نتایج جستجو برای: tki

تعداد نتایج: 3304  

2015
Tomokazu Sazuka Naoki Nihei Kazuyoshi Nakamura Shinichi Sakamoto Satoshi Fukasawa Atsushi Komaru Takeshi Ueda Tatsuo Igarashi Tomohiko Ichikawa

PURPOSE Single-agent interferon (IFN) is no longer regarded as a standard option for first-line systemic treatment of metastatic renal cell carcinoma (RCC) in Western countries. However, some patients with favorable-risk RCC may still achieve complete and long-lasting remission in response to IFN treatment. The present study compared favorable-risk Japanese patients with metastatic RCC Japanese...

Journal: :Blood 2017
Rebecca Warfvinge Linda Geironson Mikael N E Sommarin Stefan Lang Christine Karlsson Teona Roschupkina Leif Stenke Jesper Stentoft Ulla Olsson-Strömberg Henrik Hjorth-Hansen Satu Mustjoki Shamit Soneji Johan Richter Göran Karlsson

Understanding leukemia heterogeneity is critical for the development of curative treatments as the failure to eliminate therapy-persistent leukemic stem cells (LSCs) may result in disease relapse. Here we have combined high-throughput immunophenotypic screens with large-scale single-cell gene expression analysis to define the heterogeneity within the LSC population in chronic phase chronic myel...

Journal: :Clinical advances in hematology & oncology : H&O 2012
Jorge Cortes Jerald Radich Michael J Mauro

The development of tyrosine kinase inhibitors (TKIs) that inhibit signaling of the constitutive BCR-ABL protein revolutionized the treatment of chronic myelogenous leukemia (CML). These agents have dramatically changed the treatment landscape for CML, shifting the use of allogeneic stem cell transplantation to selected patients in the salvage setting. Four BCR-ABL TKIs are now commercially avai...

2014
Anna L. Illert Anna K. Seitz Christoph Rummelt Stefanie Kreutmair Richard A. Engh Samantha Goodstal Christian Peschel Justus Duyster Nikolas von Bubnoff

ABL tyrosine kinase inhibitors (TKI) like Imatinib, Dasatinib and Nilotinib are the gold standard in conventional treatment of CML. However, the emergence of resistance remains a major problem. Alternative therapeutic strategies of ABL TKI-resistant CML are urgently needed. We asked whether dual inhibition of BCR-ABL and Aurora kinases A-C could overcome resistance mediated by ABL kinase mutati...

2016
Daniele Santini Matteo Santoni Alessandro Conti Giuseppe Procopio Elena Verzoni Luca Galli Giuseppe di Lorenzo Ugo De Giorgi Delia De Lisi Maurizio Nicodemo Marco Maruzzo Francesco Massari Sebastiano Buti Emanuela Altobelli Elisa Biasco Riccardo Ricotta Camillo Porta Bruno Vincenzi Rocco Papalia Paolo Marchetti Luciano Burattini Rossana Berardi Giovanni Muto Rodolfo Montironi Stefano Cascinu Giuseppe Tonini

PURPOSE We retrospectively analyzed the risk of recurrence and conditional Disease-Free Survival (cDFS) in 63 patients with complete remission during treatment with tirosin kinase inhibitor (TKI), alone or with local treatment in metastatic renal cell carcinoma. RESULTS 37% patients achieve CR with TKI alone, while 63% with additional loco-regional treatments. 49% patients recurred after CR, ...

2016
Maria Karvela Pablo Baquero Elodie M. Kuntz Arunima Mukhopadhyay Rebecca Mitchell Elaine K. Allan Edmond Chan Kamil R. Kranc Bruno Calabretta Paolo Salomoni Eyal Gottlieb Tessa L. Holyoake G. Vignir Helgason

A major drawback of tyrosine kinase inhibitor (TKI) treatment in chronic myeloid leukemia (CML) is that primitive CML cells are able to survive TKI-mediated BCR-ABL inhibition, leading to disease persistence in patients. Investigation of strategies aiming to inhibit alternative survival pathways in CML is therefore critical. We have previously shown that a nonspecific pharmacological inhibition...

2017
Lijun Dong Dan Lei Haijun Zhang

Epidermal growth factor receptor (EGFR) mutations (EGFRm+) occur in 10-35% of non-small-cell lung cancer (NSCLC) cases and confer sensitivity to EGFR tyrosine kinase inhibitors (TKIs). EGFR TKIs are standard treatments for NSCLC patients harboring EGFR exon 19 deletions or exon 21 L858R point mutations. Despite initial benefit, most patients develop drug resistance, posing a challenge to oncolo...

2013
Yung-Luen Yu Ruey-Hwang Chou Jia-Hong Liang Wei-Jung Chang Kuo-Jung Su Yen-Ju Tseng Wei-Chien Huang Shao-Chun Wang Mien-Chie Hung

Tyrosine 211 (Y211) phosphorylation of proliferation cell nuclear antigen (PCNA) coincides with pronounced cancer cell proliferation and correlates with poor survival of breast cancer patients. In epidermal growth factor receptor (EGFR) tyrosine kinase inhibitor (TKI)-resistant cells, both nuclear EGFR (nEGFR) expression and PCNA Y211 phosphorylation are increased. Moreover, the resistance to E...

2017
Simon Baldacci Julien Mazieres Pascale Tomasini Nicolas Girard Florian Guisier Clarisse Audigier-Valette Isabelle Monnet Marie Wislez Maurice Pérol Pascal Dô Eric Dansin Charlotte Leduc Etienne Giroux Leprieur Denis Moro-Sibilot David Tulasne Zoulika Kherrouche Julien Labreuche Alexis B. Cortot

Background Several mechanisms of acquired resistance to EGFR tyrosine kinase inhibitors (TKIs) in EGFR-mutated NSCLC have been described including the T790M mutation and MET amplification. Whereas T790M mutation confers prolonged survival and sensitivity to 3rd generation TKIs, data are lacking on clinical features and outcome of MET-driven resistant EGFR-mutated NSCLC patients. Methods Patie...

Journal: :Clinical cancer research : an official journal of the American Association for Cancer Research 2015
Liang Zhang Xiaoen Wang Andrea J Bullock Marcella Callea Harleen Shah Jiaxi Song Kelli Moreno Barbara Visentin Douglas Deutschman David C Alsop Michael B Atkins James W Mier Sabina Signoretti Manoj Bhasin Roger A Sabbadini Rupal S Bhatt

PURPOSE VEGFR2 tyrosine kinase inhibition (TKI) is a valuable treatment approach for patients with metastatic renal cell carcinoma (RCC). However, resistance to treatment is inevitable. Identification of novel targets could lead to better treatment for patients with TKI-naïve or -resistant RCC. EXPERIMENTAL DESIGN In this study, we performed transcriptome analysis of VEGFR TKI-resistant tumor...

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