نتایج جستجو برای: bortezomib

تعداد نتایج: 5578  

Journal: :Molecular cancer therapeutics 2008
Zhong Chen Justin L Ricker Pramit S Malhotra Liesl Nottingham Lorena Bagain Tin Lap Lee Ning T Yeh Carter Van Waes

Head and neck squamous cell carcinomas (HNSCC) exhibit constitutive activation of transcription factors nuclear factor-kappaB (NF-kappaB) and activator protein-1 (AP-1), which are modulated by the proteasome and promote resistance to cell death. HNSCC show variable sensitivity to the proteasome inhibitor bortezomib in vitro as well as in murine xenografts and patient tumors in vivo, and the mec...

Journal: :Journal of clinical oncology : official journal of the American Society of Clinical Oncology 2010
Jean-Luc Harousseau Michel Attal Hervé Avet-Loiseau Gerald Marit Denis Caillot Mohamad Mohty Pascal Lenain Cyrille Hulin Thierry Facon Philippe Casassus Mauricette Michallet Hervé Maisonneuve Lotfi Benboubker Frédéric Maloisel Marie-Odile Petillon Iain Webb Claire Mathiot Philippe Moreau

PURPOSE To compare efficacy and safety of bortezomib plus dexamethasone and vincristine plus doxorubicin plus dexamethasone (VAD) as induction before stem-cell transplantation in previously untreated myeloma. PATIENTS AND METHODS Four hundred eighty-two patients were randomly assigned to VAD (n = 121), VAD plus dexamethasone, cyclophosphamide, etoposide, and cisplatin (DCEP) consolidation (n ...

Journal: :The Journal of biological chemistry 2009
Kuen-Feng Chen Pei-Yen Yeh Chiun Hsu Chih-Hung Hsu Yen-Shen Lu Hsing-Pang Hsieh Pei-Jer Chen Ann-Lii Cheng

Hepatocellular carcinoma (HCC) is one of the most common and aggressive human malignancies. Recombinant tumor necrosis factor-related apoptosis-inducing ligand (TRAIL) is a promising anti-tumor agent. However, many HCC cells show resistance to TRAIL-induced apoptosis. In this study, we showed that bortezomib, a proteasome inhibitor, overcame TRAIL resistance in HCC cells, including Huh-7, Hep3B...

2016

One resistance mechanism in malignant gliomas (MG) involves nuclear factor-κB (NF-κB) activation. Bortezomib prevents proteasomal degradation of NF-κB inhibitor α (NFKBIA), an endogenous regulator of NF-κB signaling, thereby limiting the effects of NF-κB on tumor survival and resistance. A presurgical phase II trial of bortezomib in recurrent MG was performed to determine drug concentration in ...

2012
Danielle M Brander Anne W Beaven

Follicular lymphoma is predominantly managed as a chronic disease, with intermittent chemo/immunotherapy reserved for symptomatic progression. It is considered incurable with conventional treatments, and current therapeutic options are associated with significant toxicities that are especially limiting in older patients. Bortezomib (PS-341; Velcade(®)), a first-in-class drug targeting the prote...

Journal: :The New England journal of medicine 2008
Jesús F San Miguel Rudolf Schlag Nuriet K Khuageva Meletios A Dimopoulos Ofer Shpilberg Martin Kropff Ivan Spicka Maria T Petrucci Antonio Palumbo Olga S Samoilova Anna Dmoszynska Kudrat M Abdulkadyrov Rik Schots Bin Jiang Maria-Victoria Mateos Kenneth C Anderson Dixie L Esseltine Kevin Liu Andrew Cakana Helgi van de Velde Paul G Richardson

BACKGROUND The standard treatment for patients with multiple myeloma who are not candidates for high-dose therapy is melphalan and prednisone. This phase 3 study compared the use of melphalan and prednisone with or without bortezomib in previously untreated patients with multiple myeloma who were ineligible for high-dose therapy. METHODS We randomly assigned 682 patients to receive nine 6-wee...

Journal: :Acta dermatovenerologica Alpina, Pannonica, et Adriatica 2012
Nicola di Meo Serena Bergamo Adina Dondas Giusto Trevisan

Bortezomib is a proteasome inhibitor that has proven to be a very effective treatment for multiple myeloma. There is considerable debate about the potential for reactivation of the varicella zoster virus (VZV) in patients with multiple myeloma during treatment with bortezomib. This report describes the case of a 70-year-old patient with multiple myeloma that developed bilateral herpes zoster sh...

2015
Adil Doganay Duru Tolga Sutlu Ann Wallblom Katarina Uttervall Johan Lund Birgitta Stellan Gösta Gahrton Hareth Nahi Evren Alici Sumitra Deb

Loss of the chromosomal region 8p21 negatively effects survival in patients with multiple myeloma (MM) that undergo autologous stem cell transplantation (ASCT). In this study, we aimed to identify the immunological and molecular consequences of del(8)(p21) with regards to treatment response and bortezomib resistance. In patients receiving bortezomib as a single first line agent without any high...

Journal: :Molecular cancer therapeutics 2009
Bokyung Sung Ajaikumar B Kunnumakkara Gautam Sethi Preetha Anand Sushovan Guha Bharat B Aggarwal

Curcumin (diferuloylmethane), a yellow pigment in turmeric, has been shown to inhibit the activation of nuclear factor-kappaB (NF-kappaB), a transcription factor closely linked to chemoresistance in multiple myeloma cells. Whether curcumin can overcome chemoresistance and enhance the activity of thalidomide and bortezomib, used to treat patients with multiple myeloma, was investigated in vitro ...

2012
Koichi Yanaba Yoshihide Asano Yayoi Tada Makoto Sugaya Takafumi Kadono Shinichi Sato

BACKGROUND Bortezomib is a proteasome inhibitor that has shown impressive efficacy in the treatment of multiple myeloma. In mice, the addition of dextran sulfate sodium (DSS) to drinking water leads to acute colitis that can serve as an experimental animal model for human ulcerative colitis. METHODOLOGY/PRINCIPAL FINDINGS Bortezomib treatment was shown to potently inhibit murine DSS-induced c...

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