). While B lymphocytes from xid mice do not proliferate to anti-immunoglobulin (Ig), we show here induction of the complete complement of cell cycle regulatory molecules, though the level of induction is about half that detected in normal B cells. Cell cycle analysis reveals that anti-Ig stimulated xid B cells ...

Journal: :Respiratory Research 2001
Kazuhiro Usui Yoji Sasahara Ryushi Tazawa Koichi Hagiwara Satoshi Tsukada Toshio Miyawaki Shigeru Tsuchiya Toshihiro Nukiwa

BACKGROUND X-linked agammaglobulinemia (XLA) is a humoral immunodeficiency caused by disruption of the Bruton's tyrosine kinase (BTK) gene. Typical XLA patients suffer recurrent and severe bacterial infections in childhood. METHODS Flow cytometric analysis of the peripheral monocytes using the anti-BTK antibody was used to characterize a 27 year old male patient with mild hypogammaglobulinemi...

2014
Yan-Feng Li Koon-Guan Lee Xijun Ou Kong-Peng Lam

Stimulation of TLR7/9 by their respective ligands leads to the activation of IκB kinase α (IKKα) and Interferon Regulatory Factor 1 (IRF-1) and results in interferon (IFN)-β production in conventional dendritic cells (cDC). However, which other signaling molecules are involved in IKKα and IRF-1 activation during TLR7/9 signaling pathway are not known. We and others have shown that Bruton's Tyro...

Journal: :Scandinavian journal of immunology 2001
M Speletas M Kanariou F Kanakoudi-Tsakalidou E Papadopoulou-Alataki K Arvanitidis E Pardali A Constantopoulos G Kartalis M Vihinen P Sideras K Ritis

Bruton's tyrosine kinase (Btk) is a nonreceptor tyrosine kinase, critical for B-cell development and function. Mutations that inactivate this kinase were found in families with X-linked agammaglobulinaemia (XLA). In this study the Btk gene was analyzed in 13 registered Greek patients with XLA phenotype originated from 12 unrelated families, in order to provide a definite diagnosis of the XLA. T...

Journal: :Journal of immunology 2007
Kishore R Alugupalli Shizuo Akira Egil Lien John M Leong

Bacteremia is one of the leading causes of death by infectious disease. To understand the immune mechanisms required for the rapid control of bacteremia, we studied Borrelia hermsii, a bacterial pathogen that colonizes the blood stream of humans and rodents to an extremely high density. A T cell-independent IgM response is essential and sufficient for controlling B. hermsii bacteremia. Mice def...

Journal: :Cancer discovery 2014
David Chiron Maurizio Di Liberto Peter Martin Xiangao Huang Jeff Sharman Pedro Blecua Susan Mathew Priyanka Vijay Ken Eng Siraj Ali Amy Johnson Betty Chang Scott Ely Olivier Elemento Christopher E Mason John P Leonard Selina Chen-Kiang

UNLABELLED Despite the unprecedented clinical activity of the Bruton tyrosine kinase (BTK) inhibitor ibrutinib in mantle cell lymphoma (MCL), acquired resistance is common. By longitudinal integrative whole-exome and whole-transcriptome sequencing and targeted sequencing, we identified the first relapse-specific C481S mutation at the ibrutinib binding site of BTK in MCL cells at progression fol...

2014
Lyubov Zaitseva Megan Y. Murray Manar S. Shafat Matthew J. Lawes David J. MacEwan Kristian M. Bowles Stuart A. Rushworth

Pharmacological targeting of BTK using ibrutinib has recently shown encouraging clinical activity in a range of lymphoid malignancies. Recently we reported that ibrutinib inhibits human acute myeloid leukemia (AML) blast proliferation and leukemic cell adhesion to the surrounding bone marrow stroma cells. Here we report that in human AML ibrutinib, in addition, functions to inhibit SDF1/CXCR4-m...

2014
Jin-Shuen Chen Li-Chien Chang Shyh-Jer Huang Chao-Wen Cheng

The importance of B-cell activation and immune complex-mediated Fc-receptor activation in the pathogenesis of immunologically mediated glomerulonephritis has long been recognized. The two nonreceptor tyrosine kinases, spleen tyrosine kinase (Syk) and Bruton's tyrosine kinase (Btk), are primarily expressed by hematopoietic cells, and participate in B-cell-receptor- and Fc-receptor-mediated activ...

Journal: :Clinical immunology 2006
Arnon Broides Wenjian Yang Mary Ellen Conley

No clear genotype/phenotype correlations have been established in patients with X-linked agammaglobulinemia (XLA). To determine if the specific mutation in Btk might be one of the factors that influences the severity of disease or if polymorphic variants in Tec, a cytoplasmic tyrosine kinase that might substitute for Btk, could contribute to the clinical phenotype, we examined the age at diagno...

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